Nd-YAG레이저에 의한 치수 절단후 잔존 치수 조직의 치유과정에 관한 전자현미경적 연구

박동성,임성삼 서울大學校 齒科大學 1994 齒大 論文集 Vol.18 No.2

렛트적출심방수축에 미치는 Niclofolan의 영향

박동성 中央醫學社 1982 中央醫學 Vol.42 No.1

기능 위주의 통신 구조에서 적응력 있는 오류제어 선정 기법 ( A Flexible Error Control Mechanism in Function Based Model )

박동성,이상헌,박재성,이재용,이상배 대한전자공학회 1996 대한전자공학회 학술대회 Vol.1996 No.1

핫프레스법에 의한 TiNi/Al6061 형상기억복합재료의 제조 및 기계적 특성에 관한 연구

박동성,이준희,이규창,박영철,Park, Dong-Sung,Lee, Jun-Hee,Lee, Guy-Chang,Park, Young-Chul 대한기계학회 2002 大韓機械學會論文集A Vol.26 No.7

Al alloy matrix composite with TiNi shape memory fiber as reinforcement has been fabricated by hot pressing to investigate microstructures and mechanical properties. The analysis of SEM and EDS showed that the composites have shown good interface bonding. The stress-strain behavior of the composites was evaluated at temperatures between 363K and room temperature as a function of prestrain, and it showed that the yield stress at 363K was higher than that of the room temperature. Especially, the yield stress of this composite increases with increasing the amount of prestrain, and it also depends on the volume fraction of fiber and heat treatment. The smartness of the composite is given due to the shape memory effect of the TiNi fiber which generates compressive residual stress in the matrix material when heated after being prestrained. Microstructural observation has revealed that interfacial reactions occur between the matrix and fiber, creating two intermetallic layers.

MINI-MAP 네트워크 관리에 관한 연구 ( A Study on the Mini-MAP Network management )

박동성,우종욱,김중규,박민용,이상배 대한전자공학회 1990 대한전자공학회 학술대회 Vol.1990 No.1

MAP 네트워크 관리 시스템에 관한 연구 ( A Study on the MAP Network Management System )

박동성,강문식,김학,박민용,이상배 대한전자공학회 1990 대한전자공학회 학술대회 Vol.1990 No.7

일본연구동향 : 인류학적인 인식론을 위하여

박동성 국민대학교 일본학연구소 2008 일본공간 Vol.3 No.-

Cervical Spondylosis의 Posterior Decompression

박동성,김승래 慶北大學校 醫科大學 1976 慶北醫大雜誌 Vol.17 No.2

세 가지 역충전재의 근단부 폐쇄 효과에 관한 전기화학적 연구

박동성,손서진,오태석,유현미,박찬제,임순호,이영규,계승범 대한치과보존학회 2004 Restorative Dentistry & Endodontics Vol.29 No.4

The purpose of this study was to evaluate the apical sealing ability of Super-EBA, MTA and Dyract-flow as retrofilling materials. Forty-eight extracted human teeth with straight and single root canal were used in this study. The root canals were prepared to a #40 apical canal size and obturated with gutter-percha. Apicoectomies were performed and root end cavities were prepared to a depth of 3mm using an ultrasonic device. The root end cavities were filled with Super-EBA, MTA or Dyract-flow. Leakage was measured using an electrochemical technique for 4 weeks. According to this study, the results were as follows. 1. Increasing leakage with time was observed in all groups. 2. No significant difference was noted among the 3 groups with time (p = 0.216). 3. No significant difference was noted among the 3 groups when measured within the same time interval (p = 0.814). The results of this study suggest that the sealing ability of Dyract-flow is equal to that of Super-EBA and MTA, and Dyract-flow may be an alternative to other materials for root-end filling. . 본 연구의 목적은 Super-EBA, MTA, Dyract-flow를 근관 역충전재로 사용하였을 때의 근단부 폐쇄 능력을 전기화학적 방법을 통해 알아보고자 하는 것이다. 48개의 단근치를 Profile을 사용하여 근관형성한 후, Gutter-percha cone으로 수직가압 충전을 시행하였다. 근단부에서 3mm 되는 부위를 bur로 절단한 후, ultrasonic device로 3mm 깊이의 역충전 와동을 형성하였다. 각 실험군은 Super-EBA, MTA, 혹은 Dyract-flow로 역충전하고 4주에 걸쳐 전기화학적인 방법으로 치근단 누출의 변화를 측정한 후, Repeated measures of ANOVA로 통계분석하였다. 그 결과 모든 시편에서 시간에 따른 치근단 누출의 증가가 관찰되었으나, 각 실험군 간에 누출의 차이는 나타나지 않았다. 이러한 결과는 Dyract-flow의 치근단 역충전재로써의 사용 가능성을 시사한다.

