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- 저자 : 최훈(Xun Cui) (검색결과 23 건)
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십전대보탕(十全大補湯)이 혈관신생(血管新生) 억제(抑制)에 미치는 효과(效果)
최훈,강희,심범상,김성훈,최승훈,안규석,Cui, Xun,Kang, Hee,Shim, Bum-Sang,Kim, Sung-Hoon,Choi, Seung-Hoon,Ahn, Kyoo-Seok 대한암한의학회 2006 大韓癌韓醫學會誌 Vol.11 No.1
Shiquandabutang is very famous prescription for tonifying vital energy. We examined the anti-metatstastic effect of Shiquandabutang with in vitro invasion assay model. We performed the following experiments and the results are listed below:Cell viability assay was carried to determine the dose of Shiquandabutang. At lower dose under 200 ${\mu}g/m{\ell}$ (89.6%) viability was very high. But, viability downed as dose grows. At the dose of 600 ${\mu}g/m{\ell}$ (54.2%) viability was almost half of that of control. And at high dose of 1000 ${\mu}g/m{\ell}$ (15.8%) viability was very pure. In BrdU incorporation assay, Shiquandabutang treated groups showed the decreased DNA synthesis rate compared with control group.(200 ${\mu}g/m{\ell}$ (64.4%), 400 ${\mu}g/m{\ell}$ (7.3%)) The results of gelatinase assay showed that Shiquandabutang decreases the gelatinolytic activity of MMP-9. We examined tube formation assay and the result was that Shiquandabutang ihhibits the tube formation at the dose of 200 ${\mu}g/m{\ell}$ and 400 ${\mu}g/m{\ell}$. We examined rat aortic ring assay and the result was that Shiquandabutang ihhibits the angiogenesis of the rat aortic ring at the dose of 400 ${\mu}g/m{\ell}$. From our research, part of the mechanism underlying anti-metastastic effect of Shiquandabutang was proven in vitro. Moreover, we knew that Shiquandabutang is more effectively inhibits the angiogenesis at high dose.
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폐열(肺熱)을 치료(治療)하는 약물의 기미론(氣味論)적 약리분석(藥理分析)에 대한 고찰(考察)
최훈 ( Xun Cui ),이청학 ( Cheong Hak Lee ) 한약응용학회 2010 한약응용학회지 Vol.10 No.2
In Traditional Oriental Medicine, the drug treatment has been explained by the thoery of "Qiweilun(氣味論)". So we studied on the drug treatment of drugs to treat FeiRe(肺熱) by "Qiweilun(氣味論)" When FeiRe(肺熱) was caused by "Wai Gan Feng Re(外感風熱)", we make a conclusion that use Mori Folium, Lonicerae Flos, Gypsum Fibrosum. And when FeiRe(肺熱) was caused by "Shi Re(實熱)", we make a conclusion that use Gardeniae Fructus, Scutellariae Radix, Fritillariae Bulbus, Anemarrhenae Rhizoma, Asparagi Radix. When FeiRe(肺熱) was caused by "Fei Zhong Fu Huo(肺中伏火)", we make a conclusion that use Adenophorae Radix, Trichosanthis Semen, Lilii Herba. When FeiRe(肺熱) was caused by "Xu Re(虛熱)", we make a conclusion that use Ophiopogonis Radix, Trichosanthis Radix.
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최훈(Xun Cui),이청학(Cheong Hak Lee),우혜숙(Hye Sook Woo) 한의병리학회 2010 동의생리병리학회지 Vol.24 No.6
In Traditional Oriental Medicine, the drug treatment has been explained by the thoery of Si Qi Wu Wei(四氣五味). Among them, Xin(辛) has effect of Sheng(升), San(散), Xing(行), and Ku(苦) has effect of Jiang xia(降下), Zao shi(燥濕), Jian yin(堅陰). The study on the medical action of Asari Radix shows that Xin ke run zao(辛可潤燥)” was derived from Sheng(升), San(散), Xing(行) efficacy of sour. And the study on the medical action of Phellodendron Bark, Anemarrhenae Rhizoma shows that Ku ke run zao(苦可潤燥)” was derived from Xie(泄) efficacy of bitter.
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김윤경,김정숙,최훈,Kim, Yun-Kyung,Kim, Chung-Seok,Cui, Xun 한국한의학연구원 2004 한국한의학연구원논문집 Vol.10 No.2
The decocting method of herbal medicines is various with a prescription or herbal medicine's characteristics. But it has common principles by which effective elements can be easily extracted with synergistic actions of herbs and which the therapeutic effect of a medicine is amplified. When decocting a herbal medicine, the volume of water is an important factor. Fire for decocting drugs and decocting time are also important factors. The excessive water and unproper decocting time and temperature can reduce the effect of the decoction. Besides a better decoction can be obtained by squeeze. According to herbal medicine's characteristics and prescription, there are herbs that should be treated after a specific method. For example, decoct first and decoct later. It also affects the therapeutic if a right taking method is not carried out. Methods of taking drugs include both the time and the method. We can confirm these decocting and taking methods in the ancient medical literatures of herbal medicine. This article deals with these things in detail.
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김공수,김민호,김창곤,김석기,조경우,최훈,Kim, Kong-Soo,Kim, Min-Ho,Kim, Chang-Gon,Kim, Suk-Kee,Cho, Kyung-Woo,Cui, Xun 대한흉부심장혈관외과학회 2000 Journal of Chest Surgery Vol.33 No.5
Background: Cardiac atrium is an endocrine gland secreting a family of natriuretic peptides. The secretion of atrial natriuretic peptide(ANP) had been shown to be controlled by variable factors. The change in atrial dynamics have been considered as one of the most prominent stimuli for the stimulation of ANP secretion. Hypoxic stress has been shown to increase cardiac ANP secretion. However, the mechanism by which hypoxia increases ANP secretion cardiac ANP secretions. However, the mechanism by which hypoxia increases ANP secretion has not to be defined. Therefore, the purpose of the present study was tow-fold: to develop a protocol to defined the effect of hypoxia on ANP secretion in perfused beating rabbit atria and to clarify the mechanism responsible for the accentuation by hypoxia of ANP secretion. Material and Method: Experiments have been done in perfused beating rabbit atria. ANP was measured by radioimmunoassay. Result: Hypoxic stimulus with nitrogen decreased atrial stroke volume. The decrease in atrial stroke volume recovered basal level during the period of recovery with oxygen. ANP secretion and the concentration of perfusate ANP in terms of extracellular fluid(ECF) translocation which reflects the rate of myocytic release of ANP were increased by hypoxia and returned to basal levels during the recovery. Changes in ECF translocation paralleled by hypoxia and returned to basal levels during the recovery. Changes in ECF translocation paralleled to that of atrial stroke volume. At the start of recovery in atrial storke volume, ECF tranalocation incrased for several minutes. The above responses were stable and reproducible. Glibenclamide treatment prevented the recovery in atrial stroke volume. Increments by hypoxia of ANP secretion and ANP concentration were suppressed by glibenclamide. Conclusion: These results indicate that hypoxia incrased atrial myocytic ANP release and that the mechanism responsible for the accentuation is partially related to the change in K+ATP channel activity.
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