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Generalized Stability Criterion for Multi-module Distributed DC System
Liu, Fangcheng,Liu, Jinjun,Zhang, Haodong,Xue, Danhong The Korean Institute of Power Electronics 2014 JOURNAL OF POWER ELECTRONICS Vol.14 No.1
The stability issues of a multi-module distributed DC power system without current-sharing loop are analyzed in this study. The physical understanding of the terminal characteristics of each sub-module is focused on. All the modules are divided into two groups based on the different terminal property types, namely, impedance (Z) and admittance (Y) types. The equivalent circuits of each group are established to analyze the stability issues, and the mathematical equations of the equivalent circuits are derived. A generalized criterion for multi-module distributed systems is proposed based on the stability criterion in a cascade system. The proposed criterion is independent of the power flow direction.
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Generalized Stability Criterion for Multi-module Distributed DC System
Fangcheng Liu,Jinjun Liu,Haodong Zhang,Danhong Xue 전력전자학회 2014 JOURNAL OF POWER ELECTRONICS Vol.14 No.1
The stability issues of a multi-module distributed DC power system without current-sharing loop are analyzed in this study. The physical understanding of the terminal characteristics of each sub-module is focused on. All the modules are divided into two groups based on the different terminal property types, namely, impedance (Z) and admittance (Y) types. The equivalent circuits of each group are established to analyze the stability issues, and the mathematical equations of the equivalent circuits are derived. A generalized criterion for multi-module distributed systems is proposed based on the stability criterion in a cascade system. The proposed criterion is independent of the power flow direction.
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Sirui Zhu,Xiaoli Liu,Mei Xue,Yu Li,Danhong Cai,Shijun Wang,Liang Zhang 고려인삼학회 2021 Journal of Ginseng Research Vol.45 No.2
Background: Acute promyelocytic leukemia (APL) is a hematopoietic malignancy driven by promyelocytic leukemia―retinoic acid receptor A (PML-RARA) fusion gene. The therapeutic drugs currently used to treat APL have adverse effects. 20(S)-ginsenoside Rh2 (GRh2) is an anticancer medicine with high effectiveness and low toxicity. However, the underlying anticancer mechanisms of GRh2-induced PML-RARA degradation and apoptosis in human APL cell line (NB4 cells) remain unclear. Methods: Apoptosis-related indicators and PML-RARA expression were determined to investigate the effect of GRh2 on NB4 cells. Z-VAD-FMK, LY294002, and C 87, as inhibitors of caspase, and the phosphatidylinositol 3-kinase (PI3K) and tumor necrosis factor-α (TNF-α ) pathways were used to clarify the relationship between GRh2-induced apoptosis and PML-RARA degradation. Results: GRh2 dose- and time-dependently decreased NB4 cell viability. GRh2-induced apoptosis, cell cycle arrest, and caspase3, caspase8, and caspase9 activation in NB4 cells after a 12-hour treatment. GRh2-induced apoptosis in NB4 cells was accompanied by massive production of reactive oxygen species, mitochondrial damage and upregulated Bax/Bcl-2 expression. GRh2 also induced PML/PML-RARA degradation, PML nuclear bodies formation, and activation of the downstream p53 pathway in NB4 cells. Z-VAD-FMK inhibited caspase activation and significantly reversed GRh2-induced apoptosis and PML-RARA degradation. GRh2 also upregulated TNF-a expression and inhibited Akt phosphorylation. LY294002, an inhibitor of the PI3K pathway, enhanced the antitumor effects of GRh2, and C 87, an inhibitor of the TNF-α pathway, reversed NB4 cell viability, and GRh2-mediated apoptosis in a caspase-8-dependent manner. Conclusion: GRh2 induced caspase-dependent PML-RARA degradation and apoptosis in NB4 cells via the Akt/Bax/caspase9 and TNF-α/caspase8 pathways.
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