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      저자 : Lingyue Shan (검색결과 2 건)
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      • Hepatoprotective Effectis of the Extracts from Rumex hanus by. Against Chronic Alcohol-Induced Liver Injury in Mice

        Lingyue Shan,Deog-Hwan Oh 한국식품영양과학회 2021 한국식품영양과학회 학술대회발표집 Vol.2021 No.10

        Alcoholic liver disease (ALD) caused by excessive alcohol intake has become a global threat. This study was designed to investigate the protective effects of bioactive ingredients extracted from Rumex hanus by. on chronic alcoholic injury of liver in mice. The extract from Rumex hanus by. (ERHB) was obtained by endotoxin antagonistic experiment in vitro. To determine the hepatoprotective effects of ERHB, mice were intragastrically administered with alcohol and treated with ERHB once daily for 6 weeks. The results indicated that endotoxin antagonism rate of 70% ethanol ERHB extract was 88.94±1.24 % in vitro. Animal experiment demonstrated that ERHB improved the activities of ALDH and ADH, reducing the activities of CYP2E1, AKP, AST and ALT significantly. Meanwhile, ERHB improved alcohol-induced dyslipidaemia of ALD mice by increasing HDL-C, decreasing the TC, TG, LDL-C and VLDL-C evidently. Additionally, ERHB ameliorated the alcohol-induced liver injury by inhibiting endotoxin-caused inflammation. Thus, the study demonstrated that ERHB exerted protective effects against alcohol-induced liver injury and could be used as functional food supplement for the treatment of ALD.

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        Rumex hanus by. Extract Protects Against Chronic Alcohol-Induced Liver Injury in Mice

        Meizi Piao,Fengwu Wang,Lingyue Shan,Yang Deng,Tiejun Chen 한국식품영양과학회 2022 Journal of medicinal food Vol.25 No.7

        Alcoholic liver disease (ALD) has become a global health problem. The hepatoprotective effects of bioactive ingredients extracted from Rumex hanus by. on chronic alcoholic liver injury was investigated for the first time. The extract from R. hanus by. (ERHB) was obtained by 70% ethanol extraction, and the endotoxin antagonism rate of ERHB was 88.94 ± 1.24% in vitro. The animal experiments demonstrated that ERHB promoted hepatic function by significantly enhancing the activities of alcohol dehydrogenase and acetaldehyde dehydrogenase, and by reducing the activities of cytochrome P450 proteins, alkaline phosphatase, aspartate aminotransferase, and alanine aminotransferase. Furthermore, ERHB improved alcohol-induced dyslipidemia by regulating lipid metabolism. In addition, ERHB ameliorated the alcohol-induced liver injury by inhibiting endotoxin-caused inflammation. Seven compounds with antagonistic activity on endotoxin were identified in ERHB. These results demonstrated that ERHB had protective effects on ALD and if the results can be confirmed in humans, it might be useful as a functional food supplement for ALD treatment.

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