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Application of Evaporative Cooling Technology in HDVC Converter Station
Guo, Jianhong,Wang, Haifeng,Ai, Chengliu,Gu, Guobiao Journal of International Conference on Electrical 2014 Journal of international Conference on Electrical Vol.3 No.1
Converter valve cooling device as the heat exchanger, is known as the converter valve's blood circulation system, Inefficiency of the equipment will not only lead to the component overheating damage, but also serious DC system outage. Therefore, reliable HVDC valve cooling system is essential for the HVDC transmission system. In this article, analysis on the two-phase flow heat transfer characteristics and security of the evaporative cooling technology are provided, the technology's advantage and feasibility are discussed.
Di Hu,Hui Zou,Tao Han,Junze Xie,Nannan Dai,Liling Zhuo,Jianhong Gu,Jianchun Bian,Yan Yuan,Xuezhong Liu,Zongping Liu 대한수의학회 2016 Journal of Veterinary Science Vol.17 No.1
Gap junctions mediate direct communication between cells; however, toxicological cascade triggered by nonessential metals can abrogate cellular signaling mediated by gap junctions. Although cadmium (Cd) is known to induce apoptosis in organs and tissues, the mechanisms that underlie gap junction activity in Cd-induced apoptosis in BRL 3A rat liver cells has yet to be established. In the present study, we showed that Cd treatment decreased the cell index (a measure of cellular electrical impedance) in BRL 3A cells. Mechanistically, we found that Cd exposure decreased expression of connexin 43 (Cx43), increased expression of p-Cx43 and elevated intracellular free Ca2+ concentration, corresponding to a decrease in gap junctional intercellular communication. Gap junction blockage pretreatment with 18b-glycyrrhizic acid (GA) promoted Cd-induced apoptosis, involving changes in expression of Bax, Bcl-2, caspase-3 and the mitochondrial transmembrane electrical potential (Δym). Additionally, GA was found to enhance ERK and p38 activation during Cd-induced activation of mitogen-activated protein kinases, but had no significant effect on JNK activation. Our results indicated the apoptosis-related proteins and the ERK and p38 signaling pathways may participate in gap junction blockage promoting Cd-induced apoptosis in BRL 3A cells.