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Nitric oxide suppresses L-type calcium currents in basilar artery smooth muscle cells in rabbits

한글로보기

https://www.riss.kr/link?id=A107672692

저자

Sharma, Naveen ; Bhattarai, Janardhan Prasad ; Hwang, Pyoung Han ; Han, Seong Kyu
발행기관
Informa UK (TaylorFrancis)
학술지명
Neurological research
권호사항

Vol.35 No.4 [2013]
발행연도
2013
작성언어
-
등재정보
SCOPUS,SCIE
자료형태
학술저널
수록면

424-428(5쪽)
제공처
ScienceON

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다국어 초록 (Multilingual Abstract)

Nitric oxide (NO) is well known to be a vasodilator, and NO donor compounds are currently used for treating vasospasm following subarachnoid hemorrhage. However, the action mechanism of cerebral vascular relaxation is not yet clear. L-type calcium channels have been determined to play an essential role in smooth muscle contraction. To investigate the role of L-type calcium channels in NO-induced relaxation of basilar smooth muscle cells, we examined the effect of the NO donor, sodium nitroprusside (SNP) on calcium (Ca2+) currents using smooth muscle cells isolated from a rabbit basilar artery.

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Nitric oxide (NO) is well known to be a vasodilator, and NO donor compounds are currently used for treating vasospasm following subarachnoid hemorrhage. However, the action mechanism of cerebral vascular relaxation is not yet clear. L-type ca...

Nitric oxide (NO) is well known to be a vasodilator, and NO donor compounds are currently used for treating vasospasm following subarachnoid hemorrhage. However, the action mechanism of cerebral vascular relaxation is not yet clear. L-type calcium channels have been determined to play an essential role in smooth muscle contraction. To investigate the role of L-type calcium channels in NO-induced relaxation of basilar smooth muscle cells, we examined the effect of the NO donor, sodium nitroprusside (SNP) on calcium (Ca2+) currents using smooth muscle cells isolated from a rabbit basilar artery.