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Luminescence and thermal-quenching properties of Dy3+-doped Ba2CaWO6 phosphors
Yu, Ruijin,Shin, Dong Soo,Jang, Kiwan,Guo, Yue,Noh, Hyeon Mi,Moon, Byung Kee,Choi, Byung Chun,Jeong, Jung Hyun,Yi, Soung Soo Elsevier 2014 Spectrochimica acta. Part A, Molecular and biomole Vol.125 No.-
A series of new double perovskite tungstate Ba2CaWO6:xDy(3+) (0.01 <= x <= 0.15) phosphors were synthesized via solid state reaction process. XRD analysis confirmed the phase formation of Ba2CaWO6:Dy3+ materials. The photoluminescence excitation and emission spectra, concentration effect, thermal-quenching, and decay property were investigated. The phosphor could be excited by the UV light region from 250 to 400 nm, and it exhibits blue (493 nm) and yellow (584 nm) emission corresponding to F-4(9/2)-H-6(15/2) transitions and F-4(9/2)-H-6(13/2) transitions, respectively. The optimum dopant concentration of Dy3+ ions in Ba2CaWO6:xDy(3+) is around 5 mol% and the critical transfer distance of Dy3+ is calculated as 14 angstrom. The thermal-quenching temperature is 436 K for Ba2CaWO6:0.05Dy(3+). The fluorescence lifetime is also determined in Ba2CaWO6:0.05Dy(3+). (C) 2014 Elsevier B.V. All rights reserved.
Mechanism of Q-spoiling in deformed optical microcavities.
Yu, Hyeon-Hye,Yi, Chang-Hwan,Kim, Chil-Min Optical Society of America 2015 Optics express Vol.23 No.9
<P>It was reported that Q spoiling in a chaotic microcavity is caused by chaos [PRL, 75, 2682 (1995)] and chaos-assisted tunneling [Nature 385, 45 (1997)]. However, even when a cavity is slightly deformed not to exhibit a broad chaotic region in phase space, high Q modes are spoiled. We find that Q spoiling in this region is caused by the transition of a whispering gallery mode (WGM) to a scarred resonance when a WGM interacts with its pair quasi-normal mode through an avoided resonance crossing. We prove that this transition induces Q spoiling in a quadrupole dielectric microcavity by showing that Q factors obtained from the Husimi functions depending on resonance deformation during the transition agree well with those obtained from the complex eigenvalues.</P>
Long spin coherence length and bulk-like spin-orbit torque in ferrimagnetic multilayers
Yu, Jiawei,Bang, Do,Mishra, Rahul,Ramaswamy, Rajagopalan,Oh, Jung Hyun,Park, Hyeon-Jong,Jeong, Yunboo,Van Thach, Pham,Lee, Dong-Kyu,Go, Gyungchoon,Lee, Seo-Won,Wang, Yi,Shi, Shuyuan,Qiu, Xuepeng,Awano Springer Science and Business Media LLC 2019 Nature materials Vol.18 No.1
Role of Gangliosides in LMNA Mutation-induced Hutchinson-Gilford progeria syndrome model
Hyeon Gyu Lim,Seo Yi Lee,Won Seok Ju,Sung Youn Heo,Yu Na Seo,Young-Kug Choo,Dong Hoon Kwak 한국당과학회 2016 한국당과학회 학술대회 Vol.2016 No.07
Gangliosides has the important roles in interactions between cells and in signal transduction to regulate growth and differentiation. Hutchinson-Gilford progeria syndrome (HGPS) is an extremely rare genetic disorder wherein symptoms resembling aspects of aging are manifested at a very early age. Model of LMNA (Lamin A/C) mutation is known as a typical model for HPGS. However, roles of gangliosides in model of LMNA mutation (HGPS) is unclear. Therefore, this study investigated the expression patterns of gangliosides in model of LMNA-mutation. LMNA-mutation cells were primary cultured from spontaneous mouse LMNA-mutation model. Distribution of gangliosides in LMNA-mutation mouse was detected by HPTLC. We successes the primary cultured bone marrow-derived mesencymal stem cells from LMNA-mutation mouse. In addition, gangliosides in several tissues including liver, kidney and brain from LMNA-mutation mouse was various expressed compare with normal tissues. So, gangliosides may be related with LMNA in cell differentiation and proliferation. Therefore, this results suggested that gangliosides act important roles in aging and HGPS.