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난소의 생식세포 기원성 종양세포에서의 c-myc, c-H-res 관련 단백 및 표피성장인자에 관한 면역조직학적 연구
현동관,신용칠,염범우 고려대학교 의과대학 1993 고려대 의대 잡지 Vol.30 No.1
The causes of malignant tumor include chemical agents, irradiation, viral infection and others. Among them, oncogenic viral infection clarified the pathogenesis of the neoplasm. Oncogenic virus has viral oncogene which has transforming activity of cells when it is incoorperated into the DNA of the host cells. These DNA of transformed cells also have the activity of transformation of recipient cells and production of infectious virus, in culture. The normal cells may have the DNA showing homologous or similar structure of viral oncogene. These cellular oncogenes may be activated by gene mutation chromosomal translocation, deletion and promotor insertion. In these cases, abnormal oncogene related proteins are produced, such as ras P21 and P62-c-myc proteins. There are many reports that oncogene related proteins are detected in variable carcinomas, sarcomas, and leukemia-lymphomas. However, there are few reports on germ cell tumors of ovary So we stained rasP21 (ras gene related protein), P62-c-myc (c-myc related protein) and EGF antisera on various ovarian germ cell tumors with the method of avidin-biotin peroxidase complex. The results are as followings, 1. The positive reaction of all three antisera against the oncoprotein is the highest in cystic teratoma. 2. In cystic teratomas, the squamous cells and sebaceous glands show the strongest positive reaction and chondroid and glial tissue shows the weakest positive reaction. 3. c-myc antiserum shows the strongest positive reaction in the most tumors comparing with that of other two antisera.