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RISS 인기검색어
- 검색키워드
- 저자 : Hailiang Ma (검색결과 3 건)
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- 유료
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Hailiang Xu,Xiaojun Ma,Dan Sun 전력전자학회 2015 JOURNAL OF POWER ELECTRONICS Vol.15 No.1
This paper proposes a reactive current assignment and control strategy for a doubly-fed induction generator (DFIG) based wind-turbine generation system under generic grid voltage sag or swell conditions. The system’s active and reactive power constrains during grid faults are investigated with both the grid- and rotor-side convertors (GSC and RSC) maximum ampere limits considered. To meet the latest grid codes, especially the low- and high-voltage ride-through (LVRT and HVRT) requirements, an adaptive reactive current control scheme is investigated. In addition, a torque-oscillation suppression technique is designed to reduce the mechanism stress on turbine systems caused by intensive voltage variations. Simulation and experiment studies demonstrate the feasibility and effectiveness of the proposed control scheme to enhance the fault ride-through (FRT) capability of DFIG-based wind turbines during violent changes in grid voltage.
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Xu, Hailiang,Ma, Xiaojun,Sun, Dan The Korean Institute of Power Electronics 2015 JOURNAL OF POWER ELECTRONICS Vol.15 No.1
This paper proposes a reactive current assignment and control strategy for a doubly-fed induction generator (DFIG) based wind-turbine generation system under generic grid voltage sag or swell conditions. The system's active and reactive power constrains during grid faults are investigated with both the grid- and rotor-side convertors (GSC and RSC) maximum ampere limits considered. To meet the latest grid codes, especially the low- and high-voltage ride-through (LVRT and HVRT) requirements, an adaptive reactive current control scheme is investigated. In addition, a torque-oscillation suppression technique is designed to reduce the mechanism stress on turbine systems caused by intensive voltage variations. Simulation and experiment studies demonstrate the feasibility and effectiveness of the proposed control scheme to enhance the fault ride-through (FRT) capability of DFIG-based wind turbines during violent changes in grid voltage.
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Jianyao Shen,Hailiang Ma,Chaoquan Wang 대한생리학회-대한약리학회 2021 The Korean Journal of Physiology & Pharmacology Vol.25 No.6
Myocardial fibrosis (MF) is the result of persistent and repeated aggravation of myocardial ischemia and hypoxia, leading to the gradual development of heart failure of chronic ischemic heart disease. Triptolide (TPL) is identified to be involved in the treatment for MF. This study aims to explore the mechanism of TPL in the treatment of MF. The MF rat model was established, subcutaneously injected with isoproterenol and treated by subcutaneous injection of TPL. The cardiac function of each group was evaluated, including LVEF, LVFS, LVES, and LVED. The expressions of ANP, BNP, inflammatory related factors (IL-1β, IL-18, TNF-α, MCP-1, VCAM- 1), NLRP3 inflammasome factors (NLRP3, ASC) and fibrosis related factors (TGF-β1, COL1, and COL3) in rats were dete cted. H&E staining and Masson staining were used to observe myocardial cell inflammation and fibrosis of rats. Western blot was used to detect the p-P65 and t-P65 levels in nucleoprotein of rat myocardial tissues. LVED and LVES of MF group were significantly upregulated, LVEF and LVFS were significantly downregulated, while TPL treatment reversed these trends; TPL treatment downregulated the tissue injury and improved the pathological damage of MF rats. TPL treatment downregulated the levels of inflammatory factors and fibrosis factors, and inhibited the activation of NLRP3 inflammasome. Activation of NLRP3 inflammasome or NF-κB pathway reversed the effect of TPL on MF. Collectively, TPL inhibited the activation of NLRP3 inflammasome by inhibiting NF-κB pathway, and improved MF in MF rats.
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