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- 저자 : Tanzer, Jason M. (검색결과 3 건)
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Understanding Dental Caries : An Infectious Disease, Not a Lesion
Tanzer, Jason M. Korean Academy of Oral Biology and the UCLA Dental 1997 International Journal of Oral Biology Vol.22 No.4
Caries is an infectious disease. Cavities are its resultant lesions. The lesions should not be confused with the disease, because it leads to misunderstandings of preventive and therapeutic possibilities. This paper reviews the fundamental evidence which establishes that dental caries is an infection caused primarily by the mutans group of streptococci. The infection is powerfully influenced by the diet, especially the consumption pattern of sucrose. While other bacteria probably can play some causative role, the evidence which implicates them is limited, and suggests these other bacteria to be relatively weakly cariogenic. Substantial information clarifies the dental plaque ecology, metabolism, transmission, and conditions which determine expression of virulence of mutans streptococci. These phenomena are strongly correlated with their metabolism of sucrose. Mutans streptococci are also associated with the inception of secondary carous lesions at the enamel/plaque interface. The initial lesions of tooth decay thus reflect a microbiologically specific dental plaque infection, influenced by diet. Much less is understood about either the microbiology of progression of lesions through dentin or secondary lesions of the deep structures of the teeth. Root surface caries, like coronal caries, is closely associated with mutans streptococcal colonization; however, Actinomyces viscosus may also be a significant cariogen in this setting. The incipient lesions of tooth decay are widely thought to reflect a complex series of demineralization and remineralization events occurring under the mutans-rich plaque in the surfaces of teeth. These events ate driven by the pattern and content of dietary carbohydrate consumption which, in turn, induces fermentative activity of the plaque, decrease and eventual increase of plaque pH, and solubilization and reprecipitation of the inorganic phase of enamel and salivary ions. The net flux of mineral components out of and into the tooth determines whether teeth ostensibly remain mineralized, demineralize, or become remineralized after development of initial lesions. Salivary flow, composition, and fluoride strongly influence this complex of fluxes, as does the consumption and fermentation of carbohydrate-rich foods, especially but not exclusively those rich in sucrose.
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Case Reports : A Case of Lichen Sclerosus et Atrophicus Accompanying Bullous Morphea
( Sirin Yasar ),( Ceyda Tanzer Mumcuoglu ),( Zehra Asiran Serdar ),( Pembegul Gunes ) 대한피부과학회 2011 Annals of Dermatology Vol.23 No.3s
Bullous morphea is a rare form of morphea characterized with bullae on or around atrophic morphea plaques. Whereas lichen sclerosus et atrophicus (LSA) is a disease the etiology of which is not fully known, and which is characterized with sclerosis. Coexistence of morphea and LSA has been identified in some cases. Some authors believe that these two diseases are different manifestations which are on the same spectrum. The 70-year-old patient stated herein, presented to us for 6 months with annular, atrophic plaques, ivory color in the middle, surrounded by living erythema, on the front and back of the trunk. Occasionally bulla formation on the plaques on the trunk lateral was identified. Fibrotic and atrophic plaques of ligneous hardness were present on the front side of tibia of both legs. In the histopathologic examination, the lesions were found concordant with bullous morphea and LSA. With colchicine 1.5 mg/day, pentoxifylline 1,200 mg/day, topical calcipotriol ointment and clobetasol propionate cream, the erythema in the patient`s lesions faded and softening in the fibrotic plaques was observed. Concomitance of bullous morphea and LSA is a rarely seen, interesting coexistence which suggests a common, as yet unknown, underlying pathogenesis. (Ann Dermatol 23(S3) S354~S359, 2011)
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A Case of Lichen Sclerosus et Atrophicus Accompanying Bullous Morphea
Sirin Yasar,Ceyda Tanzer Mumcuoglu,Zehra Asiran Serdar,Pembegul Gunes 대한피부과학회 2011 Annals of Dermatology Vol.23 No.-
Bullous morphea is a rare form of morphea characterized with bullae on or around atrophic morphea plaques. Whereas lichen sclerosus et atrophicus (LSA) is a disease the etiology of which is not fully known, and which is characterized with sclerosis. Coexistence of morphea and LSA has been identified in some cases. Some authors believe that these two diseases are different manifestations which are on the same spectrum. The 70-year-old patient stated herein,presented to us for 6 months with annular, atrophic plaques,ivory color in the middle, surrounded by living erythema, on the front and back of the trunk. Occasionally bulla formation on the plaques on the trunk lateral was identified. Fibrotic and atrophic plaques of ligneous hardness were present on the front side of tibia of both legs. In the histopathologic examination, the lesions were found concordant with bullous morphea and LSA. With colchicine 1.5 mg/day,pentoxifylline 1,200 mg/day, topical calcipotriol ointment and clobetasol propionate cream, the erythema in the patient's lesions faded and softening in the fibrotic plaques was observed. Concomitance of bullous morphea and LSA is a rarely seen, interesting coexistence which suggests a common, as yet unknown, underlying pathogenesis. (Ann Dermatol 23(S3) S354∼S359, 2011)
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