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      저자 : ( Bossng Kang ) (검색결과 2 건)
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      • Oral intake of anti-hangover substance increases metabolizing capacity of aldehyde dehydrogenase 2 in rat model: new therapeutic potentials for chronic itch?

        ( Bossng Kang ),( Chae Young Bang ),( Se Young Choung ),( Kyungwoo Choi ),( Changsun Kim ),( Yaejin Hutchison ),( Hyuk Joong Choi ) 대한피부과학회 2015 대한피부과학회 학술발표대회집 Vol.67 No.2

        Background: Aldehyde dehydrogenase 2 (ALDH 2) metabolizes acetaldehyde, the major cause of alcohol hangover symptoms. It also detoxifies endogenouscytotoxic aldehydes, such as 4-hydroxynonenal. Oxidative stress promotes lipid peroxidation of cellular membrane, leading to accumulation of reactive aldehydes that contribute to itch signaling via mast cell degranulation and activation of TRPA1 on sensory neuron. A variety of anti-hangover products are available, however, almost none of them has been proven to show enhanced metabolizing capacity of ALDH 2 in a live subject. Objectives: We aimed to test a specific product of interest. Methods: A powder sample of anti-hangover product (KISLip, Pico Entech, Korea) was examined by in vitro & in vivo experiments to measure the amount of NADH formation, generated through catalytic conversion of acetaldehyde. In-vivo examination tested the ethanol and acetaldehyde level in blood of rats with oral infusion of substance before or after ethanol intake. Results: The activities of alcohol dehydrogenase & aldehyde dehydrogenase within the anti-hangover substance were 1.84 unit/g and 0.28 unit/g. The oxidation capacities in rats were dose-dependently increased after substance gavages. Particularly, the cases with oral intake of substance 220 mg/kg after 1hr of ethanol intake have shown more meaningful decreases in acetaldehyde level in blood. Conclusion: Oral intake of anti-hangover substance potentially enhanced ALDH 2 capacity within circulation

      • Targeting aldehyde dehydrogenase 2 for treatment of aging body odor, old person smell: new therapeutic potential?

        ( Bossng Kang ),( Chae Young Bang ),( Se Young Choung ),( Heung Taek Kwon ),( Kyungwoo Choi ),( Changsun Kim ),( Yaejin Hutchison ),( Hyuk Joong Choi ) 대한피부과학회 2015 대한피부과학회 학술발표대회집 Vol.67 No.2

        Background: Aldehyde dehydrogenase 2 (ALDH 2) metabolizes acetaldehyde, the major cause of alcohol hangover symptoms. It also detoxifies other endogenous aldehydes, such as 2-nonenal. It is an unsaturated aldehyde with an unpleasant greasy and grassy odor. Oxidative stress promotes peroxidation of polyunsaturated fatty acids in cellular membrane, generating 2-nonenal that contribute to aging body odor. A variety of anti-hangover products are available, however, almost none of them has been proven to show enhanced metabolizing capacity of ALDH 2 in a live subject. Objectives: We aimed to test a specific product of interest. Methods: A powder sample of anti-hangover product(KISLip, Pico Entech, Korea) was examined by in vitro & in vivo experiments to measure the amount of NADH formation, generated through catalytic conversion of acetaldehyde. In-vivo examination tested the ethanol and acetaldehyde level in blood of rats with oral infusion of substance before or after ethanol intake. Results: The activities of alcohol dehydrogenase & aldehyde dehydrogenase within the anti-hangover substance were 1.84 unit/g and 0.28 unit/g. The oxidation capacities in experimental rats were dose-dependentlyincreased after substance gavages. Particularly, the cases with oral intake of substance 220 mg/kg after 1hr of ethanol intake have shown more meaningful decreases in acetaldehyde level in blood. Conclusion: Oral intake of anti-hangover substance potentially enhance ALDH 2 capacity within circulation.

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