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      저자 : Yongzhan Zhen (검색결과 2 건)
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        Rhein lysinate inhibits monocyte adhesion to human umbilical vein endothelial cells by blocking p38 signaling pathway

        Yajun Lin,Yongzhan Zhen,Jiang Liu,Jie Wei,Ping Tu,Gang Hu 대한약학회 2013 Archives of Pharmacal Research Vol.36 No.11

        The objective of this study was to investigate theeffect of rhein lysinate (RHL) on monocyte adhesion and itsmechanism. 3-(4,5-dimethylthiazol-2-yl)-2,5-diphenyltetrazoliumbromide (MTT) assay was used to determine thegrowth inhibition by drugs. The monocyte chemoattractantprotein (MCP)-1 levels were assayed using MCP-1 ELISA. The expression of proteins was detected by Western blottinganalysis. The results indicated that RHL inhibited monocyteadhesion in a dose- and time-dependent manner. RHL(\20 lmol/L) and lipopolysaccharide (LPS) had no effecton viability of human umbilical vein endothelial cells. Therefore, 20 lmol/L RHL was selected for this study. RHLinhibited secretion ofMCP-1 induced by LPS and expressionof intercellular adhesion molecule (ICAM)-1 and vascularcell adhesion molecule (VCAM)-1. In the meantime, bothRHL and p38 inhibitor (SB203580) inhibited phosphorylationof p38 and mitogen-activated protein kinase-activatedprotein kinase-2 (MAPKAPK-2) and transcription andexpression of ICAM-1 and VCAM-1. In conclusion, RHLinhibits the transcription and expression of ICAM-1 andVCAM-1 by the p38/MAPKAPK-2 signaling pathway, andthe effect ofRHLon transcription and expression of ICAM-1and VCAM-1 is similar to p38 inhibitor. RHL could be aprophylactic drug for atherosclerosis.

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        The protection of Rhein lysinate to liver in diabetic mice induced by high-fat diet and streptozotocin

        Yajun Lin,Gang Hu,Kai-Ji Li,Yu-Fang Zhao,Jie Wei,Yongzhan Zhen 대한약학회 2015 Archives of Pharmacal Research Vol.38 No.5

        Rhein lysinate (RHL) is the salt of lysine andrhein and the objective of this study was to investigate theprotection of RHL to liver in diabetic mice. The model oftype 2 diabetes was established by high-fat diet andstreptozotocin treatment. Malondialdehyde, superoxidedismutase (SOD) and glutathione peroxidase (GSH-Px)were measured using a spectrophotometer. Inflammatoryfactors (TNF-a and IL-6) and related proteins (ERK1/2 andSREBP-1c) were analyzed by Western blot. Tissue profilewas determined by hematoxylin and eosin staining andaccumulation of fat was examined by Nile red staining. The results indicated that plasma glucose levels of type 2diabetic mice were over 13.9 mM. Compared with modelgroup, plasma glucose levels were decreased, howeverinsulin levels were increased in RHL (25 and 50 mg/kg)-treated group. Elevated plasma triglyceride and cholesterolwere also markedly attenuated after RHL treatment. Theactivities of SOD and GSH-Px of livers were increasedafter RHL treatment. Livers of RHL-treated mice had morenormal structure and less steatosis than that of diabeticmice. Moreover, RHL decreased the expression of TNF-aand IL-6 and the phosphorylation of SREBP-1c and ERK1/2. In conclusion, RHL has a noticeable hepatic protectionin diabetic mice.

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