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Accurate and Robust Surface Measurement Using Optimal Structured Light Tracking Method
WANG, Shi,KIM, Hyongsuk,LIN, Chun-Shin,CHEN, Hongxin The Institute of Electronics, Information and Comm 2010 IEICE transactions on information and systems Vol.93 No.2
<P>Structured light vision systems are based on the fact that the pixel location of light in an image has a unique association with the object depth. However, their applications are mainly limited to the properties of the object surface and the lighting conditions. This paper presents a robust vision system for accurate acquisition of 3-D surface data based on optimal structured light. To achieve depth measurement for a dynamic scene, the data acquisition must be performed with only a single image. Our special arrangement makes the image of the light stripe remaining sharp while the background becomes blurred. Moreover, a dynamic programming approach is proposed to track the optimal path while the laser beam is invisible or divergent under extreme condition. The principle and necessary mathematics for implementing the algorithm are presented. The robustness of the system against uncalibrated errors is demonstrated.</P>
Yang, Zhiping,Zhao, Yan,Yao, Yahong,Li, Jun,Wang, Wangshi,Wu, Xiaonan Korean Society for Molecular and Cellular Biology 2016 Molecules and cells Vol.39 No.10
The cancer chemo-preventive effects of equol have been demonstrated for a wide variety of experimental tumours. In a previous study, we found that equol inhibited proliferation and induced apoptotic death of human gastric cancer MGC-803 cells. However, the mechanisms underlying equol-mediated apoptosis have not been well understood. In the present study, the dual AO (acridine orange)/EB (ethidium bromide) fluorescent assay, the comet assay, MTS, western blotting and flow cytometric assays were performed to further investigate the pro-apoptotic effect of equol and its associated mechanisms in MGC-803 cells. The results demonstrated that equol induced an apoptotic nuclear morphology revealed by AO/EB staining, the presence of a comet tail, the cleavage of caspase-3 and PARP and the depletion of cIAP1, indicating its pro-apoptotic effect. In addition, equol-induced apoptosis involves the mitochondria-dependent cell-death pathway, evidenced by the depolarization of the mitochondrial membrane potential, the cleavage of caspase-9 and the depletion of Bcl-xL and full-length Bid. Moreover, treating MGC-803 cells with equol induced the sustained activation of extracellular signal-regulated kinase (ERK), and inhibiting ERK by U0126, a MEK/ERK pathway inhibitor, significantly attenuated the equol-induced cell apoptosis. These results suggest that equol induces mitochondria-dependent apoptosis in human gastric cancer MGC-803 cells via the sustained activation of the ERK1/2 pathway. Therefore, equol may be a novel candidate for the chemoprevention and therapy of gastric cancer.
Xiaonan Wu,Zhiping Yang,Yan Zhao,Yahong Yao,Jun Li,Wangshi Wang 한국분자세포생물학회 2016 Molecules and cells Vol.39 No.10
The cancer chemo-preventive effects of equol have been demonstrated for a wide variety of experimental tumours. In a previous study, we found that equol inhibited proliferation and induced apoptotic death of human gastric cancer MGC-803 cells. However, the mechanisms underlying equol-mediated apoptosis have not been well understood. In the present study, the dual AO (acridine orange)/EB (ethidium bromide) fluorescent assay, the comet assay, MTS, western blotting and flow cytometric assays were performed to further investigate the pro-apoptotic effect of equol and its associated mechanisms in MGC-803 cells. The results demonstrated that equol induced an apoptotic nuclear morphology revealed by AO/EB staining, the presence of a comet tail, the cleavage of caspase-3 and PARP and the depletion of cIAP1, indicating its pro-apoptotic effect. In addition, equol-induced apoptosis involves the mitochondria-dependent cell-death pathway, evidenced by the depolarization of the mitochondrial membrane potential, the cleavage of caspase-9 and the depletion of Bcl-xL and full-length Bid. Moreover, treating MGC-803 cells with equol induced the sustained activation of extracellular signal-regulated kinase (ERK), and inhibiting ERK by U0126, a MEK/ERK pathway inhibitor, significantly attenuated the equol-induced cell apoptosis. These results suggest that equol induces mitochondria-dependent apoptosis in human gastric cancer MGC-803 cells via the sustained activation of the ERK1/2 pathway. Therefore, equol may be a novel candidate for the chemoprevention and therapy of gastric cancer.