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Frequency Hopping CR MAC Protocol in Cognitive Radio Ad Hoc Network
Mei Bie,Zhe Wang,Wei Xiao,Kai Song 보안공학연구지원센터 2015 International Journal of Multimedia and Ubiquitous Vol.10 No.9
To insure the fairness for the Cognitive Radio (CR) nodes with various available channels, a frequency hopping CR MAC protocol based dynamic (named DCR-MAC) is proposed. In the case of uneven distribution of frequency spectrum in cognitive Ad Hoc network, based on the numbers of workable channels of each cognitive node, DCR-MAC protocol adjusts access time dynamically, ensuring the node priority access channel number, in order to improve the overall performance of the network. Meanwhile, against the problem of the multi-channel hidden terminal which appears in cognitive network of single wireless interface configuration, through introducing the "data segment" mechanism, and informing present channels occupied condition of adjacent node via receiving node, DCR-MAC protocol broadcast of channel occupation in transmission channel, and prohibit possible data transmission of adjacent node, in order to solve this problem on this account. Finally, proving that DCR-MAC protocol is obviously excelled other FH CR MAC protocols in through put capacity, packet access delay, accessing fairness which through network simulation.
Yang Tao,Xiao-mei Bie,Feng-xia Lv,Hai-zhen Zhao,Zhao-xin Lu 한국미생물학회 2011 The journal of microbiology Vol.49 No.1
The antifungal activity and mechanism of fengycin in the presence and absence of commercial surfactin against Rhizopus stolonifer were investigated. The MIC (minimal inhibitory concentration) of fengycin without commercial surfactin added was 0.4 mg/ml while the MIC of fengycin with commercial surfactin added was 2.0 mg/ml. Fengycin acted on cell membrane and cellular organs and inhibited DNA synthesis. The antifungal effect of fengycin was reduced after commercial surfactin was added. All these results suggest that the fungal cell membrane may be the primary target of fengycin action and commercial surfactin may reduce the antifungal activity of fengycin.