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RISS 인기검색어
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- 저자 : Xuetong Zhang (검색결과 3 건)
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Advances in Fast Vessel-Wall Magnetic Resonance Imaging Using High-Density Coil Arrays
Xuetong Yin,Nan Li,Sen Jia,Xiaoliang Zhang,Ye Li 대한자기공명의과학회 2021 Investigative Magnetic Resonance Imaging Vol.25 No.4
Arteriosclerosis is the leading cause of stroke, with a fatality rate surpassing that of ischemic heart disease. High-resolution vessel wall magnetic resonance imaging is generally recognized as a non-invasive and panoramic method for the evaluation of arterial plaque; however, this method requires improved signal-tonoise ratio and scanning speed. Recent advances in high-density head and neck coil arrays are characterized by broad coverage, multiple channels, and closefitting designs. This review analyzes fast magnetic resonance imaging from the perspective of accelerated algorithms for vessel wall imaging and demonstrates the need for effective algorithms for signal acquisition using advanced radiofrequency system. We summarize different phased-array structures under various experimental objectives and equipment conditions, introduce current research results, and propose prospective research studies in the future.
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Mechanical Properties of Recycled Concrete Reinforced by Basalt Fiber and Nano-silica
Yonggui Wang,Xuetong Zhang,Jinjin Fang,Xingguo Wang 대한토목학회 2022 KSCE JOURNAL OF CIVIL ENGINEERING Vol.26 No.8
In this study, basalt fiber (BF) and nanosilica (NS) were used to improve the mechanical properties of recycled concrete (RC), thereby promoting its utilization. The effect of BF and NS on the impact performance of RC was studied using the split Hopkinson pressure bar (SHPB) test. The internal microstructure of RC was analyzed by scanning electron microscopy, mercury intrusion porosimetry and nuclear magnetic resonance test. The results showed that, as the strain rate increased, the destruction mode transitioned from fracturing to crushing. Moreover, the impact compressive strength, dynamic increase factor, and total impact toughness increased. The replacement rate has little to no influence on the failure state, impact compressive strength and specific energy absorption. BF is conducive to enhancing the integrity and improving the impact compressive strength, peak strain and impact toughness. NS can reduce the pore content of mortar and the corrosion effect of the fiber surface, enhancing the compactness of the mortar and the bridging effect of the fiber. With an increase in NS content, the impact compressive strength, peak strain, and impact toughness increase, leading to greater destruction integrity. The results show that BF and NS can more effectively enhance the mechanical properties of RC.
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TCN1 Deficiency Inhibits the Malignancy of Colorectal Cancer Cells by Regulating the ITGB4 Pathway
Zhu Xinqiang,Jiang Xuetong,Zhang Qinglin,Huang Hailong,Shi Xiaohong,Hou Daorong,Xing Chungen 거트앤리버 소화기연관학회협의회 2023 Gut and Liver Vol.17 No.3
Background/Aims: This study aimed to investigate the biological function and regulatory mechanism of TCN1 in colorectal cancer (CRC). Methods: We studied the biological function of TCN1 by performing gain-of-function and loss-offunction analyses in HCT116 cell lines; examined the effects of TCN1 on the proliferation, apoptosis, and invasion of CRC cells; and determined potential molecular mechanisms using HCT116 and SW480 CRC lines and mouse xenotransplantation models. Tumor xenograft and colonization assays were performed to detect the tumorigenicity and metastatic foci of cells in vivo. Results: TCN1 knockdown attenuated CRC cell proliferation and invasion and promoted cell apoptosis. Overexpression of TCN1 yielded the opposite effects. In addition, TCN1-knockdown HCT116 cells failed to form metastatic foci in the peritoneum after intravenous injection. Molecular mechanism analyses showed that TCN1 interacted with integrin subunit β4 (ITGB4) to positively regulate the expression of ITGB4. TCN1 knockdown promoted the degradation of ITGB4 and increased the instability of ITGB4 and filamin A. Downregulation of ITGB4 at the protein level resulted in the disassociation of the ITGB4/plectin complex, leading to cytoskeletal damage. Conclusions: TCN1 might play an oncogenic role in CRC by regulating the ITGB4 signaling pathway.
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