高濃度에타놀投與가 흰쥐 數種臟器의 酸素消費量과 肺容壓率에 미치는 影響

朴東盛,黃樹寬,朴載植,朱永恩 慶北大學校 醫科大學 1982 慶北醫大誌 Vol.23 No.2

Ethanol 投與로 因한 흰쥐 數種臟器의 酸素消費量(QO_2)과 血中乳酸濃度 및 肺容壓率의 變化를 살펴보기 위해서, 흰쥐를 材料로하여 50% ethanol을 體重 100gm 當 1.5㎖ 씩 經口적으로 單回投與하여 1時間에서 24時間까지 經時的으로 肝, 腎, 脾 및 腦組織의 QO_2를 測定하고, 血中乳酸濃度 및 肺容壓率를 測定하여 對照群과 比較하여 다음과 같은 結果를 얻었다. QO_2는 肝組織에서 對照의 2.11±0.10㎕O_2/hr/㎎D. W. 에 比해 ethanol 投與後 1∼6 時間에는 有意하게 높았으며, 特히 1 및 3時間에는 各各 2.81±0.15 및 2.82±0.07㎕O_2/hr/㎎D. W. 로서 最高値를 나타내었다. 腎組織은 對照의 5.33±0.26㎕O_2/hr/㎎D. W. 에 比해 ethanol投與後 1∼12時間에는 有意하게 높았으며, 그 中 3時間에는 7.47±0.32㎕O_2/hr/㎎D. W. 로서 最高値를 나타내었다. 腦組織은 對照의 4.20±0.14㎕O_2/hr/㎎D. W.에 比해 ethanol 投與後 1∼6時間에는 有意하게 높았으며, 特히 3時間에는 5.53±0.29㎕O_2/hr/㎎D. W로서 最高値를 나타내었다. 脾組織은 對照의 3.55±0.13㎕O_2/hr/㎎D. W.에 比해 ethanol 投與後 1∼6 時間에는 有意하게 높았으며, 이 中 3時間에는 5.81±0.23㎕O_2/hr/㎎D. W로서 最高値를 나타내었다. 卽 肝, 腎, 脾 및 腦組織 모두의 QO_2는 對照値에 比해서 ethanol 投與後 1, 3 및 6時間에 有意하게 높았으며, 이 中 3時間에 最高値를 나타낸 後 漸次 減少하여 12 및 24時間에는 別差異가 없었다. 腎은 12時間까지 有意하게 높았다. 그리고 各 組織의 QO_2의 比較에서 腎이 가장 높고, 다음이 腦, 그리고 脾, 肝의 順으로 나타났다. 血中乳酸濃度는 對照의 12.57±0.47㎎/㎗ 에 比해 ethanol 投與後 1 및 3時間에 各各 24.36±2.70㎎/㎗ 및 22.48±3.21㎎/㎗로서 有意하게 높았으며, 6∼24時間에는 別 差異가 없었다. 血中乳酸濃度 對 QO_2의 比는 對照에 比해 1 및 3時間에는 有意하게 높았고, 6 및 12時間에는 有意하게 낮았다. 組織의 QO_2 對 血中乳酸濃度의 比는 對照에 比해 ethanol 投與後 1時間에 有意하게 높다가 3時間에는 有意하게 낮으며, 다시 6 및 12時間에는 有意하게 높은 후 漸次 낮아져 24時間에는 特히 腎과 脾가 有意하게 낮았다. 組織의 QO_2×血中乳酸濃度에서 對照에 比해 ethanol 投與後 1 및 3時間에는 有意하게 높다가 漸次 減少하여 脾는 12時間에 有意하게 낮았다. 肺容積은 ethanol投與後 1∼2時間에서 낮은 壓力下에서는 대체로 正常보다 낮았고, 높은 壓力下에서는 對照値와 비슷했으며, 24時間에는 모든 壓力下에서 對照와 거의 같은 값을 나타내었다. 肺容壓率은 ethanol投與後 3時間까지 對照보다 增加된 후 그 後 次次 減少하였다. 以上을 綜合해 보면 血中 ethanol 濃度가 0.72gm/㎗ 가 될 程度의 高濃度 ethanol을 投與해서도 QO_2가 增加되며, ethanol投與 1∼3時間에 最高値를 나타낸 事實과 ethanol 投與로 因한 血中乳酸濃度의 增加에서 1時間에 最高値에 達한 점, 그리고 肺容壓率이 ethanol 投與 初期에 增加를 보이고 肺容積이 ethanol 投與後 24時間에 거의 對照値로 되돌아온다는 事實等은 意義있는 結果인 것으로 思料되는 바이다. In order to elucidate the effect of ethanol (EOH) administration on the oxygen consumption rate (QO_2) of some vital organs and lung compliance, ??a single dose of 1.5㎖ of 50% EOH per 100gm of body weight was administered orally to rats, and QO_2 of the liver, kidney, brain and spleen, and blood lactate level and lung compliance were measured from 1 hr to 24 hrs after EOH ingestion. The results were compared with the control group and summarized as follows: QO_2 of the liver was significantly elevated from 1 through 6 hrs after EOH ingestion compared with the control value of 2.11 0.10㎕O_2/hr/㎎ dry weight (D.W. ). The values at 1 and 3 hrs were 2.81±0.15 and 2.81±0.07㎕O_2/hr/㎎D. W. both being the peak