http://chineseinput.net/에서 pinyin(병음)방식으로 중국어를 변환할 수 있습니다.
변환된 중국어를 복사하여 사용하시면 됩니다.
Family Origin Names and Haplotypes of Y - DNA in Korean Population
Kim, Jong Soon,Kim, Yung Jin,Ahn, Gwang Sook,Lee, Ju Won,You, Sun Ah 한국유전학회 1996 Genes & Genomics Vol.18 No.4
The use of restriction fragment length polymorphisms (RFLPs) has been proposed for the construction of a human genetic linkage map and also become a powerful tool in human population genetics. Human Y-chromosome which is haploid and paternally inherited is valuable for investigating male-mediated gene flow and for complement maternally based studies of mtDNA. We have studied 166 samples from 4 kinds of family origin names such as Kyungju Kim, Kimhae Kim, Chunju Lee and Milyang Park based on the 49aTaq 1 polymorphisms, and a total of 25 haplotypes were observed. The B, F, and I bands is observed in all haplotypes and haplotype showing A3, B, D2, D3, F, and I bands is common haplotype except for Chunju Lee. The result obtained are summarized as follows.
Lee, Kang‐,Won,Seo, Won‐,Kyeong,Cho, You‐,Ze,Kim, Jong‐,Woo,Park, Jin‐,Soo,Moon, Byoung‐,Sub John Wiley Sons, Ltd. 2010 International Journal of Communication Systems Vol.23 No.9
<P><B>Abstract</B></P>10.1002/dac.1076.abs<P>Current IP‐level mobility management protocols are unable to satisfy the stringent handover latency requirements for supporting real‐time multimedia services between different domains in a high‐speed vehicular environment. This paper proposes an inter‐domain handover scheme for Proxy Mobile IPv6 (PMIPv6) with a new network entity, called intermediate‐mobile access gateway (iMAG). The iMAG is located between the home and the foreign domains and is connected to both domains. The proposed scheme is a proactive handover approach that performs the inter‐domain L3 handover in advance before the inter‐domain L2 handover while the Mobile Node (MN) is still connected to the iMAG in the home domain. Therefore, the proposed scheme enables the inter‐domain handover latency to reduce as low as the intra‐domain handover latency. Numerical analysis and simulation results demonstrate that the proposed scheme is able to support seamless service continuity in inter‐domain handovers. Copyright © 2009 John Wiley & Sons, Ltd.</P>
Lee, You-Hyun,Choe, Jung-Yoon,Park, Sung-Hoon,Park, Yong-Wook,Lee, Shin-Seok,Kang, Young-Mo,Nam, Eon-Jeong,Park, Won,Kwon, Seong-Ryul,Bae, Sang-Cheol,Kim, Yun-Jung,Suh, Chang-Hee,Kim, Hyoun-Ah,Hur, Na The Korean Academy of Medical Sciences 2010 JOURNAL OF KOREAN MEDICAL SCIENCE Vol.25 No.10
<P>We performed a multicenter cross-sectional study of 134 sexually active systemic lupus erythematosus (SLE) patients to investigate the prevalence of and risk factors for high risk human papilloma virus (HPV) infection and cervical cytological abnormalities among Korean women with SLE. In this multicenter cross-sectional study, HPV testing and routine cervical cytologic examination was performed. HPV was typed using a hybrid method or the polymerase chain reaction. Data on 4,595 healthy women were used for comparison. SLE patients had greater prevalence of high-risk HPV infection (24.6% vs. 7.9%, <I>P</I><0.001, odds ratio 3.8, 95% confidence interval 2.5-5.7) and of abnormal cervical cytology (16.4 vs. 2.8%, <I>P</I><0.001, OR 4.4, 95% CI 2.5-7.8) compared with controls. SLE itself was identified as independent risk factors for high risk HPV infection among Korean women (OR 3.8, 95% CI 2.5-5.7) along with ≥2 sexual partners (OR 8.5, 95% CI 1.2-61.6), and Pap smear abnormalities (OR 97.3, 95% CI 6.5-1,456.7). High-risk HPV infection and cervical cytological abnormalities were more common among Korean women with SLE than controls. SLE itself may be a risk factor for HPV infection among Korean women, suggesting the importance of close monitoring of HPV infections and abnormal Pap smears in SLE patients.</P>
Effects of defects on the morphologies of GaN nanorods grown on Si (111) substrates
Lee, Kyu Hyung,Lee, Jeong Yong,Kwon, Y.H.,Kang, Tae Won,You, J.H.,Lee, D.U.,Kim, Taewhan Cambridge University Press (Materials Research Soc 2009 Journal of materials research Vol.24 No.10
<P>Scanning electron microscopy and transmission electron microscopy images and selected area electron diffraction pattern showed that the one-dimensional GaN nanorods with [0001]-oriented single-crystalline wurzite structures were formed on Si (111) substrates by using hydride vapor-phase epitaxy without a catalyst. Although some stacking faults and inversion domain boundaries existed in the GaN nanorods, few other defects such as threading dislocations were observed. The formation of the facet plane in the N-polar region of the GaN nanorod containing an inversion domain boundary originated from the slow growth rate, followed by the lateral adatom diffusion from the Ga-polar region to reduce the length difference.</P>
Lee, Hae-Young,You, Hyun-Jung,Won, Joo-Yun,Youn, Seock-Won,Cho, Hyun-Jai,Park, Kyung-Woo,Park, Woong-Yang,Seo, Jeong-Sun,Park, Young-Bae,Walsh, Kenneth,Oh, Byung-Hee,Kim, Hyo-Soo Ovid Technologies Wolters Kluwer -American Heart A 2008 Arteriosclerosis, thrombosis, and vascular biology Vol.28 No.2
<P>BACKGROUND: The forkhead factor, FOXO3a, is known to induce apoptosis in endothelial cells (ECs). However, its effects on extracellular matrices (ECM), which are important in EC survival, remained unknown. Here, we evaluated the role of FOXO3a on EC-ECM interaction. METHODS AND RESULTS: Constitutively active FOXO3a was transduced to human umbilical vein endothelial cells by adenoviral vector (Ad-TM-FOXO3a). Ad-TM-FOXO3a transfection led to dehiscence of ECs from fibronectin-coated plates, resulting in anoikis, which was significantly reversed by matrix metalloproteinase (MMP) inhibitor, GM6001. FOXO3a increased the expression of MMP-3 (stromelysin-1) but decreased the expression of tissue inhibitors of metalloproteinases-1 (TIMP-1), which was associated with increased MMP enzymatic activity in zymography. Pathophysiologic conditions such as serum starvation or heat shock also induced activation of endogenous FOXO3a, leading to activation of MMP-3 and apoptosis, which was reversed by GM6001. Delivery of Ad-TM-FOXO3a to the intraluminal surface in vivo led to EC denudation, disrupted vascular integrity, and impaired endothelium-dependent vasorelaxation. CONCLUSIONS: Activation of MMPs and possible ECM disruption represent novel mechanisms of FOXO3a-mediated apoptosis in ECs.</P>