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RISS 인기검색어
- 검색키워드
- 저자 : Guillot, Thomas S (검색결과 2 건)
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High-field Magnetization Study of ErCo2
Guillot Maurice,Oner Yildirhan 한국물리학회 2013 THE JOURNAL OF THE KOREAN PHYSICAL SOCIETY Vol.62 No.10
In this work we present new results on the magnetic properties of the ErCo2 compound for which three magnetic phases (ferrimagnetic, parimagnetic and paramagnetic) were proposed very recently. This study is based on the investigation of the magnetization under continuous high magnetic fields (up to 330 kOe at 4.2 K and up to 230 kOe in the 4.2 - 300 K respectively). At low temperatures the stability of the ferrimagnetic structure is shown although the saturation was never found. Special attention is paid to the 35 - 100 K temperature range where the transitions between the different magnetic phases occur confirming of the paramagnetic orderings. The transition temperature Tc from the ferrimagnetic to the paramagnetic phase increases from ≈ 34 to 62 K with the applied field. For the values of H smaller than 100 kOe. At H values greater than this Tc remains constant at (62 ± 5) K. The atomic magnetic moment of Co is independent of the magnetic structure and in the order of 1µB atom−1.
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Synapsins differentially control dopamine and serotonin release.
Kile, Brian M,Guillot, Thomas S,Venton, B Jill,Wetsel, William C,Augustine, George J,Wightman, R Mark The Society 2010 The Journal of neuroscience Vol.30 No.29
<P>Synapsins are a family of synaptic vesicle proteins that are important for neurotransmitter release. Here we have used triple knock-out (TKO) mice lacking all three synapsin genes to determine the roles of synapsins in the release of two monoamine neurotransmitters, dopamine and serotonin. Serotonin release evoked by electrical stimulation was identical in substantia nigra pars reticulata slices prepared from TKO and wild-type mice. In contrast, release of dopamine in response to electrical stimulation was approximately doubled in striatum of TKO mice, both in vivo and in striatal slices, in comparison to wild-type controls. This was due to loss of synapsin III, because deletion of synapsin III alone was sufficient to increase dopamine release. Deletion of synapsins also increased the sensitivity of dopamine release to extracellular calcium ions. Although cocaine did not affect the release of serotonin from nigral tissue, this drug did enhance dopamine release. Cocaine-induced facilitation of dopamine release was a function of external calcium, an effect that was reduced in TKO mice. We conclude that synapsins play different roles in the control of release of dopamine and serotonin, with release of dopamine being negatively regulated by synapsins, specifically synapsin III, while serotonin release appears to be relatively independent of synapsins. These results provide further support for the concept that synapsin function in presynaptic terminals varies according to the neurotransmitter being released.</P>
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