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김현만,홍욱균,한지영 대한내분비학회 1991 Endocrinology and metabolism Vol.6 No.4
Although it has been well recognized that the neurofibromatosis frequently associates with endocrine anomalies such as pheochromocytoma, thyroid tumor and hyperparathyroidism, there are very few instances of neurofibromatosis associated with acromegaly. The etiological basis of this association is not clear. We report one case of neurofibromatosis concurrently occurenored with acromegaly in a 17-year-old man. A diagnosis of neurofibromatosis had been made by typical skin manifestation on physical examination followed by pathological findings of biopsied tissue from the lesion. A diagnosis of acromegaly had been made by elevated serum growth hormone level followed by detection of mass lesion on brain CT scan. His growth hormone level became lower after bromocriptine therapy. The tumor will be regularly checked up by brain CT scan for growing or changing in nature. (J Kor Soc Endocrinol 6:371~376, 1991)
이승환,이종원,이광훈,조한선,홍욱균 대한신장학회 1990 Kidney Research and Clinical Practice Vol.9 No.4
A case with acute renal failure after consuming wild mushrooms of Amanita variety is presented. The 32-year-old male patient experienced a sudden onset of nausea, vomiting, diarrhea, aMominal pain and generalized weakness 14 hours after ingestion of wild mushrooms with his family, wife and two childern. Over the next 3 days, all of his family showed symptoms, signs and laboratory findings of acute hepaic failure. All of his family except this patient died because of acute hepatic failure. Twelve days sfter ingestion of mushrooms he developed oliguria and generalized edema. Blood chemistry values revealed BUN 113 mg/ dl, creatinine 13.4 mg/dl, Ca 8.0 mg/dl, inorganic phosphorous 4.5mg/dl. Acute tubular necrosis was confirmed by renal biopsy. He recovered from renal failure with conservative management.
한국 성인에서 뇌하수체 기능부전증의 원인 및 내분비학적 특성
김현만,허갑범,이현철,이은직,홍욱균,오미희 대한내분비학회 1993 Endocrinology and metabolism Vol.8 No.4
Pituitary insufficiency can be caused by various diseases. The present study was perfomed on Korean patients with pituitary insufficiency, who were examined from January 1982 to June 1991. Cases under hormone treatment or cases with hypopituitarism developed after surgery or radiotherapy on the pituitary gland were excluded in this study. Two hundred fory senen cases with pituitary insufficiency, which was confirmed by high resolution sella CT scan and combined pituitary stimulation test, were classified basing on their primary causes. The results were as follows: 1) Anomg 247 cases with pituitary insufficiency, 169 cases (68.4%) were pituitary adenoma, 64 cases (25.9%) ischemic necrosis of pituitary gland, 3 cases (1.2%) head trauma, and 11 cases (4.5%) idiopathic 2) Seventy two out of 93 male patients with pituitary insufficiency (77.4%) were pituitary adenoma, with the most prevalent age being the 4 th 5 th decade. 3) Dighty one out of 154 female patients with pituitary insufficiency (51.6%) were pituitary adnoma and 57 cases(37.0%) were Sheehan's syndrome. Most pituitary tumors were found in the 3 rd decade but Sheehan's syndrome were found in the 6 th decade. 4) There was no significant change in nmber of the annual cases with Sheehan's syndrome. 5) Pan-or near total hypopituitarism were observed in 86.1% of cases with Sheehan's syndrome and in 40.4% of cases with pituitary insufficiency caused by tumors. It can be concluded that the most common cause of pituitary insufficiency in Korea is pituitary tumor. However, Sheehan's syndrome is the main cause of pituitary insufficiency in female patients after age of 40. And the degree of pituitary insufficiency in cases with Sheehan's syndrome is more serious than that in pituitary tumor. (J Kor Soc Endocrinol 8:390~397, 1993)
늑막조직생검으로 진단한 Pleural Paragonimiasis 1 예
김기영,이승환,박진현,이재갑,용석중,신계철,홍욱균,김향인,진춘조 대한내과학회 1991 대한내과학회지 Vol.40 No.5
We report a 28-year old female patient with pleural paragonimiasis who did not have the usual diagnostic triad for this parasitic disease. She did not have chronic hemoptysis, there were no pulmonary infiltrations, and stool, sputum and pleural fluid examinations did not yield paragonimus ova. The diagnosis was made on the basis of ova found in the pleural biopsy. Paragonimus pleural effusion was resolved with frequent thoracentesis and oral praziquantel therapy.