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The Use of Monolithic Refractories and Microwave Drying for RH Steelmaking Vessels
Kayama, Tsuneo,Hanagiri, Seiji,Sukenari, Shiro The Korean Ceramic Society 2000 The Korean journal of ceramics Vol.6 No.2
Monolithic refractory technology has been developed for RH vessels, with the purpose of reducing the total refractory cost. The technology includes the use of an improved monolithic refractory and microwave drying. The improved monolithic refractory was an alumina-spinel composition, of the type used in steel ladles, to which fine alumina was added to increase the density and corrosion resistance. The microwave drying method, previously developed and used to dry the monolithic lining in steel ladles, was modified for use in drying the dense, 500mm thick lining in RH vessels. This work has resulted in significant cost savings.
Regulation of Intestinal Homeostasis by Innate Immune Cells
Kayama, Hisako,Nishimura, Junichi,Takeda, Kiyoshi The Korean Association of Immunobiologists 2013 Immune Network Vol.13 No.6
The intestinal immune system has an ability to distinguish between the microbiota and pathogenic bacteria, and then activate pro-inflammatory pathways against pathogens for host defense while remaining unresponsive to the microbiota and dietary antigens. In the intestine, abnormal activation of innate immunity causes development of several inflammatory disorders such as inflammatory bowel diseases (IBD). Thus, activity of innate immunity is finely regulated in the intestine. To date, multiple innate immune cells have been shown to maintain gut homeostasis by preventing inadequate adaptive immune responses in the murine intestine. Additionally, several innate immune subsets, which promote Th1 and Th17 responses and are implicated in the pathogenesis of IBD, have recently been identified in the human intestinal mucosa. The demonstration of both murine and human intestinal innate immune subsets contributing to regulation of adaptive immunity emphasizes the conserved innate immune functions across species and might promote development of the intestinal innate immunity-based clinical therapy.
Regulation of Intestinal Homeostasis by Innate Immune Cells
Hisako Kayama,Junichi Nishimura,Kiyoshi Takeda 대한면역학회 2013 Immune Network Vol.13 No.6
The intestinal immune system has an ability to distinguish between the microbiota and pathogenic bacteria, and then activate pro-inflammatory pathways against pathogens for host defense while remaining unresponsive to the microbiota and dietary antigens. In the intestine, abnormal activation of innate immunity causes development of several inflammatory disorders such as inflammatory bowel diseases (IBD). Thus, activity of innate immunity is finely regulated in the intestine. To date, multiple innate immune cells have been shown to maintain gut homeostasis by preventing inadequate adaptive immune responses in the murine intestine. Additionally, several innate immune subsets, which promote Th1 and Th17 responses and are implicated in the pathogenesis of IBD, have recently been identified in the human intestinal mucosa. The demonstration of both murine and human intestinal innate immune subsets contributing to regulation of adaptive immunity emphasizes the conserved innate immune functions across species and might promote development of the intestinal innate immunity-based clinical therapy.
녹산광(鹿山光)(Kayama Mitsu) 한국유화학회 1996 한국응용과학기술학회지 Vol.13 No.3
Essentiality was proposed in the field of lipid by Burr and Burr in 1929. When rats were raised on the fat-free diet, their growth retarded and their skin and tails showed the characteristic deficient symptoms, which were relieved by the addition of ω6(n-6) polyunsaturated fatty acids such as linoleic(LA) and arachidonic(AA) acids to the basal diet. LA is dehydrogenated to r-linolenic add(GLNA) by Δ6 desaturase, then GLNA is 2 carbon chain elongated by elongase to dihoma-r-linolenic acid(DGLNA), which is desaturated by Δ5 desaturase to AA. These acids are called LA family or ω6(n-6) polyunsaturated fatty acids(PUFA). α-Linolenic acid(ALNA) is converted through the series of desaturation and elongation steps to docosahexaenic acid(DHA) via eicosapentaenoic acid(EPA). These acids belong to ALNA family or ω3(n-3)PUFA. Human who consume large amounts of EPA and DHA, which are present in fatty fish and fish oils, have increased levels of these two fatty acid in their plasma and tissue lipids at the expense of LA and AA. Alternately, vegetarians, whose intake of LA in high, have more elevated levels of LA and AA and lower levels of EPA and DHA in plasma lipids and in cell membranes than omnivores. AA and EPA are metabolized to substances called eicosanoids. Those derived form AA are known as prostanocids(prostaglandins and prostacyclins) of the 2-types and leukotrienes of the 4-series, whereas those derived from EPA are known as prostanocids of the 3-types and leukotrienes of the 5-series. DGLNA is a precursor of the 1-types of prostaglandins. The metabolites of AA and EPA have competitve functions. Ingestion of EPA from fish or fish oil replaces AA from membrane phospholipids in practical1y all cells. So this leads to a more physiological state characterized by the production of proatanoids and leukotrienes that have antithrombic, antichemotactic, antivasoconstrictive and antiinflarmmatory properties. It is evident that ω3 fatty acids can affect a number of chronic diseases through eicosanoids alone.
Effect of Water Stress on the Symbiotic Nitrogen Fixation Capacity of Ladino Clover
Jo, Jinki,Yoshida, Shigekata,Kayama, Ryosei 慶北大學校 1982 論文集 Vol.34 No.-
수분결핍이 래디노 클로-버의 공생질소고정에 미치는 영향을 수경법으로 재배한 래디노 클로-버에 대해 아세틸렌 환원법으로 질소고정능을 조사한바 그 결과는 아래와 같았다. 1) 수분결핍의 정도가 심화됨에 따라 래디노 클로-버의 공생질소고정능은 급격히 저하하였다. 2) 래디노 클로-버 각 부위의 수분함량의 변화와 공생질소고정능의 변화 사이에는 각각 높은 상관이 있음을 보여주었다. 3) 수분결핍처리를 심하게 할수록 공생질소고정능의 회복이 어려운 것으로 나타났다.