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Oxygen Vacancy and Magnetism of a Room Temperature Ferromagnet Co-doped TiO2
Ikuo Nakai,M. Sasano,K. Inui,T. Korekawa,H. Ishijima,H. Katoh,Y. J. Li,M. Kurisu 한국물리학회 2013 THE JOURNAL OF THE KOREAN PHYSICAL SOCIETY Vol.63 No.3
We report the local structure and the magnetic properties of a Co-doped TiO2 prepared by usingthe solid state reaction of milling with subsequent annealing. The Co-doped TiO2 shows a hysteresisbehavior in the magnetization curve at room temperature, which maintains the same rutile-typestructure as that of the starting material TiO2. The extended x-ray absorption fine structuremeasurement reveals that it contains some oxygen vacancies only around a Co atom which occupiesa Ti site. This supports the oxygen vacancy mediated ferromagnetic exchange mechanism.
Ikuo Saiki 고려인삼학회 2007 Journal of Ginseng Research Vol.31 No.1
We found that the main bacterial metabolite M1 is an active component of orally administered protopanxadioltype ginsenosides, and that the anti-metastatic effect by oral administration of ginsenosides may be primarily mediated through the inhibition of tumor invasion, migration and growth of tumor cells by their metabolite M1. Pharmacokinetic study after oral administration of ginsenoside Rb1 revealed that M1 was detected in serum for 24 h by HPLC analysis but Rb1 was not detected. M1, with anti-metastatic property, inhibited the proliferation of murine and human tumor cells in a time- and concentration-dependent manner in vitro, and also induced apoptotic cell death (the ladder fragmentation of the extracted DNA). The induction of apoptosis by M1 involved the up-regulation of the cyclin-dependent kinase (CDK) inhibitor p27<SUP>Kip1</SUP> as well as the down-regulation of a proto-oncogene product c-Myc and cyclin D1 in a timedependent manner. Thus, M1 might cause the cell-cycle arrest (G1 phase arrest) in tumor cells through the up/down-regulation of these cell-growth related molecules, and consequently induce apoptosis. The nucleosomal distribution of fluorescence- labeled M1 suggests that the modification of these molecules is induced by transcriptional regulation. Tumorinduced angiogenesis (neovascularization) is one of the most important events concerning tumor growth and metastasis. Neovascularization toward and into tumor is a crucial step for the delivery of nutrition and oxygen to tumors, and also functions as the metastatic pathway to distant organs. M1 inhibited the tube-like formation of hepatic sinusoidal endothelial (HSE) cells induced by the conditioned medium of colon 26-L5 cells in a concentration-dependent manner. However, M1 at the concentrations used in this study did not affect the growth of HSE cells in vitro.
Ikuo Watanobe,Yuzuru Ito,Eigo Akimoto,Yuuki Sekine,Yurie Haruyama,Kota Amemiya,Shozo Miyano,Taijiro Kosaka,Michio Machida,Toshiaki Kitabatake,Kuniaki Kojima 한국간담췌외과학회 2016 한국간담췌외과학회지 Vol.20 No.1
Portal vein thrombosis (PVT) is a rare but serious postoperative complication of pancreaticoduodenectomy (PD). We reported a case of late-onset postoperative PVT with hemorrhage from the common hepatic artery (CHA) in a 73-year-old man who underwent pylorus-preserving pancreaticoduodenectomy (PPPD) for duodenum papilla cancer, followed by reconstruction using the modified Child"s technique. The pancreaticojejunostomy was achieved by end-to-side, 2-layer invagination anastomosis without pancreatic duct stenting. Drain removal and hospital discharge were scheduled on postoperative day (POD) 18, but blood-stained fluid in the drain and sudden hematemesis were noted. Emergency surgery was performed because PVT and imaging findings were suggestive of necrosis of the lifted jejunum. Although no jejunal necrosis was identified during surgery, bleeding from the side of the CHA was detected and the bleeding point was suture-closed to achieve hemostasis. We suspected late-onset postoperative arterial hemorrhage and subsequent hematoma formation, which caused portal vein compression and PVT formation. We chose a conservative treatment strategy for PVT, taking into account the operation time, intraoperative vital signs and blood flow in the portal vein. Despite the complicated postoperative course, he was discharged home in a fully ambulatory state on POD 167.