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신용우,박향준,김유찬,송원근 대한피부과학회 1999 大韓皮膚科學會誌 Vol.37 No.7
Freys syndrome is a type of pathologic gustatory hyperhidrosis occurring as a result of injury to the nerves in the region of the parotid gland. The syndrome usually manifests as unilateral flushing, sweating, and rarely pain in response to gustatory stimuli. The most likely mechanism is aberrant reinnervation of the sweat glands of the face by the severed parotid parasympathetic nerve fibers. We report a case of Freys syndrome in a 58-year-old woman who had been treated with incision and drainage at age 10 due to an abscess occurred in the parotid area. Thereafter she complained local flushing and sweating on the left infraauricular area during mastication. She was treated with 0.5% atropine cream once a day for two weeks, resulting in marked reduction of flushing and sweating proved by Minors starch-iodine test.
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이정복,이원호,김영근,탁미진희 대한피부과학회 1980 大韓皮膚科學會誌 Vol.18 No.3
A case of crotamiton resistant scabies is described in a 21-year-old male patient The scrapings using mineral oil from lesions on the wrist and finger webs showed mites and eggs, typical of scabies. Ten percent crotamitn cream was applied 10 times in one month without any effect. We could not find any source of reinfection in the patient. After above treatment the scrapings still showed mites. and eggs. Retreatment with 1% gamma benzene hexachloride cream was effective in clearing of the skin rashes and itching sensation.
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이유신,신용우 대한피부과학회 1980 大韓皮膚科學會誌 Vol.18 No.3
It is generally accepted that tbe lepromin test is of great value in the type classification and the evaluation of the prognosis of leprosy. But the nature of the antigenicity in lepromin reaction stiIl remains uncertain. The present study was carried out to investigate the antigenic characteriatics of the early(Fernandez) and the late (Mitsuda) reactions to Mitsuda lepromin antigen. Mycobacterium leprae was purified by trypsin digestion from human lepromas, and was broken down by passing 5 times through a French Pressor at 16, 000 p.s.i.. From the broken cell suspension, cell wall, cytoplasm, very low density lipoprotein (VLDL) and lipopolysaccharide (LPS) were prepared. IntradermaI skin tests were perforrned on 30 leprosy patients classified according to the Ridley-Jopling scale (4 TT, 6 BT, 2 BB, 7 BL and 11 LL), with 0. 1ml of the Mitsuda lepromin(intact cells) broken cells, cell wall, cytoplasm, VLDL and LPS antigens. Readings for the early and the late reactions were done 72 hours and 28 days, respectively, after itradermal injections of the above antigens. The results are summerized as follows: 1. Early reactions were elicited by broken cells, cell wall and cytoplasm in all of the patients who could react to Mitsuda lepromin antigen. VLDL and LPS antigens could also elicit early reactions in 9(56)% and in 14(88%), respectively, of the 10 patients who showed positive reaction to Mitsuda lepromin antigen. These results mean that the early reaction may be due to multiple antigena, originated either from M. leprae or from contaminated human tissues. 2. Late reactions were elicited by broken cells and cell wall antigens, but not by cytoplasm antigen, in all of the patients who showed late reactions to Mitsuda lepromin antigen. These results mean that broken cells also produce a late reaction and the specific antigen(s) of the late reaction may reside in the cell wall fraction of the M. leprae.
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