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RISS 인기검색어
- 검색키워드
- 저자 : Lina Ning (검색결과 3 건)
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Spatial spectrum approach for pilot spoofing attack detection in MIMO systems
Ning, Lina,Li, Bin,Wang, Xiang,Liu, Xiaoming,Zhao, Chenglin Electronics and Telecommunications Research Instit 2021 ETRI Journal Vol.43 No.5
In this study, a spatial spectrum method is proposed to cope with the pilot spoofing attack (PSA) problem by exploiting the of uplink-downlink channel reciprocity in time-division-duplex multiple-input multiple-output systems. First, the spoofing attack in the uplink stage is detected by a threshold derived from the predefined false alarm based on the estimated spatial spectrum. When the PSA occurs, the transmitter (That is Alice) can detect either one or two spatial spectrum peaks. Then, the legitimate user (That is Bob) and Eve are recognized in the downlink stage via the channel reciprocity property based on the difference between the spatial spectra if PSA occurs. This way, the presence of Eve and the direction of arrival of Eve and Bob can be identified at the transmitter end. Because noise is suppressed by a spatial spectrum, the detection performance is reliable even for low signal-noise ratios and a short training length. Consequently, Bob can use beamforming to transmit secure information during the data transmission stage. Theoretical analysis and numerical simulations are performed to evaluate the performance of the proposed scheme compared with conventional methods.
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Cuprous Oxide Films with Hollow Cubic Cage Structure for Nonenzymatic Glucose Detection
Fang Sun,Lehong Xing,Xihui Yang,Hailiang Huang,Lina Ning 성균관대학교(자연과학캠퍼스) 성균나노과학기술원 2019 NANO Vol.14 No.4
In this study, CuO films with hollow cubic cages were prepared by a facile two-step procedure consisting of electrodeposition synthesis and subsequent direct calcination. First, Cu2O nanocubes were fabricated on ITO substrate through a simple electrodeposition procedure. Then, Cu2O nanocubes were converted to CuO hollow cubic cages without obvious morphological change through direct calcination. The obtained CuO cubic cages serving as active materials illustrated a favorable performance for nonenzymatic glucose sensing with high sensitivity of 2117.44 μA mM -1 cm -2 at a low applied potential of 0.50 V, fast-response time (less than 3 s), low detection limit of 1.0 μM and wide linear range up from 2.0 μM to 1.0 mM (R2 = 0.9983). Moreover, the good selectivity of the CuO cubic cages-based nonenzymatic glucose sensor against electroactive compounds such as ascorbic acid, uric acid and dopamine were also demonstrated. These good features indicate that the as-prepared CuO cubic cages can be used as promising electrode materials, which have a great potential in the development of sensitive and selective nonenzymatic glucose sensors.
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Yang, Xiao-Li,Zhang, Cheng-Dong,Wu, Hua-Yu,Wu, Yong-Hu,Zhang, Yue-Ning,Qin, Meng-Bin,Wu, Hua,Liu, Xiao-Chun,Lina, Xing,Lu, Shao-Ming Asian Pacific Journal of Cancer Prevention 2014 Asian Pacific journal of cancer prevention Vol.15 No.11
Trichostatin A (TSA) is a histone deacetylase (HDAC) inhibitor. We here investigated its effects on proliferation and apoptosis of the CNE2 carcinoma cell line, and attempted to establish genome-wide DNA methylation alteration due to differentially histone acetylation status. After cells were treated by TSA, the inhibitory rate of cell proliferation was examined with a CCK8 kit, and cell apoptosis was determined by flow cytometry. Compared to control, TSA inhibited CNE2 cell growth and induced apoptosis. Furthermore, TSA was found to induce genome-wide methylation alteration as assessed by genome-wide methylation array. Overall DNA methylation level of cells treated with TSA was higher than in controls. Function and pathway analysis revealed that many genes with methylation alteration were involved in key biological roles, such as apoptosis and cell proliferation. Three genes (DAP3, HSPB1 and CLDN) were independently confirmed by quantitative real-time PCR. Finally, we conclude that TSA inhibits CNE2 cell growth and induces apoptosis in vitro involving genome-wide DNA methylation alteration, so that it has promising application prospects in treatment of NPC in vivo. Although many unreported hypermethylated/hypomethylated genes should be further analyzed and validated, the pointers to new biomarkers and therapeutic strategies in the treatment of NPC should be stressed.
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