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Estimation of Corrosion Rates on Outside Surface of Pipes
Satoshi Kibata,Hisayoshi Matsuyama,Shigeyuki Tateno 제어로봇시스템학회 2009 제어로봇시스템학회 국제학술대회 논문집 Vol.2009 No.8
Corrosions of pipes had been disregarded as a subject of maintenance because corrosions in chemical plants progress slowly. However, many chemical plants were built over 30 years ago in Japan. There are many pipes whose outside surfaces is considerably corroded. Accidents which relate to corrosions may occur and lead to serious problems and vast losses. For this reason, the maintenance is greatly needed. Although, each company takes measures to cope with corrosions, successful managerial methods are not established. The purpose of this research is to know corrosion wastages from outside surface of pipes by estimating a corrosion rate in order to select pipes to be checked. To achieve the purpose, corrosion rates based on corrosion cases are estimated by using the database modeling method.
Genetic Screening for Spinocerebellar Ataxia Genes in a Japanese Single-Hospital Cohort
Ryuji Sakakibara,Fuyuki Tateno,Masahiko Kishi,Yohei Tsuyusaki,Yosuke Aiba,Hitoshi Terada,Tsutomu Inaoka,Setsu Sawai,Satoshi Kuwabara,Fumio Nomura 대한파킨슨병및이상운동질환학회 2017 Journal Of Movement Disorders Vol.10 No.3
Objective: Diagnosis of sporadic cerebellar ataxia is a challenge for neurologists. A wide range of potential causes exist, including chronic alcohol use, multiple system atrophy of cerebellar type (MSA-C), and sporadic late cortical cerebellar atrophy. Recently, an autosomal-dominant spinocerebellar ataxia (SCA) mutation was identified in a cohort of patients with non-MSA-C sporadic cerebellar ataxia. The aim of this study is to genetically screen genes involved in SCA in a Japanese single-hospital cohort. Methods: Over an 8-year period, 140 patients with cerebellar ataxia were observed. There were 109 patients with sporadic cerebellar ataxia (no family history for at least four generations, 73 patients with MSA-C, and 36 patients with non-MSA-C sporadic cerebellar ataxia) and 31 patients with familial cerebellar ataxia. We performed gene analysis comprising SCA1, 2, 3, 6, 7, 8, 12, 17, 31, and dentatorubro-pallidoluysian atrophy (DRPLA) in 28 of 31 non-MSA-C sporadic patients who requested the test. Familial patients served as a control. Results: Gene abnormalities were found in 57% of non-MSA-C sporadic cerebellar ataxia cases. Among patients with sporadic cerebellar ataxia, abnormalities in SCA6 were the most common (36%), followed by abnormalities in SCA1 (7.1%), SCA2 (3.6%), SCA3 (3.6%), SCA8 (3.6%), and DRPLA (3.6%). In contrast, gene abnormalities were found in 75% of familial cerebellar ataxia cases, with abnormalities in SCA6 being the most common (29%). For sporadic versus familial cases for those with SCA6 abnormalities, the age of onset was older (69 years vs. 59 years, respectively), and CAG repeat length was shorter (23 vs. 25, respectively) in the former than in the latter (not statistically significant). Conclusion: Autosomal-dominant mutations in SCA genes, particularly in SCA6, are not rare in sporadic cerebellar ataxia. The reason for the frequency of mutations in SCA6 remains unclear; however, the reason may reflect a higher age at onset and variable penetrance of SCA6 mutations.