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JeongHoon Park,Jae Young, Cho,Jung Sook Yeom,Jin Su Jun,Ji Sook Park,Eun Sil Park,Ji Hyun Seo,Jae Young Lim,Chan-Hoo Park,Hyang-Ok Woo The Korean Society of Pediatric Gastroenterology 2023 Pediatric gastroenterology, hepatology & nutrition Vol.26 No.3
Purpose: The starting time for probiotic supplementation in preterm infants after birth varies widely. This study aimed to investigate the optimal time for initiating probiotics to reduce adverse outcomes in preterm or very low birth weight (VLBW) infants. Methods: Medical records of preterm infants born at a gestational age (GA) of <32 weeks or VLBW infants in 2011-2020 were reviewed respectively. The infants who received Saccharomyces boulardii probiotics within 7 days of birth were grouped into an early introduction (EI) group, and those who received supplemented probiotics after 7 days of birth were part of the late introduction (LI) group. Clinical characteristics were compared between the two groups and analyzed statistically. Results: A total of 370 infants were included. The mean GA (29.1 weeks vs. 31.2 weeks, p<0.001) and birth weight (1,235.9 g vs. 1491.4 g, p<0.001) were lower in the LI group (n=223) than in the EI group. The multivariate analysis indicated that factors affecting the LI of probiotics were GA at birth (odds ratio [OR], 1.52; p<0.001) and the enteral nutrition start day (OR, 1.47; p<0.001). The late probiotic introduction was associated with a risk of late-onset sepsis (OR, 2.85; p=0.020), delayed full enteral nutrition (OR, 5.44; p<0.001), and extrauterine growth restriction (OR, 1.67; p=0.033) on multivariate analyses after adjusting for GA. Conclusion: Early supplementation of probiotics within a week after birth may reduce adverse outcomes among preterm or VLBW infants.
Implementation of an Integrated Controller for a Robot Hand Base on a Vehicle Communication System
JeongHoon Park,Chan-Woo Moon 보안공학연구지원센터 2014 International Journal of Control and Automation Vol.7 No.11
Intelligent robots commonly adopt Network-Based Control Systems (NBCS). For control of a robot hand or a robot head, many sensor data and control commands need to be transmitted in real time, so they have necessitated communication systems with high transmission capability. Traditionally, CAN is widely used for intelligent robots, but it presents bandwidths that are too low for the control of a high performance system. In this paper, the relation between the transmission period and the performance of a multi-motor control system is analyzed, and a multi-motor control system for a human robot hand is implemented based on the FlexRay communication system, which is an emerging in-vehicle communication network system. FlexRay parameter optimization for a robot system is studied, and a real position control test is conducted, to validate the implemented system.
Park Sang Won,김성헌,Park Jeonghoon,장재원,Kim SangYun 대한치매학회 2020 Dementia and Neurocognitive Disorders Vol.19 No.4
Background and Purpose: To identify biomarkers for prediction of the progression to dementia in mild cognitive impairment (MCI) patients, evaluation of brain structure changes has been validated by a comprehensive visual grading scale (CVRS) through magnetic resonance imaging (MRI). In this study, we specifically elucidated for the cognitive change of MCI patients classified based on AT(N) pathological status classification during the follow-up period of 3 years through the CVRS. Methods: The 301 patients with initial MCI visited at least once for follow-up period. The data used in this study were obtained from the Alzheimer's disease (AD) Neuroimaging Initiative study. Brain atrophy was assessed by CVRS using MRI. AT(N) profiles were classified by cerebrospinal fluid abnormality. Based on the AT(N) assessment, all individuals in this study were divided into 3 groups (normal state biomarker, suspected non-Alzheimer's pathology [SNAP], or Alzheimer's continuum). The cox regression was used to analyze the hazard ratios of CVRS for progression to dementia. Results: Sixty-three progressed and 238 remained stable to dementia and the CVRS (mean±standard deviation) had significant difference between progressive MCI and stable MCI (p<0.001). Univariate and multivariate cox regression results (p<0.001) showed the independence of initial CVRS as a predictor for the progression to dementia. Moreover, comparing the classified AT(N) pathology group, SNAP and AD, effectiveness of CVRS as a predictor was verified only in Alzheimer's continuum. Conclusions: The initial CVRS score as a predictor of dementia progression was independently validated at the stage of Alzheimer's progression among AT(N) pathologically differentiated MCI.
Regulation of dendritic arborization by BCR Rac1 GTPase-activating protein, a substrate of PTPRT.
Park, A-Reum,Oh, Daeyoung,Lim, So-Hee,Choi, Jeonghoon,Moon, Jeonghee,Yu, Dae-Yeol,Park, Sung Goo,Heisterkamp, Nora,Kim, Eunjoon,Myung, Pyung-Keun,Lee, Jae-Ran Cambridge University Press 2012 Journal of cell science Vol.125 No.19
<P>Dendritic arborization is important for neuronal development as well as the formation of neural circuits. Rac1 is a member of the Rho GTPase family that serve as regulators of neuronal development. Breakpoint cluster region protein (BCR) is a Rac1 GTPase-activating protein that is abundantly expressed in the central nervous system. Here, we show that BCR plays a key role in neuronal development. Dendritic arborization and actin polymerization were attenuated by overexpression of BCR in hippocampal neurons. Knockdown of BCR using specific shRNAs increased the dendritic arborization as well as actin polymerization. The number of dendrites in null mutant BCR(-/-) mice was considerably increased compared with that in wild-type mice. We found that the function of the BCR GTPase-activating domain could be modulated by protein tyrosine phosphatase receptor T (PTPRT), which is expressed principally in the brain. We demonstrate that tyrosine 177 of BCR was the main target of PTPRT and the BCR mutant mimicking dephosphorylation of tyrosine 177 alleviated the attenuation of dendritic arborization. Additionally the attenuated dendritic arborization found upon BCR overexpression was relieved upon co-expression of PTPRT. When PTPRT was knocked down by a specific shRNA, the dendritic arborization was significantly reduced. The activity of the BCR GTPase-activating domain was modulated by means of conversions between the intra- and inter-molecular interactions, which are finely regulated through the dephosphorylation of a specific tyrosine residue by PTPRT. We thus show conclusively that BCR is a novel substrate of PTPRT and that BCR is involved in the regulation of neuronal development via control of the BCR GTPase-activating domain function by PTPRT.</P>
Quantum nonsignaling-assisted zero-error classical capacity of qubit channels
Park, Jeonghoon,Lee, Soojoon American Physical Society 2016 Physical Review A Vol.93 No.3
<P>In this paper, we explicitly evaluate the one-shot quantum nonsignaling-assisted zero-error classical capacities M-0(QNS) for qubit channels. In particular, we show that for nonunital qubit channels M-0(QNS) = 0, which implies that in the one-shot setting nonunital qubit channels cannot transmit any information with zero probability of error even when assisted by quantum nonsignaling correlations. Furthermore, we show that for qubit channels M-0(QNS) equals the one-shot entanglement-assisted zero-error classical capacity. This means that for a single use of a qubit channel quantum nonsignaling correlations are not more powerful than shared entanglement.</P>