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KISS
Effects of various mutagens on replicon initiation and chain elongation of DNA replication was studied by pulse-labeling with (³H) - thymidine and sedimentation analysis of DNA. When treated with those mutagens to mammalian cells, each cell line show...
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RISS 인기검색어
Replication and Postreplication Reapir in Mammalian Cells Exposed to UV - Light and Alkylating Agents = Replication and Postreplication Reapir in Mammalian Cells Exposed to UV - Light and Alkylating Agents
한글로보기https://www.riss.kr/link?id=A3087188
- 저자
- 발행기관
- 학술지명
- 권호사항
-
발행연도
1983
-
작성언어
-
-
KDC
400
-
등재정보
SCOPUS,KCI등재,SCIE
-
자료형태
학술저널
- 발행기관 URL
-
수록면
102-102(1쪽)
- 제공처
- 소장기관
-
0
상세조회 -
0
다운로드
부가정보
다국어 초록 (Multilingual Abstract)
Effects of various mutagens on replicon initiation and chain elongation of DNA replication was studied by pulse-labeling with (³H) - thymidine and sedimentation analysis of DNA. When treated with those mutagens to mammalian cells, each cell line showed the reduced rate of DNA synthesis and the interrupted DNA replication in dose dependent manner. At low doses, the inhibition of replicon initiation was primarily caused and at high fluences the inhibitory effect appeared in chain elongation and termination of DNA replication. Using the method of `Pulse-Chase` we could also observed the postreplication repair, indicating that the nascent DNA growth normally occurred, although still damaged sites remained on parental DNA during DNA replication.
Effects of various mutagens on replicon initiation and chain elongation of DNA replication was studied by pulse-labeling with (³H) - thymidine and sedimentation analysis of DNA. When treated with those mutagens to mammalian cells, each cell line showed the reduced rate of DNA synthesis and the interrupted DNA replication in dose dependent manner. At low doses, the inhibition of replicon initiation was primarily caused and at high fluences the inhibitory effect appeared in chain elongation and termination of DNA replication. Using the method of `Pulse-Chase` we could also observed the postreplication repair, indicating that the nascent DNA growth normally occurred, although still damaged sites remained on parental DNA during DNA replication.
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