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PERIODIC SOLUTIONS FOR DUFFING TYPE p-LAPLACIAN EQUATION WITH MULTIPLE DEVIATING ARGUMENTS
Jiang, Ani The Korean Society for Computational and Applied M 2013 Journal of applied mathematics & informatics Vol.31 No.1
In this paper, we consider the Duffing type p-Laplacian equation with multiple deviating arguments of the form $$({\varphi}_p(x^{\prime}(t)))^{\prime}+Cx^{\prime}(t)+go(t,x(t))+\sum_{k=1}^ngk(t,x(t-{\tau}_k(t)))=e(t)$$. By using the coincidence degree theory, we establish new results on the existence and uniqueness of periodic solutions for the above equation. Moreover, an example is given to illustrate the effectiveness of our results.
PERIODIC SOLUTIONS FOR DUFFING TYPE p-LAPLACIAN EQUATION WITH MULTIPLE DEVIATING ARGUMENTS
Ani Jiang 한국전산응용수학회 2013 Journal of applied mathematics & informatics Vol.31 No.1
Abstract. In this paper, we consider the Duffing type p-Laplacian equationwith multiple deviating arguments of the form(φp(x′(t)))′+ Cx′(t) + g0(t, x(t)) +Σnk=1gk(t, x(t - τk(t))) = e(t). By using the coincidence degree theory, we establish new results on the existenceand uniqueness of periodic solutions for the above equation. Moreover,an example is given to illustrate the effectiveness of our results.
MicroRNA-214-mediated UBC9 expression in glioma
( Zhi Qiang Zhao ),( Xiao Chao Tan ),( Ani Zhao ),( Li Yuan Zhu ),( Bin Yin ),( Jiang Ang Yuan ),( Bo Qin Qiang ),( Xiao Zhong Peng ) 생화학분자생물학회(구 한국생화학분자생물학회) 2012 BMB Reports Vol.45 No.11
It has been reported that ubiquitin-conjugating enzyme 9 (Ubc9), the unique enzyme2 in the sumoylation pathway, is up-regulated in many cancers. However, the expression and regulation of UBC9 in glioma remains unknown. In this study, we found that Ubc9 was up-regulated in glioma tissues and cell lines compared to a normal control. UBC9 knockdown by small interfering RNA (siRNA) affected cell proliferation and apoptosis in T98G cells. Further experiments revealed that microRNA (miR)-214 directly targeted the 3` untranslated region (UTR) of UBC9 and that there was an inverse relationship between the expression levels of miR-214 and UBC9 protein in glioma tissues and cells. MiR-214 overexpression suppressed the endogenous UBC9 protein and affected T98G cell proliferation. These findings suggest that miR-214 reduction facilitates UBC9 expression and is involved in the regulation of glioma cell proliferation.