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      다량 토혈 후 발생한 뇌경색으로 인하여 간질증첩증을 보인 간성 뇌증 1예 = A Case of Hepatic Encephalopathy with Status Epilepticus due to Cerebral Infarction after Massive Hematemesis

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      https://www.riss.kr/link?id=A76517401

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      Status epilepticus (SE) has been described as a rare manifestation of hepatic encephalopathy. Massive bleeding and subsequent anemia may precipitate atherothrombotic cerebral infarction. Stroke is one of the possible causes of SE. A 69-year-o1d male with decompensated alcoholic liver cirrhosis presented with altered mental state and generalized convulsive SE after massive hematemesis due to varix bleeding. The hemoglobin level was 5.5 mg/dL and blood ammonia level was more than twice the normal level. The seizure was resolved after prompt and aggressive treatment for SE. The blood ammonia level was normalized after treatment for hepatic encephalopathy, but the patient' s mental state was not recovered. Brain MRI revealed multiple ischemic lesions in the both cerebral hemisphere. We present the patient as SE after acute ischemic stroke associated with anemia in hepatic encephalopathy.
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      Status epilepticus (SE) has been described as a rare manifestation of hepatic encephalopathy. Massive bleeding and subsequent anemia may precipitate atherothrombotic cerebral infarction. Stroke is one of the possible causes of SE. A 69-year-o1d male w...

      Status epilepticus (SE) has been described as a rare manifestation of hepatic encephalopathy. Massive bleeding and subsequent anemia may precipitate atherothrombotic cerebral infarction. Stroke is one of the possible causes of SE. A 69-year-o1d male with decompensated alcoholic liver cirrhosis presented with altered mental state and generalized convulsive SE after massive hematemesis due to varix bleeding. The hemoglobin level was 5.5 mg/dL and blood ammonia level was more than twice the normal level. The seizure was resolved after prompt and aggressive treatment for SE. The blood ammonia level was normalized after treatment for hepatic encephalopathy, but the patient' s mental state was not recovered. Brain MRI revealed multiple ischemic lesions in the both cerebral hemisphere. We present the patient as SE after acute ischemic stroke associated with anemia in hepatic encephalopathy.

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