Progress in medical virology . 28

목차 (Table of Contents)

• CONTENTS
• Persistent, Slow and Latent Viral Infections / Stroop, W. G. ; Baringer, J. R.[San Francisco, Calif] = 1
• Ⅰ. Introduction = 1
• Ⅱ. General Mechanisms of Persistence = 3
• Ⅲ. Persistent Infections Due to Conventional Agents = 3
• A. Progressive Multifocal Leukoencephalopathy(PML) = 3
• B. Subacute Sclerosing Panencephalitis(SSPE) = 7
• C. Progressive Rubella Panencephafitis(PRP) = 9
• D. Murine Lymphocytic Choriomeningitis(LCM) Virus Infection = 10
• E. Lactate Dehydrogenase-Elevating Virus(LDV) Infection of Mice = 12
• F. Theiler's Virus Infection of Mice = 14
• G. Aleutian Mink Disease(AMD) = 17
• Ⅳ. Persistent Latent Viral Infections = 19
• A. Equine Infectious Anemia(FIA) = 19
• B. Visna Virus Infections = 22
• C. Herpesvirus Infections = 26
• Ⅴ. Slow Virus Infections Caused by Unconventional Agents = 29
• Ⅵ. Defective Interfering Particles and Persistent Infections = 30
• Ⅶ. The Role of Viral Persistence in Nature = 31
• References = 32
• Cytomegalovirus Infection in Transplant Patients / Betts, R. F[Rochester, N. Y.] = 44
• Ⅰ. Introduction = 44
• Ⅱ. Epidemiology of CMV Infection = 45
• Ⅲ. Types of Transplants = 47
• Ⅳ. Pathogenesis of CMV Infection = 48
• A. Source of CMV in Allograft Infection = 48
• B. Reactivation of Latent Virus = 50
• C. Types of Infection in Different Pairings = 50
• Ⅴ. Clinical Manifestations of Infection with Cytomegalovirus Following Transplantation = 52
• A. Febrile Illness Due to CMV = 52
• B. Bacterial and Fungal Superinfection = 55
• C. Allograft Failure = 55
• Ⅵ. Special Problems = 57
• Ⅶ. Prevention and Treatment = 58
• A. Donor Selection = 58
• B. Vaccine = 58
• C. Passive Immunity = 59
• D. Antiviral Chemotherapy = 59
• Ⅷ. Summary = 59
• References = 60
• Measles and SSPE Viruses : Similarities and Differences / Wechsler, S. L.[Cincinnati, Ohio] ; Meissner, H. C.[Boston, Mass] = 65
• Ⅰ. Introduction = 66
• Ⅱ. The Diseases = 68
• Ⅲ. The Virion = 69
• A. Virus Classification = 69
• B. Virion Structure = 69
• C. Measles and SSPE RNA = 70
• D. Virion Proteins = 71
• 1. Location of Virion Proteins = 72
• 2. Protein Functions = 72
• Ⅳ. Intracellular Viral Proteins = 73
• A. Measles Proteins = 73
• B. SSPE Proteins = 74
• C. Relative Amounts of Intracellular Viral Proteins = 74
• Ⅴ. Viral Replication = 75
• A. RNA and Proteins = 75
• B. Viral Maturation = 76
• C. The Role of the Nucleus in Measles Virus Infections = 77
• Ⅵ. Growth Characteristics = 78
• Ⅶ. Animal Model Systems = 78
• Ⅷ. Persistent Infections with Measles Virus = 80
• Ⅸ. Immunology = 82
• A. Neutralization = 82
• B. Antigenic Cross-Reactivity = 82
• C. Host Humoral Immune Response = 82
• Ⅹ. Mechanism(s) of Measles Virus Persistence = 84
• XI. Conclusions = 85
• References = 85
• Korean Hemorrhagic Fever / Lee, H. W.[Seoul(with 1 color plate)] = 96
• Ⅰ. Introduction = 97
• Ⅱ. Etiology = 99
• A. Background = 99
• B. Isolation of Hantaan Vims, the Etiologic Agent of KHF = 99
• C. Growth of Virus and Animal Susceptibility = 100
• D. Properties of Hantaan Vims = 101
• Ⅲ. Immunology = 102
• Ⅳ. Laboratory Diagnosis = 103
• A. Specific Serologic Diagnosis = 103
• B. Isolation of the Virus = 104
• Ⅴ. Epidemiology of Korean Hemorrhagic Fever = 104
• A. Epidemiologic Types = 105
• B. Seasonal Incidence = 105
• C. Age and Sex = 107
• D. Animal Reservoir Host = 107
• E. Virus Transmission in the Reservoir Animal = 107
• Ⅵ. Clinical Course = 108
• A. Febrile Phase = 108
• B. Hypotensive Phase = 109
