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Stephen J.H. Yang,Jia Zhang,Rick C.S. Chen,Norman W.Y. Shao 한국전자통신연구원 2007 ETRI Journal Vol.29 No.6
In the mobile Internet, users generally work with handheld devices with limited computing power and small screens. Their access conditions also change frequently. In this paper, we present a novel method supporting intelligent content adaptation to better suit handheld devices. The underpinning is a unit of information (UOI)– based content adaptation method, which automatically detects semantic relationships among the components of Web contents and then reorganizes page layout to fit handheld devices based on identified UOIs. Experimental results demonstrate that our method enables more sensitive content adaptation.
Fink, Stephen P,Yang, Dong-Hoon,Barnholtz-Sloan, Jill S,Ryu, Yeon-Mi,Mikkola, Debra,Potter, John D,Lampe, Johanna W,Markowitz, Sanford D,Myung, Seung-Jae Plenum Pub. Corp.] 2013 Digestive diseases and sciences Vol.58 No.9
<P>15-Hydroxprostaglandin dehydrogenase (15-PGDH) mediates a colon neoplasia suppressor pathway, acting through metabolic antagonism of cyclooxygenase-mediated colon carcinogenesis. To determine whether the colon tumor prevention activity of 15-PGDH acts as a constant or variable effect among individuals, we determined whether 15-PGDH levels remain stable over subsite and time in the human colon, determined the extent of differences in 15-PGDH levels between different individuals, and determined whether 15-PGDH modulation mediates any part of the anti-colon tumor effect of aspirin.</P>
Chester S.J. Huang,Stephen J.H. Yang,Addison Y.S. Su 한국전자통신연구원 2012 ETRI Journal Vol.34 No.4
Unstructured peer-to-peer (p2p) networks usually employ flooding search algorithms to locate resources. However, these algorithms often require a large storage overhead or generate massive network traffic. To address this issue, previous researchers explored the possibility of building efficient p2p networks by clustering peers into communities based on their social relationships, creating social-like p2p networks. This study proposes a social relationship p2p network that uses a measure based on Hebbian theory to create a social relation weight. The contribution of the study is twofold. First, using the social relation weight, the query peer stores and searches for the appropriate response peers in social-like p2p networks. Second, this study designs a novel knowledge index mechanism that dynamically adapts social relationship p2p networks. The results show that the proposed social relationship p2p network improves search performance significantly, compared with existing approaches.
Inhibition of the prostaglandin-degrading enzyme 15-PGDH potentiates tissue regeneration
Zhang, Yongyou,Desai, Amar,Yang, Sung Yeun,Bae, Ki Beom,Antczak, Monika I.,Fink, Stephen P.,Tiwari, Shruti,Willis, Joseph E.,Williams, Noelle S.,Dawson, Dawn M.,Wald, David,Chen, Wei-Dong,Wang, Zhengh American Association for the Advancement of Scienc 2015 Science Vol.348 No.6240
<P><B>A shot in the arm for damaged tissue</B></P><P>Tissue damage can be caused by injury, disease, and even certain medical treatments. There is great interest in identifying drugs that accelerate tissue regeneration and recovery, especially drugs that might benefit multiple organ systems. Zhang <I>et al.</I> describe a compound with this desired activity, at least in mice (see the Perspective by FitzGerald). SW033291 promotes recovery of the hematopoietic system after bone marrow transplantation, prevents the development of ulcerative colitis in the intestine, and accelerates liver regeneration after hepatic surgery. It acts by inhibiting an enzyme that degrades prostaglandins, lipid signaling molecules that have been implicated in tissue stem cell maintenance.</P><P><I>Science</I>, this issue 10.1126/science.aaa2340; see also p. 1208</P><P>Agents that promote tissue regeneration could be beneficial in a variety of clinical settings, such as stimulating recovery of the hematopoietic system after bone marrow transplantation. Prostaglandin PGE2, a lipid signaling molecule that supports expansion of several types of tissue stem cells, is a candidate therapeutic target for promoting tissue regeneration in vivo. Here, we show that inhibition of 15-hydroxyprostaglandin dehydrogenase (15-PGDH), a prostaglandin-degrading enzyme, potentiates tissue regeneration in multiple organs in mice. In a chemical screen, we identify a small-molecule inhibitor of 15-PGDH (SW033291) that increases prostaglandin PGE2 levels in bone marrow and other tissues. SW033291 accelerates hematopoietic recovery in mice receiving a bone marrow transplant. The same compound also promotes tissue regeneration in mouse models of colon and liver injury. Tissues from 15-PGDH knockout mice demonstrate similar increased regenerative capacity. Thus, 15-PGDH inhibition may be a valuable therapeutic strategy for tissue regeneration in diverse clinical contexts.</P>
Chiung-Hui Peng,Shang-Jen Chang,Stephen S. Yang 대한배뇨장애요실금학회 2016 International Neurourology Journal Vol.20 No.4
A 28-year-old female with a 1-year history of ketamine abuse developed ketamine-associated urinary symptoms that were refractory to conservative treatment after the complete cessation of ketamine use. Smooth voiding with increased bladder capacity and minimal postvoid residual urine volume were achieved by performing an augmentation enterocystoplasty. An uneventful pregnancy with the vaginal delivery of a healthy baby occurred postoperatively.