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      • Fast Jacket-Haar Transform with Any Size

        Liu, Guibo,Huang, Dazu,Luo, Dayong,Lei, Wang,Guo, Ying,Lee, Moonho Hindawi Limited 2015 Mathematical problems in engineering Vol.2015 No.-

        <P>Jacket-Haar transform has been recently generalized from Haar transform and Jacket transform, but, unfortunately, it is not available in a case where the lengthNis not a power of 2. In this paper, we have proposed an arbitrary-length Jacket-Haar transform which can be conveniently constructed from the 2-point generalized Haar transforms with the fast algorithm, and thus it can be constructed with any sizes. Moreover, it can be further extended with elegant structures, which result in the fast algorithms for decomposing. We show that this approach can be practically applied for the electrocardiogram (ECG) signal processing. Simulation results show that it is more efficient than the conventional fast Fourier transform (FFT) in signal processing.</P>

      • SCIESCOPUSKCI등재

        Ginsenoside compound K protects human umbilical vein endothelial cells against oxidized low-density lipoprotein-induced injury via inhibition of nuclear factor-κB, p38, and JNK MAPK pathways

        Lu, Shan,Luo, Yun,Zhou, Ping,Yang, Ke,Sun, Guibo,Sun, Xiaobo The Korean Society of Ginseng 2019 Journal of Ginseng Research Vol.43 No.1

        Background: Oxidized low-density lipoprotein (ox-LDL) causes vascular endothelial cell inflammatory response and apoptosis and plays an important role in the development and progression of atherosclerosis. Ginsenoside compound K (CK), a metabolite produced by the hydrolysis of ginsenoside Rb1, possesses strong anti-inflammatory effects. However, whether or not CK protects ox-LDL-damaged endothelial cells and the potential mechanisms have not been elucidated. Methods: In our study, cell viability was tested using a 3-(4, 5-dimethylthiazol-2yl-)-2,5-diphenyl tetrazolium bromide (MTT) assay. Expression levels of interleukin-6, monocyte chemoattractant protein-1, tumor necrosis factor-${\alpha}$, intercellular adhesion molecule-1, and vascular cell adhesion molecule-1 were determined by enzyme-linked immunosorbent assay and Western blotting. Mitochondrial membrane potential (${\Delta}{\Psi}m$) was detected using JC-1. The cell apoptotic percentage was measured by the Annexin V/ propidium iodide (PI) assay, lactate dehydrogenase, and caspase-3 expression. Apoptosis-related proteins, nuclear factor $(NF)-{\kappa}B$, and mitogen-activated protein kinases (MAPK) signaling pathways protein expression were quantified by Western blotting. Results: Our results demonstrated that CK could ameliorate ox-LDL-induced human umbilical vein endothelial cells (HUVECs) inflammation and apoptosis, $NF-{\kappa}B$ nuclear translocation, and the phosphorylation of p38 and c-Jun N-terminal kinase (JNK). Moreover, anisomycin, an activator of p38 and JNK, significantly abolished the anti-apoptotic effects of CK. Conclusion: These results demonstrate that CK prevents ox-LDL-induced HUVECs inflammation and apoptosis through inhibiting the $NF-{\kappa}B$, p38, and JNK MAPK signaling pathways. Thus, CK is a candidate drug for atherosclerosis treatment.

      • KCI등재

        A New Copyright Protection Scheme for Depth Map in 3D Video

        ( Zhaotian Li ),( Yuesheng Zhu ),( Guibo Luo ),( Biao Guo ) 한국인터넷정보학회 2017 KSII Transactions on Internet and Information Syst Vol.11 No.7

        In 2D-to-3D video conversion process, the virtual left and right view can be generated from 2D video and its corresponding depth map by depth image based rendering (DIBR). The depth map plays an important role in conversion system, so the copyright protection for depth map is necessary. However, the provided virtual views may be distributed illegally and the depth map does not directly expose to viewers. In previous works, the copyright information embedded into the depth map cannot be extracted from virtual views after the DIBR process. In this paper, a new copyright protection scheme for the depth map is proposed, in which the copyright information can be detected from the virtual views even without the depth map. The experimental results have shown that the proposed method has a good robustness against JPEG attacks, filtering and noise.

