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      KCI등재후보 SCOPUS

      복부대동맥류 혈관 내 스텐트삽입술 후 복부대동맥류 확장의 치료 = Current Management of the Growing Sac after Endovascular Aneurysm Repair

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      https://www.riss.kr/link?id=A104609578

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      다국어 초록 (Multilingual Abstract)

      Endovascular aneurysm repair (EVAR) has become the preferred treatment for abdominal aortic aneurysm. EVAR results in sac exclusion and subsequent sac depressurization, which prevents aneurysm rupture and aneurysm related death. Its benefits have led to a widespread adaptation. However, EVAR has transformed abdominal aortic aneurysm from an ailment with the definitive cure (open surgical repair) into a chronic disease process with the need for a close, life-long surveillance and increased potential for secondary interventions. Following EVAR, endoleak can occur, and incidence varies widely ranging from 15% to 52%. Endoleak can lead to sac growth and concern for rupture. Treatment depends on the leak type. Type I and III endoleaks should be treated. There is general consensus that type II endoleaks can be monitored except in cases of sac enlargement >5 mm. Treatment of type V endoleak, or "endotension" is controversial. These endoleaks have been associated with the first generation high porosity Gore Excluder stent graft. In these cases, relining the stent graft with resultant halt of sac growth has been descried. With the next generation of low porosity Gore Excluder, endotension is a less commonplace. Nonetheless, sac growth in the absence of endoleak can occur with any stent graft system, and surgical conversion may be warranted. Needless to say, this decision is made on an individual case basis. Management of sac growth is varied and can generally be categorized by approach (transarterial, translumbar, transcaval, and laparoscopic) or by method of repair (embolization, proximal/distal extension, endostaple, and surgical conversion). Extension pieces are used to seal type I endoleaks wheng there is adequate neck length to extend the seal. Use of fenestrated or "chimney" grafts can extend coverage to the pararenal aorta. When there is insufficient additional neck to obtain the seal, a Palmaz stent or noncompliant balloon can be considered. Recent approval of an endovascular stapler is a novel method for treating type I endoleaks. Type II endoleak treatment is conceptually similar to the treatment of a vascular malformation. An attempt should be made to embolize the inflow and outflow vessels, as well as the endoleak nidus. Laparoscopic branch vessel ligation or sac plication has been described. Finally, rather than responding to the endoleaks that occur, a strategy of preemptive action to prevent their appearance should be considered, though this is not widely practiced. Aneurysm sac "thrombization" involves embolization of the sac with a combination of glue and coil during the time of initial stent graft implantation. This may decrease the subsequent development of endoleak. Preoperative ligation or embolization of a patent inferior mesenteric artery is performed at some centers. Finally, the aforementioned endostapler can be used to prevent future endoleak and graft migration, particularly in hostile neck anatomy.
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      Endovascular aneurysm repair (EVAR) has become the preferred treatment for abdominal aortic aneurysm. EVAR results in sac exclusion and subsequent sac depressurization, which prevents aneurysm rupture and aneurysm related death. Its benefits have led ...

