<P>The mechanistic role of a glycine-rich RNA-binding protein designated atRZ-1a that contributes to enhance cold tolerance in <I>Arabidopsis</I> was investigated. Overexpression of atRZ-1a did not affect the expression of various co...
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https://www.riss.kr/link?id=A107625656
2006
-
SCOPUS,SCIE
학술저널
793-798(6쪽)
0
상세조회0
다운로드다국어 초록 (Multilingual Abstract)
<P>The mechanistic role of a glycine-rich RNA-binding protein designated atRZ-1a that contributes to enhance cold tolerance in <I>Arabidopsis</I> was investigated. Overexpression of atRZ-1a did not affect the expression of various co...
<P>The mechanistic role of a glycine-rich RNA-binding protein designated atRZ-1a that contributes to enhance cold tolerance in <I>Arabidopsis</I> was investigated. Overexpression of atRZ-1a did not affect the expression of various cold-responsive genes such as COR6.6, COR15a, COR47, RD29A, RD29B and LTI29. Proteome analyses revealed that overexpression of atRZ-1a modulated the expression of several stress-responsive genes, and the transcript levels and RNA stability of these target genes were not affected by atRZ-1a. atRZ-1a successfully complements the cold sensitivity of <I>Escherichia coli</I> lacking four cold shock proteins. These results strongly suggest that atRZ-1a plays a role as an RNA chaperone during the cold adaptation process.</P>