• C. Oliguric Phase = 109
• D. Diuretic Phase = 110
• E. Convalescent Phase = 110
• References = 110
• Cell Growth Transformation by Herpes Simplex Virus / Knipe, D. M.[Boston, Mass] = 114
• Ⅰ. Introduction = 115
• Ⅱ. Herpes Simplex Virus = 115
• A. Structure of the HSV Genome = 116
• B. Replication of HSV in Cell Culture = 117
• C. Growth Transformation of Cells by HSV Virions = 117
• D. Growth Transformation by Transfection of HSV DNA Fragments = 120
• E. Biochemical Transformation = 123
• F. Relation between Biochemical and Morphological Transformation = 124
• G. Viral Antigens in Transformed and Tumor Cells = 125
• H. Viral Nucleic Acids in Transformed and Tumor Cells = 127
• 1. Effects of HSV on the Host Cell = 128
• 1. Chromosome Damage = 128
• 2. Stimulation of Cellular DNA Replication = 129
• 3. Alterations of the Host Cell Cytoskeleton = 129
• 4. Activation of RNA Tumor Virus Genomes = 129
• J. Summary on HSV = 130
• Ⅲ. Epstein-Barr Virus = 130
• Ⅳ. Human Cytomegalovirus = 133
• Ⅴ. Equine Herpesviruses = 133
• Ⅵ. Simian Herpesviruses = 134
• Ⅶ. General Summary = 135
• Addendum = 136
• Acknowledgments = 136
• References = 137
• Epstein-Barr-Virus Nuclear Antigen / Vonka, V. ; Hirsch, I.[Prague] = 145
• Ⅰ. Introduction = 146
• Ⅱ. Early Studies : Discovery of the Soluble Antigen(SA) of EB Virus = 146
• Ⅲ. Discovery of EB-Virus Nuclear Antigen(EBNA) and Evidence for Its Identity with SA = 147
• Ⅳ. Significance of the Presence of EBNA Antibody = 149
• A. Delayed Appearance of EBNA Antibody in Primary EB Virus Infection = 150
• B. EBNA Antibody in Patients with Various Malignant Diseases = 152
• C. EBNA Antibody in Various Ethnic Groups = 154
• Ⅴ. Relationship between EBNA and Nuclear Antigens of Other Herpesviruses = 155
• Ⅵ. Biochemical Characterization of EBNA = 156
• A. Purification of EBNA = 156
• B. Interaction of EBNA with DNA = 159
• 1. EBNA Binding to Acid-Fixed Nuclei(AFNB) = 159
• 2. Problems Involved in Establishing Specificity of the Reaction = 159
• 3. Interactions of EBNA-Anti-EBNA-Antibody Complexes with DNA and EBNA-DNA Complexes with Anti-EBNA Antibody = 161
• Ⅶ. Biology of EBNA = 161
• A. Origin of EBNA = 161
• B. EBNA in Primarily Infected Cells = 162
• C. Regulation of EBNA Synthesis = 164
• D. Function of EBNA = 165
• Ⅷ. Summary = 167
• Addendum = 168
• Acknowledgments = 169
• References = 169
• Benefits, Risks and Costs of Viral Vaccines / Koplan, J. P. ; Axnick, N. W.[Atlanta, Ga] = 180
• Introduction = 180
• Smallpox = 182
• Polio = 183
• Measles = 185
• Rubella = 186
• Mumps = 187
• Influenza = 188
• Summary = 189
• References = 190
• A Look at the P4 Virus Containment Laboratory / van der Green, G.[Antwerp] ; Trexler, P. C.[Guildford] = 192
• Ⅰ. Introduction = 192
• Ⅱ. What Are Class 4 Viruses? = 193
• Ⅲ. Methods of Physical Containment = 196
• A. Primary Containment = 196
• 1. Isolators with Rigid Walls = 197
• 2. Isolators with Flexible Walls = 197
• 3. One-Piece Plastic Positive-Pressure Suit = 199
• B. Secondary Containment = 200
• Ⅳ. Discussion = 202
• Ⅴ. Summary = 206
• References = 206
• Taxonomy and Nomenclature of Viruses, 1982 / Melnick, J. L.[Houston, Tex] = 208
• Introduction = 208
• Viruses with a DNA Genome = 209
• Viruses with an RNA Genome = 212
• Hepatitis B Viruses = 216
• Virus Species = 217
• Nomenclature of Influenza Virus Subtypes = 218
• Nomenclature of Poliovirus Strains = 220
• Comment = 220
• References = 221
• Subject Index = 222
• Cumulative Author Index, vol. 25-28 = 226
• Cumulative Title Index. vol. 25-28 = 231