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KCIWe report the case of a 70-year-old female who developed acute transverse myelitis 1 month after herpes zoster infection and was diagnosed with aquaporin-4 (AQP4) antibody-positive neuromyelitis optica spectrum disorder. The patient was treated with h...
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대상포진-유발성 항아쿠아포린-4 항체 양성 시신경척수염범주질환: 증례 보고 및 문헌 고찰 = Aquaporin-4 Antibody-Positive Neuromyelitis Optica Spectrum Disorder Triggered by Herpes Zoster: A Case Report and Literature Review
한글로보기https://www.riss.kr/link?id=A110105382
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저자
Daecheol Moon (Department of Medicine, Graduate School, Kyung Hee University) ; Dong Kyu Kim (Department of Neurology, Kyung Hee University Hospital, Kyung Hee University College of Medicine) ; Seong-il Oh (Department of Neurology, Kyung Hee University Hospital, Kyung Hee University College of Medicine)
- 발행기관
- 학술지명
- 권호사항
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발행연도
2025
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작성언어
English
- 주제어
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등재정보
KCI등재후보
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자료형태
학술저널
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수록면
60-65(6쪽)
- 제공처
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0
상세조회 -
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부가정보
다국어 초록 (Multilingual Abstract)
We report the case of a 70-year-old female who developed acute transverse myelitis 1 month after herpes zoster infection and was diagnosed with aquaporin-4 (AQP4) antibody-positive neuromyelitis optica spectrum disorder. The patient was treated with high-dose corticosteroid and intravenous immunoglobulin therapy and her serum AQP4 antibody seroconverted to negative 3 months later. This case suggests that varicella-zoster virus reactivation may trigger transient AQP4 autoimmunity and highlights the clinical significance of post-treatment seroreversion in distinguishing infection-triggered from idiopathic neuromyelitis optica spectrum disorder.
We report the case of a 70-year-old female who developed acute transverse myelitis 1 month after herpes zoster infection and was diagnosed with aquaporin-4 (AQP4) antibody-positive neuromyelitis optica spectrum disorder. The patient was treated with high-dose corticosteroid and intravenous immunoglobulin therapy and her serum AQP4 antibody seroconverted to negative 3 months later. This case suggests that varicella-zoster virus reactivation may trigger transient AQP4 autoimmunity and highlights the clinical significance of post-treatment seroreversion in distinguishing infection-triggered from idiopathic neuromyelitis optica spectrum disorder.
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