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( Ergenekon Karagoz ),( Alpaslan Tanoglu ),( Asim Ulcay ),( Veysel Ozalper ),( Vedat Turhan ),( Muammer Kara ),( Levent Gorenek ) 대한내과학회 2014 대한내과학회 추계학술발표논문집 Vol.2014 No.1
Background: The neutrophil to lymphocyte ratio (NLR), an infi ammation index, has been suggested to predict prognosis of various infi ammatory and neoplastic diseases. However, there are only a few studies examining the relationship between NLR and liver fi brosis score in HBV infected cases in literature. In this study, we aimed to determine the relationship between neutrophil to lymphocyte ratio (NLR) and fi brosis level. Methods: 144 biopsy proven naive chronic hepatitis B cases and 42 healthy subjects were included in the study. Previously treated patients by antiviral therapy and patients with anti-Delta positive were excluded from the study. Hepatitis B patients were divided into two groups with fi brosis scores of 0-1 and 2-6 according to ISHAC score. Results: Of the 144 cases, 125 (86.9 %) were male, 19 (13.1%) were female. fibrosis scores of 84 cases (58.3 %) were = 2 while 60 cases had fi brosis scores < 2 (41.7 %). There was a significant difference between Naive chronic hepatitis B group and healthy subjects in terms of NLR.The mean serum NLR was 1.822±0.853 in chronic hepatitis B patient group while it was found to be 2.29±1.408 in healthy subjects (p<0,05).There was not a signifi cant correlation existed between fi brosis score and NLR. NLR level was 1,906±1.027 in patients with fi brosis score 0-1 while this level was 1.762±0.702 in patients with fi brosis score 2-6 (p=0,576). Conclusions: In comparisons of healthy subjects with Hepatitis B infected patients, NLR was found to be lower in patients with Hepatitis B. Further studies are needed to determine the relationship between NLR and Hepatitis B.
( Veysel Ozalper ),( Ibrahim Cetindagli ),( Ergenekon Karagoz ),( Emrullah Solmazgul ),( Cihan Top ) 대한내과학회 2014 대한내과학회 추계학술발표논문집 Vol.2014 No.1
Objective: Hyperkalemia is a common and serious clinical problem that occurs often due to impaired urinary potassium excretion because of acute or chronic kidney disease or drugs that effect renin-angiotensin-aldosterone axis. We report a hyperkalemic patient with 9.0mEq/L without any ECG changes. Case Report: A 86-year-old-woman suffering fatigue within two days had admitted emergency service because of sudden bilateral lower extremity paralysis. She had esential hypertension, ischemic heart disease and chronic kidney disease and was using Spironolactone, silazopril, asetilsalisilik, isosorbid-5-mononitrat ve karvedilol. Initial neurological examination of extremities was; 2/5 of lower extremity, 4/5 of upper extremity. Initial laboratory fi ndings potassium:9,1mmol/L, sodium:141mmol/L, creatinine: 1,9mg/dl. In his arterial blood gas analyse, pH:7.27, HCO3:16mEq/L, pCO2:30mm- Hg, potassium:9,1mmol/L. Spironolactone and silazopril treatment had stopped. With an intensive electrolyte-follow-up, She was commenced on intravenous fl uid, injection calcium gluconate and glucose/insulin infusion. Within two hours of treatment her potassium level decreased by 1.5 mmol/L. After eight hours of intensive treatment, potassium level reached below 6,5mEq/L. Five Days later, after three days of close follow-up with normal potassium level, the patient had discharged. Conclusion: ECG differancies like fl attening in U wawe, expansion in QRS, taper in T wawe, arrhytmias, asystoles can be seen in hypercalemia. Especially in the circumstances that serum potassium level is above 8 mEq/L ECG differancy is expected to be seen. So that, it is suggested that biochemical elevation in hypercalemic cases is verifi ed with ECG. Although it is rarely seen like in our cases, it shouldn`t be forgotten that severe hypercalemia can be seen without ECG differancy.