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- 저자 : Hsiang Lan LIU (검색결과 2 건)
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Diverging from the normal path : The life course of an elderly woman in Kinmen county
Hsiang Lan LIU Asian Center for Women's Studies : Ewha Womans Uni 2020 Asian Journal of Women's Studies(AJWS) Vol.26 No.4
Kinmen, also known as Quemoy, is a unique offshore island of the Republic of China (ROC) that had an unusual and strategic position during the Cold War from 1946 to 1992. After 1949, Kinmen was forced to become involved in the military standoff between Chiang Kai-shek’s Republic of China in Taiwan(ROC) and Mao Zedong’s People’s Republic of China (PRC) during the cold war. Kinmen was under martial law for nearly 50 years from 1946 to 1992. The martial law was lifted in 1992 and Kinmen has become the frontline for cross-strait exchanges between the ROC and mainland China. Men’s heroic stories during the war have been studied and published, while women’s voices are systematically ignored. In order to examine the roles and value of women in Kinmen’s history, a case study methodology was used here. Su was one such unique woman, whose life story is investigated here. My main findings were about how she, as an active agent, used opportunities in order to overcome the major structural pressures of her life, while Kinmen underwent some major transitions. This paper is based on an account of this woman’s extraordinary struggles in different periods of time. 金門是中華民國相當獨特的離島,其特殊性在於冷戰從1946年到1992年間,她被迫成為冷戰戰場,捲進了中國國民黨蔣介石與中國共產黨毛澤東間的衝突。 從1946年到1992年,金門進入到軍事統治近50年,直到1992年解除戒嚴,轉型為中國與中華民國兩岸交流的前線。金門男性在戰爭時期的英勇事蹟已被記載與發表,但女性的聲音受系統性地忽略。為檢視金門女性位置和金門歷史的關係,本文採用個案研究方法,呈現一位女性的生命位置與金門史的關聯。主要發現是女性是一個積極的主體能動者,在不同金門重大變遷時運用機會超越結構性的壓力,此作為一個證據呈現金門女性在冷戰時期的不凡。
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Liu, Bin,Ho, Hsiang-Ting,Brunello, Lucia,Unudurthi, Sathya D.,Lou, Qing,Belevych, Andriy E.,Qian, Lan,Kim, Do Han,Cho, Chunghee,Janssen, Paul M. L.,Hund, Thomas J.,Knollmann, Bjorn C.,Kranias, Evangel Oxford University Press 2015 Cardiovascular research Vol.108 No.2
<P><B>Aims</B></P><P>Cardiac calsequestrin (CASQ2) and histidine-rich Ca-binding protein (HRC) are sarcoplasmic reticulum (SR) Ca-binding proteins that regulate SR Ca release in mammalian heart. Deletion of either CASQ2 or HRC results in relatively mild phenotypes characterized by preserved cardiac structure and function, although CASQ2 knockout (KO), or Cnull, shows increased arrhythmia burden under conditions of catecholaminergic stress. We hypothesized that given the apparent overlap of functions of CASQ2 and HRC, simultaneous ablation of both would deteriorate the cardiac phenotype compared with the single knockouts.</P><P><B>Methods and results</B></P><P>In contrast to this expectation, double knockout (DKO) mice lacking both CASQ2 and HRC exhibited normal cardiac ejection fraction and ultrastructure. Moreover, the predisposition to catecholamine-dependent arrhythmia that characterizes the Cnull phenotype was alleviated in the DKO mice. At the myocyte level, DKO mice displayed Ca transients of normal amplitude; additionally, the frequency of spontaneous Ca waves and sparks in the presence of isoproterenol were decreased markedly compared with Cnull. Furthermore, restitution of SR Ca release was slowed in DKO myocytes compared with Cnull cells.</P><P><B>Conclusion</B></P><P>Our results suggest that rather than being functionally redundant, CASQ2 and HRC modulate cardiac ryanodine receptor-mediated (RyR2) Ca release in an opposing manner. In particular, while CASQ2 stabilizes RyR2 rendering it refractory in the diastolic phase, HRC enhances RyR2 activity facilitating RyR2 recovery from refractoriness.</P>
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