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Confirmation of the VeLLO L1148−IRS: star formation at very low (column) density
Kauffmann, J.,Bertoldi, F.,Bourke, T. L.,Myers, P. C.,Lee, C. W.,Huard, T. L. Blackwell Publishing Ltd 2011 Monthly notices of the Royal Astronomical Society Vol.416 No.3
<P><B>ABSTRACT</B></P><P>We report the detection of a compact (∼5 arcsec; about 1800 au projected size) CO outflow from L1148−IRS. This confirms that this <I>Spitzer</I> source is physically associated with the nearby (≈325 pc) L1148 dense core. Radiative transfer modelling suggests an internal luminosity of 0.08 to 0.13 L<SUB>⊙</SUB>. This validates L1148−IRS as a Very Low Luminosity Object (VeLLO; <I>L</I>≤ 0.1 L<SUB>⊙</SUB>). The L1148 dense core has unusually low densities and column densities for a star‐forming core. It is difficult to understand how L1148−IRS might have formed under these conditions. Independent of the exact final mass of this VeLLO (which is likely <0.24 M<SUB>⊙</SUB>), L1148−IRS and similar VeLLOs might hold some clues about the isolated formation of brown dwarfs.</P>
Pathomechanism of oxidative stress in cardiovascularrenal remodeling and therapeutic strategies
Ravarotto Verdiana,Bertoldi Giovanni,Stefanelli Lucia Federica,Nalesso Federico,Calò Lorenzo A. 대한신장학회 2022 Kidney Research and Clinical Practice Vol.41 No.5
The high prevalence of cardiovascular disease in patients with chronic kidney disease indicates significant interactions between pathogenic pathways operating in the kidney and heart. These interactions involve all cell types (endothelial cells, smooth muscle cells, macrophages, and others), components of the vasculature, glomeruli, and heart that are susceptible to oxidative damage and structural alterations. A vicious cycle occurs whereby harmful factors such as reactive oxygen species and inflammation damage of vascular structures that themselves become sources of additional dangerous/toxic components released into the local environment. The evidence of this vicious cycle in chronic kidney disease should therefore lead to add other factors to both traditional and nontraditional risk factors. This review will examine the processes occurring during progressive kidney dysfunction with regard to vascular injury, renal remodeling, cardiac hypertrophy, and the transversal role of oxidative stress in the development of these complications.