      • KCI등재

        Ginsenoside compound K protects human umbilical vein endothelial cells against oxidized low-density lipoprotein-induced injury via inhibition of nuclear factor-kB, p38, and JNK MAPK pathways

        Shan Lu,Yun Luo,Ping Zhou,Ke Yang,Guibo Sun,Xiaobo Sun 고려인삼학회 2019 Journal of Ginseng Research Vol.43 No.1

        Background: Oxidized low-density lipoprotein (ox-LDL) causes vascular endothelial cell inflammatory response and apoptosis and plays an important role in the development and progression of atherosclerosis. Ginsenoside compound K (CK), a metabolite produced by the hydrolysis of ginsenoside Rb1, possesses strong anti-inflammatory effects. However, whether or not CK protects ox-LDL-damaged endothelial cells and the potential mechanisms have not been elucidated. Methods: In our study, cell viability was tested using a 3-(4, 5-dimethylthiazol-2yl-)-2,5-diphenyl tetrazolium bromide (MTT) assay. Expression levels of interleukin-6, monocyte chemoattractant protein-1, tumor necrosis factor-a, intercellular adhesion molecule-1, and vascular cell adhesion molecule-1 were determined by enzyme-linked immunosorbent assay and Western blotting. Mitochondrial membrane potential (DJm) was detected using JC-1. The cell apoptotic percentage was measured by the Annexin V/ propidium iodide (PI) assay, lactate dehydrogenase, and caspase-3 expression. Apoptosis-related proteins, nuclear factor (NF)-kB, and mitogen-activated protein kinases (MAPK) signaling pathways protein expression were quantified by Western blotting. Results: Our results demonstrated that CK could ameliorate ox-LDL-induced human umbilical vein endothelial cells (HUVECs) inflammation and apoptosis, NF-kB nuclear translocation, and the phosphorylation of p38 and c-Jun N-terminal kinase (JNK). Moreover, anisomycin, an activator of p38 and JNK, significantly abolished the anti-apoptotic effects of CK. Conclusion: These results demonstrate that CK prevents ox-LDL-induced HUVECs inflammation and apoptosis through inhibiting the NF-kB, p38, and JNK MAPK signaling pathways. Thus, CK is a candidate drug for atherosclerosis treatment.

      • KCI등재

        SuperDepthTransfer: Depth Extraction from Image Using Instance-Based Learning with Superpixels

        ( Yuesheng Zhu ),( Yifeng Jiang ),( Zhuandi Huang ),( Guibo Luo ) 한국인터넷정보학회 2017 KSII Transactions on Internet and Information Syst Vol.11 No.10

        In this paper, we primarily address the difficulty of automatic generation of a plausible depth map from a single image in an unstructured environment. The aim is to extrapolate a depth map with a more correct, rich, and distinct depth order, which is both quantitatively accurate as well as visually pleasing. Our technique, which is fundamentally based on a preexisting DepthTransfer algorithm, transfers depth information at the level of superpixels. This occurs within a framework that replaces a pixel basis with one of instance-based learning. A vital superpixels feature enhancing matching precision is posterior incorporation of predictive semantic labels into the depth extraction procedure. Finally, a modified Cross Bilateral Filter is leveraged to augment the final depth field. For training and evaluation, experiments were conducted using the Make3D Range Image Dataset and vividly demonstrate that this depth estimation method outperforms state-of-the-art methods for the correlation coefficient metric, mean log10 error and root mean squared error, and achieves comparable performance for the average relative error metric in both efficacy and computational efficiency. This approach can be utilized to automatically convert 2D images into stereo for 3D visualization, producing anaglyph images that are visually superior in realism and simultaneously more immersive.

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