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생활하수처리를 위한 CNR Process 설계요소 연구
왕창근,전문환 충남대학교 환경문제연구소 2000 環境硏究 Vol.18 No.-
A biological nutrient removal system combining A2O process with biofilm was studied in this work and the characteristics of nitrogen and phophorus removal was especially investigated for the Cilium Nutrient Removal Process(CNR process). The CNR process consisted of anaerobic, anoxic, aerobic zone in series, and aerobic zone contains packed cilium media. The CNR has benefits in solving the problems which inhibit denitrification and phophorus uptake in anoxic reactor due to the lack of organic compounds. The bench-scale experiment was performed using Tae-jon and Um-am domestic wastewater and the influent was accomplished with step-feed loading to the reactor. Removal efficiency of contaminants including BOD, COD, TN and TP was relatively high.
Jung, Yu-Ri,Lee, Young-Jung,Lee, Nam-Jin,Lin, Chun-Mai,Moon, Jun-Hawn,Chai, Hee-Yul,Kang, Jong-Koo Korean Society of ToxicologyKorea Environmental Mu 2010 Toxicological Research Vol.27 No.3
Hepatic fibrosis represents the main complication of most chronic liver disorders and, regardless of its etiology, is characterized by excessive deposition of extracellular matrix components. In this study, we examined that 1-O-Hexyl-2,3,5-Trimethylhydroquinone (HTHQ), a potent anti-oxidative agent, could prevent experimental hepatic fibrosis induced by dimethylnitrosamine (DMN) in male SD rats. Except for vehicle control group, other groups were induced hepatic fibrosis by intraperitoneal injection with DMN (10 mg/ml/kg) on 3 consecutive days weekly for 4 weeks. During the same 4 weeks, control and DMN groups were given vehicle and HTHQ 50, 100 and 200 groups were orally administered HTHQ (50, 100, 200 mg/kg respectively). In HTHQ 100 and 200 groups, relative liver weight and serum chemistry level improved significantly. HTHQ reduced hydroxyproline (p < 0.05) and malondialdehyde (p < 0.05) level in the liver. Histopathological examination of H&E, Masson's trichrome stain showed the reduced fibrotic septa in HTHQ 100 and 200 groups. HTHQ administration showed reduced mRNA level of PDGF (Platelet-derived growth factor), $\alpha$-SMA ($\alpha$-smooth muscle actin) and TGF-$\beta$ (transforming growth factor-$\beta$) than DMN-induced hepetic fibrosis animals in the liver tissue. In this study, we showed that HTHQ improves against DMN-induced liver fibrosis in male SD rats.
Yu-Ri Jung,Young-Jung Lee,Nam-Jin Lee,Chun-Mai Lin,Jun-Hawn Moon,Hee-Yul Chai,Jong-Koo Kang 한국독성학회 2010 Toxicological Research Vol.26 No.3
Hepatic fibrosis represents the main complication of most chronic liver disorders and, regardless of its etiology, is characterized by excessive deposition of extracellular matrix components. In this study, we examined that 1-O-Hexyl-2,3,5-Trimethylhydroquinone (HTHQ), a potent anti-oxidative agent, could prevent experimental hepatic fibrosis induced by dimethylnitrosamine (DMN) in male SD rats. Except for vehicle control group, other groups were induced hepatic fibrosis by intraperitoneal injection with DMN (10 ㎎/㎖/㎏) on 3 consecutive days weekly for 4 weeks. During the same 4 weeks, control and DMN groups were given vehicle and HTHQ 50, 100 and 200 groups were orally administered HTHQ (50, 100, 200 ㎎/㎏ respectively). In HTHQ 100 and 200 groups, relative liver weight and serum chemistry level improved significantly. HTHQ reduced hydroxyproline (p<0.05) and malondialdehyde (p<0.05) level in the liver. Histopathological examination of H&E, Masson’s trichrome stain showed the reduced fibrotic septa in HTHQ 100 and 200 groups. HTHQ administration showed reduced mRNA level of PDGF (Plateletderived growth factor), α-SMA (α-smooth muscle actin) and TGF-β (transforming growth factor-β) than DMN-induced hepetic fibrosis animals in the liver tissue. In this study, we showed that HTHQ improves against DMN-induced liver fibrosis in male SD rats.