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      KCI등재 SCOPUS

      Fine Structural Study on Compensation Effect of Red Ginseng against Methylmercury Injury to Brain and Gill of Fightingfish = 메틸수은으로 손상된 버들붕어 뇌와 아가미에 대한 홍삼의 보상효능에 관한 미세구조적 연구

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      https://www.riss.kr/link?id=A100571091

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      다국어 초록 (Multilingual Abstract)

      The toxic effects of methylmercury on the ultrastructures of the brain and gill tissues of fightingfish and compensative effects of red ginseng were investigated by means of electron microscopy. The brain neuron of methylmercury exposure group showed dilatation of dendrite and axon, numerical decrease of ribosomes, partial loss of nucleoplasm and cytoplasm and considerable swelling of mitochondria as compared with the normal neuron. And necrotic cell with ruptured nucleus and vacuolated mitochondria was noticeable. While, slight swelling of mitochondria, some dilation of dendrite and axon and numerical increase of ribosome occurred in the neuron of methylmercury-red ginseng treatment group as compared with the methylmercury exposure group. In the gill lamella of methylmercury exposure group, collapse of pillar cells and arms, dilated epithelial cell and thickened membrane were observed. While, in the gill lamella of methylmercury-red ginseng treatment group, arms were slightly disintegrated and basement membrane was some thickend as compared with the methylmercury exposure group. From the above results, it is concluded that red ginseng has detoxication effect on methylmercury toxicity and so takes compensative effect on injured tissues caused by methylmercury intoxication.
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      The toxic effects of methylmercury on the ultrastructures of the brain and gill tissues of fightingfish and compensative effects of red ginseng were investigated by means of electron microscopy. The brain neuron of methylmercury exposure group showed ...

      The toxic effects of methylmercury on the ultrastructures of the brain and gill tissues of fightingfish and compensative effects of red ginseng were investigated by means of electron microscopy. The brain neuron of methylmercury exposure group showed dilatation of dendrite and axon, numerical decrease of ribosomes, partial loss of nucleoplasm and cytoplasm and considerable swelling of mitochondria as compared with the normal neuron. And necrotic cell with ruptured nucleus and vacuolated mitochondria was noticeable. While, slight swelling of mitochondria, some dilation of dendrite and axon and numerical increase of ribosome occurred in the neuron of methylmercury-red ginseng treatment group as compared with the methylmercury exposure group. In the gill lamella of methylmercury exposure group, collapse of pillar cells and arms, dilated epithelial cell and thickened membrane were observed. While, in the gill lamella of methylmercury-red ginseng treatment group, arms were slightly disintegrated and basement membrane was some thickend as compared with the methylmercury exposure group. From the above results, it is concluded that red ginseng has detoxication effect on methylmercury toxicity and so takes compensative effect on injured tissues caused by methylmercury intoxication.

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