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형방패독산(荊防敗毒散)과 독활지황탕(獨活地黃湯)이 Wistar rat의 노화(老化)에 미치는 영향(影響)
안택원,이수영,Ahn, Taek-Won,Lee, Soo-Young 사상체질의학회 2005 사상체질의학회지 Vol.17 No.3
1. Objectives The purpose of this study is to find out effects of Hyeongbangpaedok-san(HBPDS) and Dokhwaljihwang(DHJH) against decline of physical function as aging. 2. Methods Administrating HBPDS and DHJH to 40-week-old Wistar rat for 10 weeks so, I researched weight change, weight change of internal organs, and hematological and serological changes. 3. Results & Conclusions 1. Both examining groups, which were taken HBPDS and DHJH, got more weight than control group. But that was regardless. 2. Both examining groups got more weight on internal organs than control group. But that was regardless, too. 3. Both examining groups decreased in amount of MDA in serum, as contrasted with control group. But it was regardless. 4. Both examining groups improved on hematological condition. WBC, RBC, Hgb, monocytes and eosinophil rates were decreasing and HCT and PLT were increasing. Especially monocytes(p<0.001) and eosinophil(p<0.05) rate of DHJH taken group was decreased remarkably. 5-1. Both examining groups show decline in each item of functional examination of liver, such as ALT, AST, T-bilirubin, T-protein, ALB, A/G. T-chol, TG, etc. HBPDS taken group showed meaningful decline in Albumin(p<0.01) and A/G(p<0.01) and DHJH taken group showed meaningful decline in T-bilirubin(p<0.01). 5-2. Both examining groups showed decline in items of functional examination of kidneys. Specially HBPDS taken group showed meaningful decline in CRN rate(p<0.05) and DHJH taken group showed meaningful decline in BUN rate(p<0.05). As those results, HBPDS and DHJH are effective against decline of physical function as aging.
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加味通栓化瘀湯이 血栓症과 腦虛血症 腦損傷에 미치는 影響에 대한 實驗的 硏究
安澤源,金炳卓 대전대학교 韓醫學연구소 1999 한의학연구소 논문집 Vol.8 No.1
The effect of KamiTongJonHaaATang extracts on hypercholesterolemia, platelet aggregation, pulm onary thrombosis, KCN-induced coma, forcal brain ischemia, cytotoxicity of PC12 cells induced by amyloid β protein(25-35), and NO production in RAW cells stimulated lipopolysaccharide were inve stigated, respectively. The results were summarized as follows: 1. KTJHAT extracts showed a significant decrease of serum total cholesterol, triglyceride, phosph olipid, LDL -cholesterol, and VLDL-cholesterol in hypercholesterolemia induced by 2% cholesterol diet in NZW rabbit. 2. KTJHAT extracts induced a significant inhibition of human platelet aggregation induced by thr ombin and ADP but did not affect human platelet aggregation induced by collagen. 3. KTJHAT extracts showed a protective effect on pulmonary thrombosis induced by collagen an d epinephrine. 4. KTJHAT extracts prolonged the duration of KCN--induced coma. 5. KTJHAT extracts showed a significant decrease of brain ischemic area and edema in MCA oc clusion. Also, KTJHAT extracts showed a decrease of neurologic grade in hind limb but did not aff ect neurologic grade in fore limb. 6. KTJHAT extracts showed a protective effect on cytotoxicity of PC 12 cells induced by amyloid ;3 protein(25-35) in a dose dependent manner. 7. KTJHAT extracts showed a significant decrease of NO production in RAW cells induced by lipopolysaccharide. These results suggested that KTJHAT extracts might be usefully applied for prevention and treatement of thrombosis and brain damage
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