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Junxiong Lin,Peng Wang,Lei Zhu 한국섬유공학회 2023 Fibers and polymers Vol.24 No.2
Silver nanoparticles (Ag NPs) were synthesized on cotton fabrics through in-situ radical reduction of Ag+with macromolecularalcohol radicals generated from a polymer blend under UV light. This light-induced approach permitted directphotopatterning of Ag particles on the surface of cotton fabric. The cotton fabrics were colored brown by the Ag NPs owingto their surface plasmon resonance characteristics, and the lighter brown or darker brown color could be adjusted by controllingthe light exposure time or Ag+concentration in the synthesizing process. The Ag NPs loaded fabrics were investigatedusing SEM, EDX and XPS techniques. The results showed that Ag+ions were successfully reduced to metallic Ag NPs, andthese particles could be clearly observed by the morphological observation study. The Ag NPs loaded cotton fabric exhibitedsatisfactory washing and rubbing colorfastness. In addition, the Ag NPs on the surface endowed the cotton fabric with goodantibacterial activity and conductive performance.
Jun Zhou,Junxiong Wang 대한기계학회 2022 JOURNAL OF MECHANICAL SCIENCE AND TECHNOLOGY Vol.36 No.2
The number of fault sources is helpful for accurately extracting fault features. The existing source number estimation methods have certain difficulties when realizing the source estimation of mechanical vibration signals and acoustic signals in a strong background noise environment. A variety of the fault source signals are coupled with each other, resulting in that the number of fault sources in the actual working conditions is unknown, and sometimes the number of observation signals is less than the number of fault source signals due to the limitation of sensor installation condition. A novel method for underdetermined source number estimation based on the combination of morphological filtering (MF), adaptive variational modal decomposition (AVMD) and singular value decomposition (SVD), named MF-AVMD-SVD, is proposed. First, MF is used to filter out background noise signals and enhance the impact characteristics of fault signals. Second, AVMD is used to resolve the filtered observation signals to obtain intrinsic mode function (IMF); these IMFs are recombined to obtain the covariance matrix of multidimensional observation signals. Finally, new non-zero eigenvectors are obtained by SVD, and the accurate number of rolling bearing fault source signals is determined according to the maximum value of the adjacent eigenvalue ratio. This method is applied to the simulation signal; the number of four sources can be accurately estimated by the two-channel simulation mixed signal under the condition that the signal to noise ratio (SNR) is -15 dB or even -21 dB. It can also be effectively estimated that the number of vibration sources is three from the vibration signals and acoustic signals of the double-channel rolling bearing composite fault.
Xiong Song,Liangui Nie,Junrong Long,Junxiong Zhao,Xing Liu,Liuyang Wang,Da Liu,Sen Wang,Shengquan Liu,Jun Yang 대한약리학회 2023 The Korean Journal of Physiology & Pharmacology Vol.27 No.1
Hypothyroidism alone can lead to myocardial fibrosis and result in heart failure, but traditional hormone replacement therapy does not improve the fibrotic situation. Hydrogen sulfide (H2S), a new gas signaling molecule, possesses antiinflammatory, antioxidant, and anti-fibrotic capabilities. Whether H2S could improve hypothyroidism-induced myocardial fibrosis are not yet studied. In our study, H2S could decrease collagen deposition in the myocardial tissue of rats caused by hypothyroidism. Furthermore, in hypothyroidism-induced rats, we found that H2S could enhance cystathionine-gamma-lyase (CSE), not cystathionine β-synthase (CBS), protein expressions. Finally, we noticed that H2S could elevate autophagy levels and inhibit the transforming growth factor-β1 (TGF-β1) signal transduction pathway. In conclusion, our experiments not only suggest that H2S could alleviate hypothyroidism- induced myocardial fibrosis by activating autophagy and suppressing TGF-β1/ SMAD family member 2 (Smad 2) signal transduction pathway, but also show that it can be used as a complementary treatment to conventional hormone therapy.