      Endovascular aneurysm repair (EVAR) has become the preferred treatment for abdominal aortic aneurysm. EVAR results in sac exclusion and subsequent sac depressurization, which prevents aneurysm rupture and aneurysm related death. Its benefits have led to a widespread adaptation. However, EVAR has transformed abdominal aortic aneurysm from an ailment with the definitive cure (open surgical repair) into a chronic disease process with the need for a close, life-long surveillance and increased potential for secondary interventions. Following EVAR, endoleak can occur, and incidence varies widely ranging from 15% to 52%. Endoleak can lead to sac growth and concern for rupture. Treatment depends on the leak type. Type I and III endoleaks should be treated. There is general consensus that type II endoleaks can be monitored except in cases of sac enlargement >5 mm. Treatment of type V endoleak, or "endotension" is controversial. These endoleaks have been associated with the first generation high porosity Gore Excluder stent graft. In these cases, relining the stent graft with resultant halt of sac growth has been descried. With the next generation of low porosity Gore Excluder, endotension is a less commonplace. Nonetheless, sac growth in the absence of endoleak can occur with any stent graft system, and surgical conversion may be warranted. Needless to say, this decision is made on an individual case basis. Management of sac growth is varied and can generally be categorized by approach (transarterial, translumbar, transcaval, and laparoscopic) or by method of repair (embolization, proximal/distal extension, endostaple, and surgical conversion). Extension pieces are used to seal type I endoleaks wheng there is adequate neck length to extend the seal. Use of fenestrated or "chimney" grafts can extend coverage to the pararenal aorta. When there is insufficient additional neck to obtain the seal, a Palmaz stent or noncompliant balloon can be considered. Recent approval of an endovascular stapler is a novel method for treating type I endoleaks. Type II endoleak treatment is conceptually similar to the treatment of a vascular malformation. An attempt should be made to embolize the inflow and outflow vessels, as well as the endoleak nidus. Laparoscopic branch vessel ligation or sac plication has been described. Finally, rather than responding to the endoleaks that occur, a strategy of preemptive action to prevent their appearance should be considered, though this is not widely practiced. Aneurysm sac "thrombization" involves embolization of the sac with a combination of glue and coil during the time of initial stent graft implantation. This may decrease the subsequent development of endoleak. Preoperative ligation or embolization of a patent inferior mesenteric artery is performed at some centers. Finally, the aforementioned endostapler can be used to prevent future endoleak and graft migration, particularly in hostile neck anatomy.

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      참고문헌 (Reference)

      1 Fairman RM, "Zenith Investigators. Factors predictive of early or late aneurysm sac size change following endovascular repair" 43 : 649-656, 2006

      2 Baum RA, "Treatment of type 2 endoleaks after endovascular repair of abdominal aortic aneurysms: comparison of transarterial and translumbar techniques" 35 : 23-29, 2002

      3 Scali ST, "Transcaval embolization as an alternative technique for the treatment of type II endoleak after endovascular aortic aneurysm repair" 57 : 869-874, 2013

      4 Chaikof EL, "The care of patients with an abdominalaortic aneurysm: the Society for Vascular Surgery practice guidelines" 50 (50): S2-49, 2009

      5 Piazza M, "Role of aneurysm sac embolization during endovascular aneurysm repair in the prevention of type II endoleak-related complications" 57 : 934-941, 2013

      6 Schanzer A, "Predictors of abdominal aortic aneurysm sac enlargement after endovascular repair" 123 : 2848-2855, 2011

      7 Aziz A, "Outcomes of percutaneous endovascular intervention for type II endoleak with aneurysm expansion" 55 : 1263-1267, 2012

      8 Nabi D, "Open surgical repair after failed endovascular aneurysm repair: is endograft removal necessary?" 50 : 714-721, 2009

      9 Ward TJ, "Nowakowski FS, Ellozy SH, et al. Preoperative inferior mesenteric artery embolization before endovascular aneurysm repair: decreased incidence of type II endoleak and aneurysm sac enlargement with 24-month follow-up" 24 : 49-55, 2013

      10 Hogg ME, "Long-term sac behavior after endovascular abdominal aortic aneurysm repair with the Excluder lowpermeability endoprosthesis" 53 : 1178-1183, 2011

      1 Fairman RM, "Zenith Investigators. Factors predictive of early or late aneurysm sac size change following endovascular repair" 43 : 649-656, 2006

      2 Baum RA, "Treatment of type 2 endoleaks after endovascular repair of abdominal aortic aneurysms: comparison of transarterial and translumbar techniques" 35 : 23-29, 2002

      3 Scali ST, "Transcaval embolization as an alternative technique for the treatment of type II endoleak after endovascular aortic aneurysm repair" 57 : 869-874, 2013

      4 Chaikof EL, "The care of patients with an abdominalaortic aneurysm: the Society for Vascular Surgery practice guidelines" 50 (50): S2-49, 2009

