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Evolution of TCP in High Speed Networks
Ivan Petrov,Toni Janevski 보안공학연구지원센터 2015 International Journal of Future Generation Communi Vol.8 No.2
TCP congestion control protocols have low performances in high speed wide area networks mainly because their slow response with large congestion windows. This TCP behavior has initiated new design phase of alternative protocols that provide improved traffic utilization in high bandwidth delay product networks. The paper presents survey of various high speed sender side congestion control proposals that preserve the fundamental host to host principle. Solutions focus on variety of problems occurring in high speed environment with intention to eliminate congestion collapses and to ensure effective resource utilization. Internet data transfer does not depend only on that how TCP will utilize the network capacity, we have to stress that TCP must cooperate with existing transmitting data protocols through the same network in order to assure fair resource sharing. Part of the paper scope are state of the art high speed TCP proposals, we explore their congestion control techniques, strengths, weaknesses and we try to detect future TCP development possibilities.
Time-optimal Velocity Planning Along Predefined Path for Static Formations of Mobile Robots
Toni Petrinic,Mišel Brezak,Ivan Petrovic 제어·로봇·시스템학회 2017 International Journal of Control, Automation, and Vol.15 No.1
This paper is concerned with the problem of finding a time-optimal velocity profile along the predefinedpath for static formations of mobile robots in order to traverse the path in shortest time and to satisfy, for eachmobile robot in the formation, velocity, acceleration, tip over and wheel slip prevention constraints. Time-optimalvelocity planning is achieved using so called bang-bang control where minimum and maximum accelerations ofthe formation are alternating. The developed trajectory planning algorithm is demonstrated on the formation ofdifferential drive mobile robots.
Tm,Ho:KLu(WO4)2 laser mode-locked near 2 μm by single-walled carbon nanotubes.
Aleksandrov, Veselin,Gluth, Alexander,Petrov, Valentin,Buchvarov, Ivan,Choi, Sun Young,Kim, Mi Hye,Rotermund, Fabian,Mateos, Xavier,D?az, Francesc,Griebner, Uwe Optical Society of America 2014 Optics express Vol.22 No.22
<P>We demonstrate passive mode-locking of a Tm,Ho-codoped crystalline laser operating on the Ho3+-ion transition I-5(7)-->I-5(8) near 2 mu(m) using a single-walled carbon nanotube saturable absorber. The Tm,Ho:KLu(WO4)(2) laser emits nearly transform-limited pulses with duration of 2.8 ps at a repetition rate of 91 MHz. The output power amounts to 97 mW. (C) 2014 Optical Society of America</P>
( Predrag Sikiric ),( Ki-baik Hahm ),( Alenka Boban Blagaic ),( Ante Tvrdeic ),( Katarina Horvat Pavlov ),( Andrea Petrovic ),( Antonio Kokot ),( Slaven Gojkovic ),( Ivan Krezic ),( Domagoj Drmic ),( 대한간학회 2020 Gut and Liver Vol.14 No.2
We reviewed again the significance of the stable gastric pentadecapeptide BPC 157 as a likely mediator of Robert’s stomach cytoprotection/adaptive cytoprotection and organoprotection and as novel mediator of Selye’s stress coping response to reestablish homeostasis. Specific points of BPC 157 therapy and the original concept of Robert’s cytoprotection/adaptive cytoprotection/organoprotection are discussed, including the beneficial effects of BPC 157. First, BPC 157 protects stomach cells and maintains gastric integrity against various noxious agents (Robert’s killing cell by contact) and is continuously present in the gastric mucosa and gastric juice. Additionally, BPC 157 protects against the adverse effects of alcohol and nonsteroidal anti-inflammatory drugs on the gastric epithelium and other epithelia, that is, skin, liver, pancreas, heart (organoprotection), and brain, thereby suggesting its use in wound healing. Additionally, BPC 157 counteracts gastric endothelial injury that precedes and induces damage to the gastric epithelium and generalizes “gastric endothelial protection” to protection of the endothelium of other vessels (thrombosis, prolonged bleeding, and thrombocytopenia). BPC 157 also has an effect on blood vessels, resulting in vessel recruitment that circumvents vessel occlusion and the development of additional shunting and rapid bypass loops to rapidly reestablish the integrity of blood flow (ischemic/reperfusion colitis, duodenal lesions, cecal perforation, and inferior vena caval occlusion). Lastly, BPC 157 counteracts tumor cachexia, muscle wasting, and increases in pro-inflammatory/procachectic cytokines, such as interleukin-6 and tumor necrosis factor-α, and significantly corrects deranged muscle proliferation and myogenesis through changes in the expression of FoxO3a, p-AKT, p-mTOR, and p-GSK-3β (mitigating cancer cachexia). (Gut Liver 2020;14:153-167)