levels, respectively. QO_2 of the kidney was significantly elevated from 1 through 12 hrs compared with the control value of 5.33±0.26㎕O_2/hr/㎎D. W. The value at 3 hrs showed the heighest value of 7.47±0.32㎕O_2/hr/㎎ D. W. QO_2 of the brain was significantly elevated from 1 through 6 hrs compared with the control value of 4.20±0.14㎕O_2/hr/㎎D. W. The value at 3 hrs showed the peak level of 5.53±0.29㎕O_2/hr/㎎D. W. QO_2 of the spleen was significantly elevated from 1 through 6 hrs compared with the control value of 3.55±0.13㎕O_2/hr/㎎D. W. The peak level was 5.81 0.23㎕O_2/hr/㎎D. W. at 3 hrs. Thus, QO_2 of the liver, kidney, brain and spleen were uniformly elevated after EOH ingestion showing the peak level at 3 hrs and returning to the control level by 12 or 24 hrs. Also, among the studied organs, the kidney showed the highest QO_2 value, followed by the brain, spleen and liver in the order named. The blood lactate was also elevated significantly at 1 and 3 hrs after EOH ingestion, the figures being 24.36 2.70 and 22.48 3.21 ㎎/㎗, respectively, compared with the value of 12.57±0.47 ㎎/㎗. The blood lactate vs. QO_2 ratio was significantly elevated at 1 and 3 hrs after EOH ingestion and was significantly lowered at 6 and 12 hrs aompared with control. The QO_2 vs. blood lactate ratio was significantly elevated at 1 hr after EOH ingestion, significantly lowered at 3 hrs, significantly elevated at 6 and 12 hrs, again significantly in the kidney and spleen, lowered at 24 hrs compared with the control value. The product of QO_2 times the blood lactate was significantly elevated at 1 and 3 hrs after EOH ingestion and gradually lowered subsequently, and significantly lowered in the spleen at 12 hrs compared with the control value. The dynamic lung volume from 1 to 2 hrs after EOH ingestion compared with the control was smaller under lower pressures but similar under higher pressures. At 24 hrs after EOH ingestion it was almost the same as the control under all pressures. The lung compliance was increased until 3 hrs after EOH ingestion but was decreased gradually thereafter. From the above, we should note that the QO_2 is increased by ingestion of EOH as concentrated as 0.72gm/㎗ showing its peak value from 1 to 3 hrs after ingestion, that the blood lactate is increased by EOH ingestion with the peak at 1 hr, and that the lung compliance is increased during the initial period with the dynamic lung volume returning almost to the control value in 24 hrs after EOH ingestion.