      5 Piazza M, "Role of aneurysm sac embolization during endovascular aneurysm repair in the prevention of type II endoleak-related complications" 57 : 934-941, 2013

      6 Schanzer A, "Predictors of abdominal aortic aneurysm sac enlargement after endovascular repair" 123 : 2848-2855, 2011

      7 Aziz A, "Outcomes of percutaneous endovascular intervention for type II endoleak with aneurysm expansion" 55 : 1263-1267, 2012

      8 Nabi D, "Open surgical repair after failed endovascular aneurysm repair: is endograft removal necessary?" 50 : 714-721, 2009

      9 Ward TJ, "Nowakowski FS, Ellozy SH, et al. Preoperative inferior mesenteric artery embolization before endovascular aneurysm repair: decreased incidence of type II endoleak and aneurysm sac enlargement with 24-month follow-up" 24 : 49-55, 2013

      10 Hogg ME, "Long-term sac behavior after endovascular abdominal aortic aneurysm repair with the Excluder lowpermeability endoprosthesis" 53 : 1178-1183, 2011

      11 Mehta M, "Long-term outcomes of secondary procedures after endovascular aneurysm repair" 52 : 1442-1449, 2010

      12 Sarac TP, "Long-term follow-up of type II endoleak embolization reveals the need for close surveillance" 55 : 33-40, 2012

      13 Kelso RL, "Late conversion of aortic stent grafts" 49 : 589-595, 2009

      14 Donayre CE, "Initial clinical experience with a sacanchoring endoprosthesis for aortic aneurysm repair" 53 : 574-582, 2011

      15 Uthoff H, "Geisbusch P. Direct percutaneous sac injection for postoperative endoleak treatment after endovascular aortic aneurysm repair" 56 : 965-972, 2012

      16 Schermerhorn ML, "Endovascular vs. open repair of abdominal aortic aneurysms in the Medicare population" 358 : 464-474, 2008

      17 United Kingdom EVAR Trial Investigators, "Endovascular versus open repair of abdominal aortic aneurysm" 362 : 1863-1871, 2010

      18 Grisafi JL, "Endoluminal treatment of type IA endoleak with Onyx" 52 : 1346-1349, 2010

      19 Gelfand DV, "Clinical significance of type II endoleak after endovascular repair of abdominal aortic aneurysm" 20 : 69-74, 2006

      20 Koole D, "Annual rupture risk of abdominal aortic aneurysm enlargement without detectable endoleak after endovascular abdominal aortic repair" 54 : 1614-1622, 2011

      21 Schlosser FJ, "Aneurysm rupture after EVAR: can the ultimate failure be predicted?" 37 : 15-22, 2009

      22 Jimenez JC, "Acute and chronic open conversion after endovascular aortic aneurysm repair: a 14-year review" 46 : 642-647, 2007

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      학술지 이력

      학술지 이력
      연월일 이력구분 이력상세 등재구분
      2027 평가예정 재인증평가 신청대상 (재인증)
      2021-01-01 평가 등재학술지 유지 (재인증) KCI등재
      2018-01-01 평가 등재학술지 유지 (등재유지) KCI등재
      2015-01-01 평가 등재학술지 선정 (계속평가) KCI등재
      2014-07-11 학술지명변경 한글명 : 대한혈관외과학회지 -> Vascular Specialist International KCI등재후보
      2014-02-18 학술지명변경 외국어명 : Korean Journal of Vascular and Endovascular Surgery -> Vascular specialist international KCI등재후보
      2013-01-01 평가 등재후보 1차 FAIL (등재후보1차) KCI등재후보
      2011-01-01 평가 등재후보학술지 선정 (신규평가) KCI등재후보
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      기준연도 WOS-KCI 통합IF(2년) KCIF(2년) KCIF(3년)
      2016 0.04 0.04 0.04
      KCIF(4년) KCIF(5년) 중심성지수(3년) 즉시성지수
      0.03 0.03 0.289 0
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