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      側腦室內 Substance P가 家兎 腎臟機能에 미치는 影響 = Effect of Intraventricular Substance P on the Renal Function of Rabbit

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      https://www.riss.kr/link?id=A19594902

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      In this study, the effects of substance P, administered intracranioventricularly, on the renal function of the rabbit were investigated.
      1) Intraventricular substance P(0.01, 0.1 and 10ng) elicited antidiuresis, and decreased the renal plasma flow, glomerular filtration rate and urinary excreted amounts of sodium and potassium, but did not influence to free water clearance.
      2) Correlation coefficient between changes of urine volume and of renal plasma flow, glomerular filtration rate or urinary sodium excretion was statistically significant.
      3) Systemic blood pressure was not affected in a dose of 0.01ng, but increased in 0.1ng(10mmHg) or 10ng(20∼25mmHg).
      4) Antidiuretic effects of intraventricular substance P were partially inhibited by the renal nerve degeneration.
      From the above results, it is suggested that the antidiuretic effect of intraventricular substance P is dae to renal hemodynamic changes, partially induced by renal sympathetic nerve activation.
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      In this study, the effects of substance P, administered intracranioventricularly, on the renal function of the rabbit were investigated. 1) Intraventricular substance P(0.01, 0.1 and 10ng) elicited antidiuresis, and decreased the renal plasma flow, g...

      In this study, the effects of substance P, administered intracranioventricularly, on the renal function of the rabbit were investigated.
      1) Intraventricular substance P(0.01, 0.1 and 10ng) elicited antidiuresis, and decreased the renal plasma flow, glomerular filtration rate and urinary excreted amounts of sodium and potassium, but did not influence to free water clearance.
      2) Correlation coefficient between changes of urine volume and of renal plasma flow, glomerular filtration rate or urinary sodium excretion was statistically significant.
      3) Systemic blood pressure was not affected in a dose of 0.01ng, but increased in 0.1ng(10mmHg) or 10ng(20∼25mmHg).
      4) Antidiuretic effects of intraventricular substance P were partially inhibited by the renal nerve degeneration.
      From the above results, it is suggested that the antidiuretic effect of intraventricular substance P is dae to renal hemodynamic changes, partially induced by renal sympathetic nerve activation.

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