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Aaij, R.,Adeva, B.,Adinolfi, M.,Ajaltouni, Z.,Akar, S.,Albrecht, J.,Alessio, F.,Alexander, M.,Ali, S.,Alkhazov, G.,Alvarez Cartelle, P.,Alves Jr, A.A.,Amato Jr, S.,Amerio Jr, S.,Amhis Jr, Y.,An Jr, L. Elsevier 2017 Physics letters: B Vol.771 No.-
<P><B>Abstract</B></P> <P>A search for <I>CP</I> violation in <SUP> D ± </SUP> → <SUP> η ′ </SUP> <SUP> π ± </SUP> and D s ± → <SUP> η ′ </SUP> <SUP> π ± </SUP> decays is performed using proton–proton collision data, corresponding to an integrated luminosity of 3 <SUP> fb − 1 </SUP> , recorded by the LHCb experiment at centre-of-mass energies of 7 and 8 TeV. The measured <I>CP</I>-violating charge asymmetries are <SUB> A CP </SUB> ( <SUP> D ± </SUP> → <SUP> η ′ </SUP> <SUP> π ± </SUP> ) = ( − 0.61 ± 0.72 ± 0.53 ± 0.12 ) % and <SUB> A CP </SUB> ( D s ± → <SUP> η ′ </SUP> <SUP> π ± </SUP> ) = ( − 0.82 ± 0.36 ± 0.22 ± 0.27 ) % , where the first uncertainties are statistical, the second systematic, and the third are the uncertainties on the <SUB> A CP </SUB> ( <SUP> D ± </SUP> → K S 0 <SUP> π ± </SUP> ) and <SUB> A CP </SUB> ( D s ± → ϕ <SUP> π ± </SUP> ) measurements used for calibration. The results represent the most precise measurements of these asymmetries to date.</P>
Cancer-derived exosomal Alu RNA promotes colorectal cancer progression
Magliacane Trotta Sara,Adinolfi Antonio,D’Orsi Luca,Panico Sonia,Mercadante Grazia,Mehlen Patrick,Ambati Jayakrishna,De Falco Sandro,Tarallo Valeria 생화학분자생물학회 2024 Experimental and molecular medicine Vol.56 No.-
Inflammation plays a crucial role in cancer progression, but the relevance of the inflammasome remains unclear. Alu RNA was the first endogenous nucleic acid shown to activate the NLRP3 (nucleotide-binding domain leucine-rich repeat containing 3) inflammasome. Here, we showed that Alu RNA can induce epithelial-to-mesenchymal transition (EMT) through NLRP3 inflammasome activation and IL-1β release in colorectal cancer (CRC) cells. Alu RNA is stored, transported and transferred to CRC cells by exosomes. Exosomal Alu RNA promotes tumorigenesis by inducing invasion, metastasis and EMT via NLRP3 inflammasome activation. Consistent with these data, we found that significantly increased Alu RNA expression correlates with the induction of NLRP3 priming in human CRC patients. Furthermore, the level of Alu RNA in circulating exosomes correlates with CRC progression in a preclinical model. These findings reveal the direct involvement of Alu RNA in cancer pathogenesis, and its presence in CRC cell-derived exosomes could be used as a noninvasive diagnostic biomarker.
Measurement of the ratio of branching fractionsB(B0→K*0γ)/B(Bs0→ϕγ)
Aaij, R.,Abellan Beteta, C.,Adeva, B.,Adinolfi, M.,Adrover, C.,Affolder, A.,Ajaltouni, Z.,Albrecht, J.,Alessio, F.,Alexander, M.,Alkhazov, G.,Alvarez Cartelle, P.,Alves, A. A.,Amato, S.,Amhis, Y.,Ande American Physical Society 2012 PHYSICAL REVIEW D - Vol.85 No.11
LHCb Collaboration,Aaij, R.,Abellan Beteta, C.,Adeva, B.,Adinolfi, M.,Adrover, C.,Affolder, A.,Ajaltouni, Z.,Albrecht, J.,Alessio, F.,Alexander, M.,Alkhazov, G.,Alvarez Cartelle, P.,Alves, A.A.,Amato, North-Holland Pub. Co 2011 Physics letters: B Vol.706 No.1
The first observation of the decay B@?<SUB>s</SUB><SUP>0</SUP>→D<SUP>0</SUP>K<SUP>@?0</SUP> using pp data collected by the LHCb detector at a centre-of-mass energy of 7 TeV, corresponding to an integrated luminosity of 36 pb<SUP>-1</SUP>, is reported. A signal of 34.4+/-6.8 events is obtained and the absence of signal is rejected with a statistical significance of more than nine standard deviations. The B@?<SUB>s</SUB><SUP>0</SUP>→D<SUP>0</SUP>K<SUP>@?0</SUP> branching fraction is measured relative to that of B@?<SUP>0</SUP>→D<SUP>0</SUP>ρ<SUP>0</SUP>: B(B@?<SUB>s</SUB><SUP>0</SUP>→D<SUP>0</SUP>K<SUP>@?0</SUP>)B(B@?<SUP>0</SUP>→D<SUP>0</SUP>ρ<SUP>0</SUP>)=1.48+/-0.34+/-0.15+/-0.12, where the first uncertainty is statistical, the second systematic and the third is due to the uncertainty on the ratio of the B<SUP>0</SUP> and B<SUB>s</SUB><SUP>0</SUP> hadronisation fractions.