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- 저자 : Chenxi Ruan (검색결과 2 건)
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Mechanical Properties and Microstructure of Polypropylene Fiber Reinforced Cement Mortar Soil
Bo Ruan,Hui Ding,Jidong Teng,Wei Deng,Shilong Zheng,Chenxi Ruan 대한토목학회 2021 KSCE JOURNAL OF CIVIL ENGINEERING Vol.25 No.6
A series of unconfined compressive strength tests and flexural strength tests are carried out to evaluate the improved effect of polypropylene fiber on the defects of cement mortar soil. The following factors, including the fiber content, cement content, sand content and curing age, are studied to investigate the influences on the mechanical properties and microstructure of the samples. The results show that the unconfined compressive strength (UCS), residual strength and flexural strength of the fiber reinforced cement mortar soil (FRCMS) substantially increase with increasing fiber content. The peak strain and ratio of the flexural-compression strength (Rfcs) of the FRCMS first increase and then decrease with an increase in fiber content,and the optimal fiber content is 3.5%. The brittleness index of the FRCMS is found to be inversely proportional to fiber content. The results suggest that the addition of an appropriate amount of fibers can substantially improve the plasticity and lateral stress capacity of the FRCMS. The strength of the FRCMS improves with the increase in cement content, sand content and curing age within a certain range. The microstructure of the FRCMS are analyzedby scanning electron microscopy (SEM) tests.
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Expression profiling identified IL-8 as a regulator of homotypic cell-in-cell formation
( Banzhan Ruan ),( Chenxi Wang ),( Ang Chen ),( Jianqing Liang ),( Zubiao Niu ),( You Zheng ),( Jie Fan ),( Lihua Gao ),( Hongyan Huang ),( Xiaoning Wang ),( Qiang Sun ) 생화학분자생물학회(구 한국생화학분자생물학회) 2018 BMB Reports Vol.51 No.8
Homotypic cell-in-cell (CIC) structures forming between cancer cells were proposed to promote tumor evolution via entosis, a nonapoptotic cell death process. However, the mechanisms underlying their formation remained poorly understood. We performed a microarray analysis to identify genes associated with homotypic CIC formation. Cancer cells differing in their ability to form homotypic CIC structures were selected for the study. Association analysis identified 73 probe sets for 62 candidate genes potentially involved in CIC formation. Among them, twenty-one genes were downregulated while 41 genes were upregulated. Pathway analysis identified a gene interaction network centered on IL-8, which was upregulated in high CIC cells. Remarkably, CIC formation was significantly inhibited by IL-8 knockdown and enhanced upon recombinant IL-8 treatment, which correlated with altered cell-cell adhesion and expression of adhesive molecules such as P-cadherin and γ-catenin. Together, our work identified IL-8 as a positive regulator of homotypic CIC formation via enhancing intercellular adhesion. [BMB Reports 2018; 51(8): 412-417]
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