http://chineseinput.net/에서 pinyin(병음)방식으로 중국어를 변환할 수 있습니다.
변환된 중국어를 복사하여 사용하시면 됩니다.
H+-ATPase Pump 결손을 가진 원위부 신세관성 산중이 전신성 홍반성 낭창의 첫번째 증세로 나타난
송영준 ( Young Jun Song ),김정은 ( Jung Eun Kim ),이종우 ( Jong Woo Lee ),이동훈 ( Dong Hun Lee ),조현경 ( Hyeon Kyeong Cho ),신규택 ( Gyu Tae Shin ),김흥수 ( Heung Soo Kim ),임현이 ( Hyun Ee Yim ),김진 ( Jin Kim ),임선우 ( Sun W 대한신장학회 2003 Kidney Research and Clinical Practice Vol.22 No.1
Systemic lupus erythematosus (SLE) is a multi-system disease with marked variability in its manifestation. Tubulointerstitial involvement is well recognized in SLE. But usually the tubular dysfunction is latent and usually presents after diagnosis of SLE. We report a 20 years old female whose initial symptom of SLE was distal renal tubular acidosis (RTA). She presented with severe muscle weakness at emergency room with laboratory fingding consistent with distal RTA. After several months she developed fever, arthritis, serologic fingding which was compatible to diagnose SLE. We report a case whose initial symptom of SLE had been distal RTA. (Korean J Nephrol 2003;22(1):148-155)
임선교 ( Sun Gyo Lim ),김정은 ( Jeong Eun Kim ),이종우 ( Jong Woo Lee ),이동훈 ( Dong Hun Lee ),임승관 ( Seung Kwan Lim ),박인휘 ( In Whee Park ),조현경 ( Hyeon Kyeong Cho ),김흥수 ( Heung Soo Kim ),신규택 ( Gyu Tae Shin ),임현이 대한신장학회 2003 Kidney Research and Clinical Practice Vol.22 No.1
Anti-glomerular basement membrane antibody mediated rapidly progressiv glomerulonephritis is a rare autoimmune disease. It is characterized by acute renal failure and crescentic glomeruli with linear immune deposits along glomerular basement membrane mediated by anti-GBM antibodies. We report a case of a sixty-years-old man with generalized edema and hematuria. On admission, BUN/Creatinine was 118/19.6 ng/dL, Hb was 10.2 g/bL. On urinalysis, protein was 3+, and many RBCs were found. Renal biopsy specimen which contained 8 glomeruli showed active cellular crescent formation in all glomeruli. On immunofluorescent staining specimen, there were 4 glomeruli which showed strong IgG linear staining along the glomerular basement membrane and mild C3 & Ciq deposit along the capillary walls. The titer of anti-GBM antibody was 123 EU by ELISA (normal : <10 EU). We treated with high dose of corticosteroid and plasmapheresis, but renal function was not recovered even after 3 months of hemodialysis.