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RISS 인기검색어
- 검색키워드
- 저자 : Encabo, Jaymee R (검색결과 2 건)
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Suppression of two tungro viruses in rice by separable traits originating from cultivar Utri Merah.
Encabo, Jaymee R,Cabauatan, Pepito Q,Cabunagan, Rogelio C,Satoh, Kouji,Lee, Jong-Hee,Kwak, Do-Yeon,De Leon, Teresa B,Macalalad, Reena Jesusa A,Kondoh, Hiroaki,Kikuchi, Shoshi,Choi, Il-Ryong APS Press 2009 Molecular plant-microbe interactions Vol.22 No.10
<P>Rice tungro disease (RTD) is caused by Rice tungro spherical virus (RTSV) and Rice tungro bacilliform virus (RTBV) transmitted by green leafhoppers. Rice cv. Utri Merah is highly resistant to RTD. To define the RTD resistance of Utri Merah, near-isogenic lines (NIL, BC(5) or BC(6)) developed from Utri Merah and susceptible cv. Taichung Native 1 (TN1) were evaluated for reactions to RTSV and RTBV. TW16 is an NIL (BC(5)) resistant to RTD. RTBV was able to infect both TN1 and TW16 but the levels of RTBV were usually significantly lower in TW16 than in TN1. Infection of RTSV was confirmed in TN1 by a serological test but not in TW16. However, the global gene-expression pattern in an RTSV-resistant NIL (BC(6)), TW16-69, inoculated with RTSV indicated that RTSV can also infect the resistant NIL. Infection of RTSV in TW16 was later confirmed by reverse-transcription polymerase chain reaction but the level of RTSV was considerably lower in TW16 than in TN1. Examination for virus accumulation in another NIL (BC(6)), TW16-1029, indicated that all plants of TW16-1029 were resistant to RTSV, whereas the resistance to RTBV and symptom severity were segregating among the individual plants of TW16-1029. Collectively, these results suggest that RTD resistance of Utri Merah involves suppression of interacting RTSV and RTBV but the suppression trait for RTSV and for RTBV is inherited separately.</P>
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( Kristian Anteolin Punzalan ),( Regidor Encabo ) 대한내과학회 2014 대한내과학회 추계학술대회 Vol.2014 No.1
Background: Infl ammation plays a key role in the development of coronary atherosclerosis and acute coronary syndrome (ACS). Recent studies suggested that specifi c subtypes of leukocytes can be used as predictor in assessing cardiovascular risk in which the value is higher when neutrophil to lymphocyte ratio (NLR) is used. Objective: To evaluate NLR on admission as an independent predictor of in-hospital mortality among ACS patients. Methods: This study retrospectively evaluated patients admitted and diagnosed with ACS at Cardinal Santos Medical Center. Chart records of 280 ACS patients were screened and evaluated. Patients who have history of percutaneous coronary intervention, malignancy, concurrent infection, recent surgery, current use of immunosuppressors and anti-infl ammatory drugs were excluded from the study. Patients were stratifi ed into 2 groups based on a cut-off of 3.5 NLR done from previous studies. NLR was labeled as Low if <3.5 and High if >3.5. The major outcome of this study was in-hospital mortality. Results: Correlation analysis showed a signifi cant positive correlation between demographic profi les, NLR and mortality among STEMI (P-value 0.038) and NSTEMI (P-value 0.041). In the univariate logistic analysis, signifi cant predictors of in-hospital mortality were sex (OR 12.091, P-value 0.002) and NLR (OR 10.909, P-value 0.050) among STEMI while dyslipidemia (OR 2.316, P-value 0.046) and NLR (OR 5.741, P-value 0.019) among NSTEMI. On multivariate analysis, NLR was found to be an independent predictor of in-hospital mortality for both STEMI (OR 10.234, P-value 0.048) and NSTEMI (OR 5.354, P-value 0.021). Conclusions: NLR on admission, is an independent predictor of in-hospital mortality among patients diagnosed with STEMI and NSTEMI but not UA.
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