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A. F. Abd El‑Rehim,D. M. Habashy,H. Y. Zahran,H. N. Soliman 대한금속·재료학회 2021 METALS AND MATERIALS International Vol.27 No.10
An artificial neural network (ANN) model was used for the simulation and prediction of the mechanical properties ofSn-9Zn-Cu solder alloys. Sn-9Zn-Cu solder alloys containing different Cu contents (0, 1, 2, 3, 4 and 5 wt%) were successfullyprepared by permanent mold casting. The specimens were heated in a protective argon atmosphere at 433 K for 24 h,followed by water quenching at 298 K. Finally, the heat-treated samples were aged at 373 K for different time intervals (ta = 2,4, 8, 16 and 32 h), followed by water quenching at 298 K. The phases present in the current alloys were detected by X-raydiffraction analysis. For morphological characterization, a scanning electron microscope operated at 20 kV was tilized. Themechanical properties of the samples were studied using hardness measurements. The variations in the hardness data withincreasing aging time were determined based on the structural transformations that take place in the alloys. The ANN modelwas applied to the hardness measurements to simulate and predict the Vickers hardness of Sn-Zn-Cu alloys with mean squareerror values equal 9.55E-06 and 9.44E-06 for training and validation data respectively after 281 epochs. The simulated andpredicted results were consistent with the experimental results.
E1/E2 of Hepatitis C Virus Genotype-4 and Apoptosis
Zekri, Abdel-Rahman N,Sobhy, Esraa,Hussein, Nehal,Ahmed, Ola S,Hussein, Amira,Shoman, Sahar,Soliman, Amira H,El-Din, Hanaa M Alam Asian Pacific Journal of Cancer Prevention 2016 Asian Pacific journal of cancer prevention Vol.17 No.7
Several studies have addressed the possible role of hepatitis C virus genotype-4 (HCV GT4) in apoptosis. However, this still not fully understood. In the current study a re-constructed clone of E1/E2 polyprotein region of the HCV GT4 was transfected into the Huh7 cell line and a human apoptotic PCR array of 84 genes was used to investigate its possible significance for apoptosis. Out of the 84 genes, only 35 showed significant differential expression, 12 genes being up-regulated and 23 down-regulated. The highest-up regulated genes were APAF1 (apoptotic peptidase-activating factor 1), BID (BH3 interacting domain death agonist) and BCL 10 (B-cell CLL/lymphoma protein 10) with fold regulation of 33.2, 30.1 and 18.9, respectively. The most down-regulated were FAS (TNF receptor super family), TNFRSF10B (tumor necrosis factor receptor super-family member 10b) and FADD (FAS-associated death domain) with fold regulation of -30.2, -27.7 and -14.9, respectively. These results suggest that the E1/E2 proteins may be involved in HCV-induced pathogenesis by modulating apoptosis through the induction of the intrinsic apoptosis pathway and disruption of the BCL2 gene family.