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Zhao, Wei,E, Lei,Ya, Jing,Liu, Zhifeng,Zhou, Heping Korean Chemical Society 2012 Bulletin of the Korean Chemical Society Vol.33 No.7
Perovskite structured barium titanate particles ($BaTiO_3$) platelets were synthesized by molten salt synthesis and topochemical microcrystal conversion. As the precursors of $BaTiO_3$, plate-like $BaBi_4Ti_4O_{15}$ particles were first synthesized by the reaction of $Bi_4Ti_3O_{12}$, $BaCO_3$, and $TiO_2$ at $1080^{\circ}C$ for 3 h in $BaCl_2$-KCl molten salt. After the topochemical reactions, layer-structured $BaBi_4Ti_4O_{15}$ particles transformed to the perovskite $BaTiO_3$ platelets. $BaTiO_3$ particles with thickness of approximately $0.5{\mu}m$ and a length of $10-15{\mu}m$ retained the morphology feature of the $BaBi_4Ti_4O_{15}$ precursor. For <001> $Pb(Mg_{1/3}Nb_{2/3})O_3-32.5PbTiO_3$ (PMNT)-5 wt % PbO piezoelectric ceramics textured with 5 vol % of $BaTiO_3$ templates, the Lotgering factor reached 0.82, and $d_{33}$ was 870 pC/N.
Wei Zhao,Lei E,Jing Ya,Zhifeng Liu,Heping Zhou 대한화학회 2012 Bulletin of the Korean Chemical Society Vol.33 No.7
Perovskite structured barium titanate particles (BaTiO3) platelets were synthesized by molten salt synthesis and topochemical microcrystal conversion. As the precursors of BaTiO3, plate-like BaBi4Ti4O15 particles were first synthesized by the reaction of Bi4Ti3O12, BaCO3, and TiO2 at 1080 °C for 3 h in BaCl2-KCl molten salt. After the topochemical reactions, layer-structured BaBi4Ti4O15 particles transformed to the perovskite BaTiO3 platelets. BaTiO3 particles with thickness of approximately 0.5 μm and a length of 10-15 μm retained the morphology feature of the BaBi4Ti4O15 precursor. For <001> Pb(Mg1/3Nb2/3)O3-32.5PbTiO3 (PMNT)-5 wt % PbO piezoelectric ceramics textured with 5 vol % of BaTiO3 templates, the Lotgering factor reached 0.82, and d33 was 870 pC/N.
Jing Ya,Li An,Zhifeng Liu,Lei E,Wei Zhao,Dan Zhao,Chengcheng Liu 한국화학공학회 2012 Korean Journal of Chemical Engineering Vol.29 No.6
TiO2 nanowire/nanotube electrodes were synthesized by anodization of titanium foils in ethylene glycol solution containing 0.5 wt% NH4F and 1 wt% water at 60 V for 6 h. The microstructure and morphology of the asprepared electrodes were investigated by XRD and SEM. A possible formation mechanism and oxidation parameters of nanocomposite structure were discussed. The relationship between structural characteristics of TiO2 nanowire/nanotube electrodes and its photoelectrochemical characterization were evaluated by electrochemical analyzer and photocatalytic degradation of methylene blue (MB) solution. Furthermore, these TiO2 nanowire/nanotube electrodes promoted the photoelectrochemical characterization due to the larger surface areas, enhanced light harvesting and electron transport rate. The results show that photocurrent density of 1.44mA/cm2 and photocatalytic degradation of 95.51% was achieved for TiO2 nanowire/nanotube electrodes, which were 0.55mA/cm2 and 20.52% higher than the TiO2 nanotube electrodes under a similar condition, respectively.
Comparison of Microbial Diversity and Composition in the Jejunum and Colon of Alcohol-Dependent Rats
( Yang Fan ),( Zhao Ya-e ),( Wei Ji-dong ),( Lu Yu-fan ),( Zhang Ying ),( Sun Ya-lun ),( Ma Meng-yu ),( Zhang Rui-ling ) 한국미생물생명공학회(구 한국산업미생물학회) 2018 Journal of microbiology and biotechnology Vol.28 No.11
Alcohol dependence is a global public health problem, yet the mechanisms of alcohol dependence are incompletely understood. The traditional view has been that ethanol alters various neurotransmitters and their receptors in the brain and causes the addiction. However, an increasing amount of experimental evidence suggests that gut microbiota also influence brain functions via gut-to-brain interactions, and may therefore induce the development of alcohol use disorders. In this study, a rat model of alcohol dependence and withdrawal was employed, the gut microbiota composition was analyzed by high-throughput 16S rRNA gene sequencing, and the metagenome function was predicted by PICRUSt software. The results suggested that chronic alcohol consumption did not significantly alter the diversity and richness of gut microbiota in the jejunum and colon, but rather markedly changed the microbiota composition structure in the colon. The phyla Bacteroidetes and eight genera including Bacteroidales S24-7, Ruminococcaceae, Parabacteroides, Butyricimonas, et al were drastically increased, however the genus Lactobacillus and gauvreauii in the colon were significantly decreased in the alcohol dependence group compared with the withdrawal and control groups. The microbial functional prediction analysis revealed that the proportions of amino acid metabolism, polyketide sugar unit biosynthesis and peroxisome were significantly increased in the AD group. This study demonstrated that chronic alcohol consumption has a dramatic effect on the microbiota composition structure in the colon but few effects on the jejunum. Inducement of colonic microbiota dysbiosis due to alcohol abuse seems to be a factor of alcohol dependence, which suggests that modulating colonic microbiota composition might be a potentially new target for treating alcohol addiction.
( Shuqin Ding ),( Yuliang Qu ),( Shaoqi Yang ),( Ya’e Zhao ),( Guangxian Xu ) 한국미생물생명공학회(구 한국산업미생물학회) 2019 Journal of microbiology and biotechnology Vol.29 No.6
Autophagy is crucial for immune defense against Mycobacterium tuberculosis (Mtb) infection. Mtb can evade host immune attack and survival within macrophages by manipulating the autophagic process. MicroRNAs (miRNAs) are small, non-coding RNAs that are involved in regulating vital genes during Mtb infection. The precise role of miRNAs in autophagy with the exits of Mtb remains largely unknown. In this study, we found miR-1958, a new miRNA that could regulate autophagy by interacting with 3'UTR of autophagy-related gene 5 (Atg5). In addition, Mtb infection triggered miR-1958 expression in RAW264.7 cells. What's more, miR- 1958 overexpression blocked autophagic flux by impairing the fusion of autophagosomes and lysosomes. Overexpression of miR-1958 reduced Atg5 expression and LC3 puncta while inhibition of miR-1958 brought an increase of Atg5 and LC3 puncta; the opposite results were observed in detection of p62. The survival of Mtb in RAW264.7 cells transfected with mimic of miR-1958 was enhanced. Taken together, our research demonstrated that a novel miR-1958 could inhibit autophagy by interacting with Atg5 and favored intracellular Mtb survival in RAW264.7 cells.