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Kotani Kohei,Kawada Norifumi 거트앤리버 소화기연관학회협의회 2024 Gut and Liver Vol.18 No.1
In chronic liver disease, hepatic stellate cell activation and degeneration of liver sinusoidal endothelial cells lead to structural changes, which are secondary to fibrosis and the presence of regenerative nodules in the sinusoids, and to functional changes, which are related to vasoconstriction. The combination of such changes increases intrahepatic vascular resistance and causes portal hypertension. The subsequent increase in splanchnic and systemic hyperdynamic circulation further increases the portal blood flow, thereby exacerbating portal hypertension. In clinical practice, the hepatic venous pressure gradient is the gold-standard measure of portal hypertension; a value of ≥10 mm Hg is defined as clinically significant portal hypertension, which is severe and is associated with the risk of liver-related events. Hepatic venous pressure gradient measurement is somewhat invasive, so evidence on the utility of risk stratification by elastography and serum biomarkers is needed. The various stages of cirrhosis are associated with different outcomes. In viral hepatitis-related cirrhosis, viral suppression or elimination by nucleos(t)ide analog or direct-acting antivirals results in recompensation of liver function and portal pressure. However, careful follow-up should be continued, because some cases have residual clinically significant portal hypertension even after achieving sustained virologic response. In this study, we reviewed the current and future prospects for portal hypertension.
Correlation between Charge Asphericity and Dzyaloshinskii-Moriya Interaction
Sanghoon Kim,Kohei Ueda,Gyungchoon Go,Peong-Hwa Jang,Kyung-Jin Lee,Abderrezak Belabbes,Aurelien Manchon,Motohiro Suzuki,Yoshinori Kotani,Tetsuya Nakamura,Kohji Nakamura,Tomohiro Koyama,Daichi Chiba,Ki 한국자기학회 2018 한국자기학회 학술연구발표회 논문개요집 Vol.2018 No.3