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RISS 인기검색어
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- 저자 : Gollamudi, Sudheer (검색결과 2 건)
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Linear versus Non-linear Interference Cancellation
Buehrer, R.Michael,Nicoloso, Steven P.,Gollamudi, Sridhar The Korean Institute of Communications and Informa 1999 Journal of communications and networks Vol.1 No.2
In this paper we compare linear and non-linear inter-ference cancellation for systems employing code division multi-ple access (CDMA) techniques. Specifically, we examine linear and non-linear parallel interference cancellation(also called multi-stage cancellation) in relationship to other multiuser detection al-gorithms. We show the explicit relationship between parallel inter-ference cancellation and the decorrelator (or direct matrix inver-sion). This comparison gives insight into the performance of paral-lel interference cancellation (PIC) and leads to vetter approaches. We also show that non-linear PIC approaches with explicit chan-nel setimation can provide performance improvement over linear PIC, especially when using soft non-linear symbol estimates. The application of interference cancellation to non-linear modulation techniques is also presented along with a discussion on minimum mean-squared error(MMSE) symbol estimation techniques. These are shown to further improve the performance of parallel cancella-tion.
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T Lymphocytes Negatively Regulate Lymph Node Lymphatic Vessel Formation
Kataru, Raghu P.,Kim, Honsoul,Jang, Cholsoon,Choi, Dong Kyu,Koh, Bong Ihn,Kim, Minah,Gollamudi, Sudheer,Kim, Yun-Keun,Lee, Seung-Hyo,Koh, Gou Young Elsevier 2011 Immunity Vol.34 No.1
<P><B>Summary</B></P><P>Lymph node lymphatic vessels (LNLVs) serve as a conduit to drain antigens from peripheral tissues to within the lymph nodes. LNLV density is known to be positively regulated by vascular endothelial growth factors secreted by B cells, macrophages, and dendritic cells (DCs). Here, we show that LNLV formation was negatively regulated by T cells. In both steady and inflammatory states, the density of LNLVs was increased in the absence of T cells but decreased when T cells were restored. Interferon-γ secretion by T cells suppressed lymphatic-specific genes in lymphatic endothelial cells and consequently caused marked reduction in LNLV formation. When T cells were depleted, recruitment of antigen-carrying DCs to LNs was augmented, reflecting a compensatory mechanism for antigen presentation to T cells through increased LNLVs. Thus, T cells maintain the homeostatic balance of LNLV density through a negative paracrine action of interferon-γ.</P> <P><B>Graphical Abstract</B></P><P><ce:figure id='dfig1'></ce:figure></P><P><B>Highlights</B></P><P>► Lymphatic vessels (LVs) are absent in the T cell zone of lymph nodes (LNs) ► T cells reduce LNLV density by secreting IFN-<B>γ</B> ► IFN-<B>γ</B> downregulates lymphatic-specific genes ► IFN-<B>γ</B> prevents lymphangiogenesis</P>
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